Zobrazeno 1 - 10
of 136
pro vyhledávání: '"Agustin P. Dalmasso"'
Autor:
John P. Carney, Jill Schappa Faustich, Matthew T. Lahti, Paul E. Ashworth, Agustin P. Dalmasso, Yuriy Moklyak, Richard W. Bianco
Publikováno v:
Journal of Investigative Surgery, Vol 35, Iss 2, Pp 371-377 (2022)
Background Transcatheter aortic valve replacement (TAVR) is an effective therapy in treating high-risk patients suffering from aortic stenosis. Animal models used to evaluate safety and efficacy of TAVR devices prior to clinical use lack a stenotic a
Externí odkaz:
https://doaj.org/article/1c9614a3400d4bcf84add278826b1558
Autor:
Jill T. Schappa Faustich, Paul E. Ashworth, Yuriy Moklyak, John P. Carney, Matthew T. Lahti, Agustin P. Dalmasso, Richard W. Bianco
Publikováno v:
Journal of Investigative Surgery. 35:371-377
Transcatheter aortic valve replacement (TAVR) is an effective therapy in treating high-risk patients suffering from aortic stenosis. Animal models used to evaluate safety and efficacy of TAVR devices prior to clinical use lack a stenotic aortic annul
Autor:
Margaret A. Lindorfer, Marna E. Ericson, Julia Nguyen, Trevor Killeen, Gregory M. Vercellotti, Terry Robert Schaid, Fuad Abdulla, Chunsheng Chen, John D. Belcher, Ronald P. Taylor, Agustin P. Dalmasso
Innate immune complement activation may contribute to sickle cell disease (SCD) pathogenesis. Ischemia-reperfusion physiology is a key component of the inflammatory and vaso-occlusive milieu in SCD and is associated with complement activation. C5a is
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0e4cd19ec201b19aa9fd0f4914de6966
https://europepmc.org/articles/PMC6986874/
https://europepmc.org/articles/PMC6986874/
Autor:
Richard W. Bianco, Yuriy Moklyak, Agustin P. Dalmasso, John P. Carney, Matthew T. Lahti, Steven S. Qi
Publikováno v:
Structural Heart. 4:14
Autor:
John P, Carney, Lindsey M, Zhang, Jeffrey J, Larson, Matthew T, Lahti, Nicholas A, Robinson, Agustin P, Dalmasso, Richard W, Bianco
Publikováno v:
The Journal of heart valve disease. 26(4)
Xenograft conduits have been used successfully to repair congenital heart defects, but are prone to failure over time. Hence, in order to improve patient outcomes, better xenografts are being developed. When evaluating a conduit's performance and saf
Autor:
Agustin P. Dalmasso, Barbara A. Benson, Karen R. Wasiluk, Alexander K. Tsai, Daniel Goldish, Gregory M. Vercellotti
Publikováno v:
Journal of Biological Chemistry. 289:838-847
Injury to endothelial cells (ECs) often results in cell retraction and gap formation. When caused by antigen aggregation or complement, this injury can be prevented by pretreatment of the ECs with IL-4, suggesting that IL-4 modifies the intercellular
Autor:
Agustin P. Dalmasso
Publikováno v:
Xenotransplantation. 19:137-143
Dalmasso AP. On the intersections of basic and applied research in xenotransplantation. Xenotransplantation 2012; 19: 137–143. © 2012 John Wiley & Sons A/S. Abstract: I am very grateful to the Council and members of the International Xenotransplan
Autor:
Dame Idossa, Agustin P. Dalmasso, Sylvester M. Black, Gregory M. Vercellotti, Barbara A. Benson
Publikováno v:
Xenotransplantation. 18:343-354
Black SM, Benson BA, Idossa D, Vercellotti GM, Dalmasso AP. Protection of porcine endothelial cells against apoptosis with interleukin-4. Xenotransplantation 2011; 18: 343–354. © 2011 John Wiley & Sons A/S. Abstract: Background: Apoptosis is cruci
Autor:
Barbara A. Benson, Brett K. Levay-Young, Mark S. Rutherford, Bjorn H. Batdorf, Sylvester M. Black, Agustin P. Dalmasso, Megan E. Schott
Publikováno v:
European Journal of Immunology. 40:803-812
We have shown previously that cytokines IL-4 and IL-13 induce protection in porcine vascular endothelial cells (EC) against killing by the membrane attack complex (MAC) of human complement. This protection is intrinsic, not due to changes in compleme
Endothelial cell activation and injury by the terminal pathway of complement is important in various pathobiological processes, including xenograft rejection. Protection against injury by human complement can be induced in porcine endothelial cells (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b513086dc380a44d12560939dc7fdee5
https://europepmc.org/articles/PMC4519407/
https://europepmc.org/articles/PMC4519407/