Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Aera Jo"'
Publikováno v:
Journal of molecular biology. 434(2)
Phosphatidylinositol 3-kinase-related protein kinases (PIKKs) play critical roles in various metabolic pathways related to cell proliferation and survival. The TELO2-TTI1-TTI2 (TTT) complex has been proposed to recognize newly synthesized PIKKs and t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6c6b8338c82952df8527f6fae4488279
https://pubmed.ncbi.nlm.nih.gov/34838521
https://pubmed.ncbi.nlm.nih.gov/34838521
Publikováno v:
J Mol Biol
The Smc5/6 complex facilitates chromosome replication and DNA break repair. Within this complex, a subcomplex composed of Nse1, Nse3 and Nse4 is thought to play multiple roles through DNA binding and regulating ATP-dependent activities of the complex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2789df4e0ef5626ae29f6e97b2d443d1
https://pubmed.ncbi.nlm.nih.gov/33676928
https://pubmed.ncbi.nlm.nih.gov/33676928
Autor:
Yaqi Liu, Ok kyu Song, Fuyang Li, Ae Kyoung Kim, Yunje Cho, Sang Eun Lee, Taeyoon Kim, Aera Jo, Gwanghyun Gwon, Youngran Kim, Sihyun Sung
Publikováno v:
The EMBO Journal. 35:743-758
ATP‐dependent DNA end recognition and nucleolytic processing are central functions of the Mre11/Rad50 (MR) complex in DNA double‐strand break repair. However, it is still unclear how ATP binding and hydrolysis primes the MR function and regulates
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ddbb32889ae281f53b5b814d32d06c72
https://doi.org/10.15252/embj.201592462
https://doi.org/10.15252/embj.201592462
Autor:
Kyeong Sik Jin, Jong-Bong Lee, Kyuwon Baek, Yunje Cho, Youngchang Kim, Aera Jo, Gwang Hyeon Gwon, Yaoyao Fu
Publikováno v:
The EMBO Journal. 33:1061-1072
The Mus81-Eme1 complex is a structure-selective endonuclease with a critical role in the resolution of recombination intermediates during DNA repair after interstrand cross-links, replication fork collapse, or double-strand breaks. To explain the mol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73fb0dc9c5f6c1ef5fae906bd5828a3e
https://doi.org/10.1002/embj.201487820
https://doi.org/10.1002/embj.201487820
Autor:
Anthony M. Carr, Aera Jo, Fenghua Yuan, Gwang Hyeon Gwon, Yanbin Zhang, Yunje Cho, Youngchang Kim, Adam T. Watson, Youngran Kim, Yaqi Liu, Thomas J. Etheridge
Fanconi anemia (FA) is an autosomal recessive genetic disorder caused by defects in any of 15 FA genes responsible for processing DNA interstrand cross-links (ICLs). The ultimate outcome of the FA pathway is resolution of cross-links, which requires
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::43ec3ff50f45b30961c207c8976dd514
http://sro.sussex.ac.uk/id/eprint/50604/4/Genes_Dev.-2014-Gwon-2276-90.pdf
http://sro.sussex.ac.uk/id/eprint/50604/4/Genes_Dev.-2014-Gwon-2276-90.pdf
Autor:
Gwang Hyeon Gwon1, Youngran Kim1, Yaqi Liu1, Watson, Adam T.2, Aera Jo1, Etheridge, Thomas J.2, Fenghua Yuan3, Yanbin Zhang3, Young Chang Kim4, Carr, Anthony M.2, Yunje Cho1 yunje@postech.ac.kr
Publikováno v:
Genes & Development. 10/15/2014, Vol. 28 Issue 20, p2276-2290. 15p.