Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Adya Saran Sinha"'
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 17 (2024)
Corticotropin-releasing hormone (CRH) neurons play an important role in the regulation of neuroendocrine responses to stress. The excitability of CRH neurons is regulated by inhibitory GABAergic inputs. However, it is unclear when GABAergic regulatio
Externí odkaz:
https://doaj.org/article/210707cbe1c4462e984146e135a8d94c
Autor:
Adya Saran Sinha, Tianying Wang, Miho Watanabe, Yasushi Hosoi, Eisei Sohara, Tenpei Akita, Shinichi Uchida, Atsuo Fukuda
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 15 (2022)
The with-no-lysine (WNK) family of serine-threonine kinases and its downstream kinases of STE20/SPS1-related proline/alanine-rich kinase (SPAK) and oxidative stress-responsive kinase-1 (OSR1) may regulate intracellular Cl− homeostasis through phosp
Externí odkaz:
https://doaj.org/article/3e1185f64de744b69d71d7b650f7dd6f
Autor:
Ruksana Yesmin, Miho Watanabe, Adya Saran Sinha, Masaru Ishibashi, Tianying Wang, Atsuo Fukuda
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 15 (2022)
The excitatory action of gamma-aminobutyric-acid (GABA) in the median-eminence (ME) led to the steady-state release of corticotropin-releasing hormone (CRH) from CRH axon terminals, which modulates the hypothalamic-pituitary-adrenal (HPA) axis. Howev
Externí odkaz:
https://doaj.org/article/d1681886b6214344b69e8ce8f78713b8
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 12 (2018)
Exposure to prenatal stress (PS) and mutations in Gad1, which encodes GABA synthesizing enzyme glutamate decarboxylase (GAD) 67, are the primary risk factors for psychiatric disorders associated with abnormalities in parvalbumin (PV)-positive GABAerg
Externí odkaz:
https://doaj.org/article/79071e270f6d4feea781a79d2056aa92
Autor:
Ruksana Yesmin, Miho Watanabe, Adya Saran Sinha, Masaru Ishibashi, Tianying Wang, Atsuo Fukuda
Publikováno v:
SSRN Electronic Journal.
Publikováno v:
Frontiers in cellular neuroscience. 12
Exposure to prenatal stress (PS) and mutations in