Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Agustín Sánchez-Temprano"'
Autor:
Marta Varela-Eirín, Paula Carpintero-Fernández, Amanda Guitián-Caamaño, Adrián Varela-Vázquez, Alejandro García-Yuste, Agustín Sánchez-Temprano, Susana B. Bravo-López, José Yañez-Cabanas, Eduardo Fonseca, Raquel Largo, Ali Mobasheri, José Ramón Caeiro, María D. Mayán
Publikováno v:
Cell Death and Disease, Vol 13, Iss 8, Pp 1-13 (2022)
Abstract The accumulation of senescent cells is a key characteristic of aging, leading to the progression of age-related diseases such as osteoarthritis (OA). Previous data from our laboratory has demonstrated that high levels of the transmembrane pr
Externí odkaz:
https://doaj.org/article/9c94a9f71b8848c09400880a1b60b8b2
Autor:
Jessica González‐Sánchez, Agustín Sánchez‐Temprano, Tania Cid‐Díaz, Regina Pabst‐Fernández, Carlos S. Mosteiro, Rosalía Gallego, Ruben Nogueiras, Xesús Casabiell, Gillian S. Butler‐Browne, Vincent Mouly, José Luis Relova, Yolanda Pazos, Jesús P. Camiña
Publikováno v:
Journal of Cachexia, Sarcopenia and Muscle, Vol 9, Iss 6, Pp 1063-1078 (2018)
Abstract Background This study was performed to test the therapeutic potential of obestatin, an autocrine anabolic factor regulating skeletal muscle repair, to ameliorate the Duchenne muscular dystrophy (DMD) phenotype. Methods and results Using a mu
Externí odkaz:
https://doaj.org/article/ac85ac9944404db6b8a8d3a098d48356
Publikováno v:
ChemBioChem.
Autor:
Alfonso Blanco, Antonio Casado-Díaz, Paula Carpintero-Fernández, Agustín Sánchez-Temprano, Mustapha Kandouz, M. Varela-Eirin, Maria D. Mayan, Adrian Varela-Vazquez, Alfonso Calañas Continente, Eduardo Fonseca, José R. Caeiro, Carlos L. Paíno, Virginia Mato
Publikováno v:
RUC. Repositorio da Universidade da Coruña
instname
RUC: Repositorio da Universidade da Coruña
Universidade da Coruña (UDC)
Aging (Albany NY)
instname
RUC: Repositorio da Universidade da Coruña
Universidade da Coruña (UDC)
Aging (Albany NY)
[Abstract] Articular cartilage and synovial tissue from patients with osteoarthritis (OA) show an overactivity of connexin43 (Cx43) and accumulation of senescent cells associated with disrupted tissue regeneration and disease progression. The aim of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c694dce7fb94080b515bb644f4a791ab
https://hdl.handle.net/2183/26271
https://hdl.handle.net/2183/26271
Autor:
Jessica, González-Sánchez, Agustín, Sánchez-Temprano, Tania, Cid-Díaz, Regina, Pabst-Fernández, Carlos S, Mosteiro, Rosalía, Gallego, Ruben, Nogueiras, Xesús, Casabiell, Gillian S, Butler-Browne, Vincent, Mouly, José Luis, Relova, Yolanda, Pazos, Jesús P, Camiña
Publikováno v:
Journal of Cachexia, Sarcopenia and Muscle
Background This study was performed to test the therapeutic potential of obestatin, an autocrine anabolic factor regulating skeletal muscle repair, to ameliorate the Duchenne muscular dystrophy (DMD) phenotype. Methods and results Using a multidiscip