Zobrazeno 1 - 10
of 68
pro vyhledávání: '"Ajai A Dandekar"'
Autor:
Yu-Ying Phoebe Hsieh, Wanting Sun, Janet M Young, Robin Cheung, Deborah A Hogan, Ajai A Dandekar, Harmit S Malik
Publikováno v:
PLoS Biology, Vol 22, Iss 6, p e3002694 (2024)
Fungi and bacteria coexist in many polymicrobial communities, yet the molecular basis of their interactions remains poorly understood. Here, we show that the fungus Candida albicans sequesters essential magnesium ions from the bacterium Pseudomonas a
Externí odkaz:
https://doaj.org/article/2a21b5d9164a439aa58deebd1547c39b
Publikováno v:
PLoS Pathogens, Vol 20, Iss 3, p e1012078 (2024)
XRE-cupin family proteins containing an DNA-binding domain and a cupin signal-sensing domain are widely distributed in bacteria. In Pseudomonas aeruginosa, XRE-cupin transcription factors have long been recognized as regulators exclusively controllin
Externí odkaz:
https://doaj.org/article/ed47e7fd85a54b2cae6a6b54b6b76679
Autor:
Caleb M Schwartzkopf, Autumn J Robinson, Mary Ellenbecker, Dominick R Faith, Amelia K Schmidt, Diane M Brooks, Lincoln Lewerke, Ekaterina Voronina, Ajai A Dandekar, Patrick R Secor
Publikováno v:
PLoS Pathogens, Vol 19, Iss 2, p e1010925 (2023)
The opportunistic pathogen Pseudomonas aeruginosa PAO1 is infected by the filamentous bacteriophage Pf4. Pf4 virions promote biofilm formation, protect bacteria from antibiotics, and modulate animal immune responses in ways that promote infection. Fu
Externí odkaz:
https://doaj.org/article/e25d2e5c1a504c2b804c4ab1f43c9be5
Autor:
Omri Wurtzel, Deborah R Yoder-Himes, Kook Han, Ajai A Dandekar, Sarit Edelheit, E Peter Greenberg, Rotem Sorek, Stephen Lory
Publikováno v:
PLoS Pathogens, Vol 8, Iss 9, p e1002945 (2012)
One of the hallmarks of opportunistic pathogens is their ability to adjust and respond to a wide range of environmental and host-associated conditions. The human pathogen Pseudomonas aeruginosa has an ability to thrive in a variety of hosts and cause
Externí odkaz:
https://doaj.org/article/215be2ac9dda4370ae88d1aa7a674d8a
A Metabolic Trade-Off Modulates Policing of Social Cheaters in Populations of Pseudomonas aeruginosa
Publikováno v:
Frontiers in Microbiology, Vol 9 (2018)
Pseudomonas aeruginosa uses quorum sensing (QS) to regulate the production of public goods such as the secreted protease elastase. P. aeruginosa requires the LasI–LasR QS circuit to induce elastase and enable growth on casein as the sole carbon and
Externí odkaz:
https://doaj.org/article/0f89b185a71643ab9dc42894a573bf9c
Autor:
Caleb M. Schwartzkopf, Autumn J. Robinson, Mary Ellenbecker, Dominick R. Faith, Diane M. Brooks, Lincoln Lewerke, Ekaterina Voronina, Ajai A. Dandekar, Patrick R. Secor
The opportunistic pathogenPseudomonas aeruginosaPAO1 is infected by the filamentous bacteriophage Pf4. Pf4 virions promote biofilm formation, protect bacteria from antibiotics, and modulate animal immune responses in ways that promote infection. Furt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b4ff491f0edc8d958429b2a762108305
https://doi.org/10.1101/2022.10.11.511824
https://doi.org/10.1101/2022.10.11.511824
Publikováno v:
mSystems. 7
In people with the genetic disease cystic fibrosis (CF), bacterial infections involving the opportunistic pathogen Pseudomonas aeruginosa are a significant cause of morbidity and mortality. P. aeruginosa uses a cell-cell signaling mechanism called qu
Autor:
Maia Kivisaar, Kalai Mathee, Herbert P. Schweizer, Teresa R. de Kievit, Martin Welch, Ajai A. Dandekar, Dao Nguyen, Diane McDougald, Lars E. P. Dietrich, Craig Winstanley, Kim R. Hardie
Publikováno v:
Journal of Medical Microbiology
Autor:
Perez C, E. P. Greenberg, Amy L. Schaefer, Ajai A. Dandekar, Kyle L. Asfahl, Nicole E. Smalley
The bacterium Pseudomonas aeruginosa is an opportunistic pathogen and it thrives in many different saprophytic habitats. In this bacterium acyl-homoserine lactone quorum sensing (QS) can activate expression of over 100 genes, many of which code for e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e0ea523ba02803d42c3eebcff9d515f8
https://doi.org/10.1101/2021.07.01.450773
https://doi.org/10.1101/2021.07.01.450773
Publikováno v:
Scientific Reports, Vol 9, Iss 1, Pp 1-14 (2019)
SCIENTIFIC REPORTS
Scientific Reports
SCIENTIFIC REPORTS
Scientific Reports
In cystic fibrosis (CF) airways, the opportunistic pathogen Pseudomonas aeruginosa evolves from an acute to a chronic infection phenotype. Yet, the in vivo factors influencing the evolutionary trajectory of P. aeruginosa are poorly understood. This s