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Autor:
Torsten Wüstenberg, Susanne Erk, Linda Haddad, Sylvia Richter, Eckart D. Gundelfinger, Andreas Heinz, Phöbe Schmierer, Emrah Düzel, Stephanie H. Witt, Constanze I. Seidenbecher, Maria Garbusow, Sven Cichon, Heike Tost, Sebastian Mohnke, Marcella Rietschel, Henrik Walter, Thomas W. Mühleisen, Hartmut Schütze, Markus M. Noethen, Björn H. Schott, Oliver Grimm, Anne Assmann, Joram Soch, Andreas Meyer-Lindenberg, Lydia Pöhland, Nina Romanczuk-Seiferth, Marieke Klein, Adrian Barman
Publikováno v:
Translational Psychiatry
Translational Psychiatry 4(3), e372 (2014). doi:10.1038/tp.2014.10
Translational Psychiatry 4(3), e372-e372 (2014). doi:10.1038/tp.2014.10
Translational Psychiatry 4(3), e372 (2014). doi:10.1038/tp.2014.10
Translational Psychiatry 4(3), e372-e372 (2014). doi:10.1038/tp.2014.10
Recent genome-wide association studies have pointed to single-nucleotide polymorphisms (SNPs) in genes encoding the neuronal calcium channel CaV1.2 (CACNA1C; rs1006737) and the presynaptic active zone protein Piccolo (PCLO; rs2522833) as risk factors