Zobrazeno 1 - 10
of 110
pro vyhledávání: '"Adrian I. Katz"'
Publikováno v:
Journal of Hypertension. 27:2452-2457
Because a newly described salt-inducible kinase 1 (SIK1) network is responsible for increases in active cell sodium transport in response to elevated intracellular sodium, we hypothesized that this network could mediate the effects of the mutant (hyp
Publikováno v:
British Journal of Pharmacology. 158:486-493
Background and purpose: Dopamine inhibits renal cell Na+,K+-ATPase activity and cell sodium transport by promoting the internalization of active molecules from the plasma membrane, whereas angiotensin II (ATII) stimulates its activity by recruiting n
Autor:
Hiroshi Takemori, Karin Stenström, Alejandro M. Bertorello, Adrian I. Katz, Jean Zwiller, Mattias Sjöström, Kristina Eneling
Publikováno v:
Proceedings of the National Academy of Sciences. 104:16922-16927
In mammalian cells, active sodium transport and its derived functions (e.g., plasma membrane potential) are dictated by the activity of the Na + ,K + -ATPase (NK), whose regulation is essential for maintaining cell volume and composition, as well as
Publikováno v:
Annals of the New York Academy of Sciences. 986:587-594
The Na(+),K(+)-ATPase traffics between the plasma membrane and intracellular compartments in response to acute changes in membrane receptor activation. These effects are accomplished by a time-dependent interaction of the Na(+),K(+)-ATPase alpha-subu
Autor:
Per Olof Berggren, Alejandro M. Bertorello, Guillermo A. Yudowski, Adrian I. Katz, Carlos H. Pedemonte, Riad Efendiev
Publikováno v:
Proceedings of the National Academy of Sciences. 97:6556-6561
Endocytosis of Na + ,K + -ATPase molecules in response to G protein-coupled receptor stimulation requires activation of class I A phosphoinositide-3 kinase (PI3K-I A ) in a protein kinase C-dependent manner. In this paper, we report that PI3K-I A , t