Zobrazeno 1 - 10
of 27
pro vyhledávání: '"Ahmad POURSADEGH ZONOUZI"'
Autor:
Samira Shakerizadeh, Mohammadi Shekari, Abdolazim Nejatizadeh, Aliakbar Poursadegh zonouzi, Hedieh fardmanesh, Ahmad Poursadegh zonouzi
Publikováno v:
Jorjani Biomedicine Journal, Vol 4, Iss 2, Pp 48-57 (2017)
Background and objectives: Deregulation in the expression of microRNAs is involved in the pathogenesis of various malignancies. Impaired microRNAs processing pathway is one possible mechanism for global deregulation of the miRNAs. Exportin 5 (XPO5) i
Externí odkaz:
https://doaj.org/article/4de3b244734746dbbdcee904d6950ebe
Autor:
Marzieh NASERI, Masoud AKBARZADEHLALEH, Marjan MASOUDI, Najmeh AHANGARI, Ali Akbar POURSADEGH ZONOUZI, Ahmad POURSADEGH ZONOUZI, Leila SHAMS, Azim NEJATIZADEH
Publikováno v:
Iranian Journal of Public Health, Vol 47, Iss 1 (2017)
Background: Autosomal recessive non-syndromic hearing loss (ARNSHL) a most frequent hereditary type of hearing impairment, exhibit tremendous genetic heterogeneity. We aimed to determine the contribution of three common DFNB loci (DFNB4, DFNB28, and
Externí odkaz:
https://doaj.org/article/c43595458d784b69b61d684b565be462
Autor:
Marjan MASOUDI, Najmeh AHANGARI, Ali Akbar POURSADEGH ZONOUZI, Ahmad POURSADEGH ZONOUZI, Azim NEJATIZADEH
Publikováno v:
Iranian Journal of Public Health, Vol 45, Iss 5 (2016)
Background: Autosomal recessive non-syndromic hearing loss (ARNSHL) is the most common hereditary form of deafness, and exhibits a great deal of genetic heterogeneity. So far, more than seventy various DFNB loci have been mapped for ARNSHL by linkage
Externí odkaz:
https://doaj.org/article/cd7dd1fe6a514c8c86651d67ce00e422
Autor:
Masoud Akbarzadeh Laleh, Marzieh Naseri, Ali Akbar Poursadegh Zonouzi, Ahmad Poursadegh Zonouzi, Marjan Masoudi, Najmeh Ahangari, Leila Shams, Azim Nejatizadeh
Publikováno v:
Journal of Research in Medical Sciences, Vol 22, Iss 1, Pp 99-99 (2017)
Background: We aimed to determine the contribution of four DFNB loci and mutation analysis of gap junction beta-2 (GJB2) and GJB4 genes in autosomal recessive nonsyndromic hearing loss (ARNSHL) in South of Iran. Materials and Methods: A total of 36 l
Externí odkaz:
https://doaj.org/article/2a9c924d1103438aaa6dda6deb30df95
Autor:
Ebrahim Eftekhar, Kamila Kamali, Sara Aghakhani Chegeni, Ahmad Poursadegh Zonouzi, Ali Akbar Poursadegh Zonouzi, Farhad Ghadiri Soufi
Publikováno v:
Endocrine Regulations, Vol 52, Iss 3, Pp 123-127 (2018)
Objectives. It has been shown that dysregulation of miRNAs expression contributes to the pathogenesis and progression of the diabetes and diabetes-related complications. Drosha, DGCR8, Dicer, and Ago-2 are involved in the miRNA maturation. The aim of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b99fae0ff3f97df93db86207152c6165
https://doaj.org/article/60ce62fdee6a46b2a9f816e81e3a971a
https://doaj.org/article/60ce62fdee6a46b2a9f816e81e3a971a
Autor:
Mohammad Shekari, Ali Akbar Poursadegh Zonouzi, Majid Tozihi, Samira Shakerizadeh, Azim Nejatizadeh, Mohammad Rahmati-Yamchi, Hedieh Fardmanesh, Ahmad Poursadegh Zonouzi
Publikováno v:
Breast Disease. 37:55-62
BACKGROUND Impaired miRNAs processing pathway is one interesting scenario for global downregulation of the miRNAome in various types of malignancy. We previously reported that DGCR8 and Dicer genes dysregulated in patients with breast cancer. OBJECTI
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f355de32037db1b33c25261e4f5944a
https://doi.org/10.3233/bd-170274
https://doi.org/10.3233/bd-170274
Autor:
Ahmad Poursadegh Zonouzi, Sayed Mostafa Hosseini, Younes Aftabi, Roya Azadkhah, Asghar Hosseinzadeh, Nasser Pouladi, Shahla Danaii, Nasrin Bargahi, Alireza Azani, Ali Akbar Poursadegh Zonouzi, Hourieh Khani, Leida Heidary
Publikováno v:
European Journal of Obstetrics & Gynecology and Reproductive Biology. 215:93-100
Many lines of evidence suggest that reduced production of nitric oxide (NO) due to single nucleotide polymorphisms in endothelial nitric oxide synthase (eNOS) gene may affect the implantation and maintenance of pregnancy. Accordingly, our objective w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28c323e217550488f9155217f50d81ad
https://doi.org/10.1016/j.ejogrb.2017.05.024
https://doi.org/10.1016/j.ejogrb.2017.05.024
Autor:
Yadollah Omidi, Fariba Mahmoodpoor, Nasser Samadi, Mohammadreza Ardalan, Ahmad Poursadegh Zonouzi, Sepideh Zununi Vahed
Publikováno v:
Archives of Medical Research. 48:96-104
Chronic allograft dysfunction (CAD) is the major cause of renal allograft loss and can only be diagnosed by invasive histological examinations. The current study aimed to determine whether or not the circulating miR-125a, miR-150, miR-192, miR-200b,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::49b4a0c4479a84f091d2361d67ef6631
https://doi.org/10.1016/j.arcmed.2017.03.004
https://doi.org/10.1016/j.arcmed.2017.03.004
Autor:
Samira Shakerizadeh, Asghar Hosseinzadeh, Ahmad Poursadegh Zonouzi, Abolfazl Movafagh, Hedieh Fardmanesh, Ali Akbar Poursadegh Zonouzi, Mohammad Shekari, Shohreh Alizadeh Shargh
Publikováno v:
Gene. 581:146-151
High-throughput experimental studies have indicated that the miRNAome is globally downregulated in various types of malignancy, and dysregulation of miRNAs processing component(s) is one possible mechanism for this phenomenon. Despite the progression
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::985db5fd73c959fd02aa8d92163268cb
https://doi.org/10.1016/j.gene.2016.01.033
https://doi.org/10.1016/j.gene.2016.01.033
Publikováno v:
Blood Coagulation & Fibrinolysis. 27:308-312
Recently much attention has been paid to the possibly considerable role of the thrombophilic gene polymorphisms in the pathogenesis of deep venous thromboembolism (DVT). However, the reported results are controversial. Hence, this study aimed to disc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::012de5f576d0bc5bad757098807c3269
https://doi.org/10.1097/mbc.0000000000000430
https://doi.org/10.1097/mbc.0000000000000430