Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Adrián M Ramos"'
Autor:
Sol Carriazo, Adrián M Ramos, Ana B Sanz, Maria Dolores Sanchez-Niño, Mehmet Kanbay, Alberto Ortiz
Publikováno v:
Toxins, Vol 12, Iss 3, p 151 (2020)
Multiple physiological variables change over time in a predictable and repetitive manner, guided by molecular clocks that respond to external and internal clues and are coordinated by a central clock. The kidney is the site of one of the most active
Externí odkaz:
https://doaj.org/article/1932f043838640edbe2132c0d9eaf8d0
Autor:
Alvaro C Ucero, Sergio Berzal, Carlos Ocaña-Salceda, Mónica Sancho, Mar Orzáez, Angel Messeguer, Marta Ruiz-Ortega, Jesús Egido, María J Vicent, Alberto Ortiz, Adrián M Ramos
Publikováno v:
PLoS ONE, Vol 8, Iss 1, p e51992 (2013)
The polyglutamic acid/peptoid 1 (QM56) nanoconjugate inhibits apoptosis by interfering with Apaf-1 binding to procaspase-9. We now describe anti-inflammatory properties of QM56 in mouse kidney and renal cell models.In cultured murine tubular cells, Q
Externí odkaz:
https://doaj.org/article/6f5a9a96097b45528fc4ebcc8d5f0ccc
Autor:
Priscila Villalvazo, Sol Carriazo, Jorge Rojas-Rivera, Adrián M Ramos, Alberto Ortiz, Maria Vanessa Perez-Gomez
Publikováno v:
Clinical Kidney Journal. 15:1973-1980
Systemic lupus erythematosus (SLE) is a chronic and inflammatory autoimmune disease of unknown origin that may cause kidney disease, i.e. lupus nephritis (LN). Within a wider trend towards an expanding field of genetic causes of kidney disease, two r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::46e712a1df47aff91bbac73604bc9104
https://doi.org/10.1093/ckj/sfac152
https://doi.org/10.1093/ckj/sfac152
Autor:
Gina Córdoba-David, Jorge García-Giménez, Regiane Cardoso Castelo-Branco, Susana Carrasco, Pablo Cannata, Alberto Ortiz, Adrián M. Ramos
Publikováno v:
Frontiers in pharmacology. 13
The type I interferon (TI-IFN) pathway regulates innate immunity, inflammation, and apoptosis during infection. However, the contribution of the TI-IFN pathway or upstream signaling pathways to tubular injury in kidney disease is poorly understood. U
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b0f97fc371cd69c8eef240adea9be120
https://pubmed.ncbi.nlm.nih.gov/36386242
https://pubmed.ncbi.nlm.nih.gov/36386242
Autor:
Priscila, Villalvazo, Sol, Carriazo, Jorge, Rojas-Rivera, Adrián M, Ramos, Alberto, Ortiz, Maria Vanessa, Perez-Gomez
Publikováno v:
Clinical kidney journal. 15(11)
Systemic lupus erythematosus (SLE) is a chronic and inflammatory autoimmune disease of unknown origin that may cause kidney disease, i.e. lupus nephritis (LN). Within a wider trend towards an expanding field of genetic causes of kidney disease, two r
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::4e7b1c43ff953ae5c9e2b45aa3c14410
https://pubmed.ncbi.nlm.nih.gov/36324999
https://pubmed.ncbi.nlm.nih.gov/36324999
Autor:
Cristian, González-Guerrero, José Luis, Morgado-Pascual, Pablo, Cannata-Ortiz, María Angeles, Ramos-Barron, Carlos, Gómez-Alamillo, Manuel, Arias, Sergio, Mezzano, Jesús, Egido, Marta, Ruiz-Ortega, Alberto, Ortiz, Adrián M, Ramos
Publikováno v:
The Journal of pathology. 246(2)
The chemokine CCL20 activates the CCR6 receptor and has been implicated in the pathogenesis of glomerular injury. However, it is unknown whether it contributes to acute kidney injury (AKI). We identified CCL20 as upregulated in a systems biology stra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::41dcd9c6b652308ff9b6c9cd63f38d6c
https://pubmed.ncbi.nlm.nih.gov/29984403
https://pubmed.ncbi.nlm.nih.gov/29984403
Autor:
Sergio, Berzal, Cristian, González-Guerrero, Sandra, Rayego-Mateos, Álvaro, Ucero, Carlos, Ocaña-Salceda, Jesús, Egido, Alberto, Ortiz, Marta, Ruiz-Ortega, Adrián M, Ramos
Publikováno v:
Journal of cellular physiology. 230(7)
The tubular epithelium may be intrinsically involved in promoting kidney injury by junctional instability, epithelial-mesenchymal transition (EMT) and extracellular matrix remodelling. In this work, we investigated whether the pleiotropic and proinfl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::6c748ffbd8f5026fb7579a5a74e97c47
https://pubmed.ncbi.nlm.nih.gov/25536182
https://pubmed.ncbi.nlm.nih.gov/25536182
Autor:
M Concepción, Izquierdo, Ana B, Sanz, M Dolores, Sánchez-Niño, M Vanessa, Pérez-Gómez, Marta, Ruiz-Ortega, Jonay, Poveda, Olga, Ruiz-Andrés, Adrián M, Ramos, Juan A, Moreno, Jesús, Egido, Alberto, Ortiz
Publikováno v:
Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia. 32(6)
There are no pathophysiolgical therapeutic approaches to acute kidney injury (AKI) and the mortality remains high. In addition chronic kidney disease (CKD) predisposes to AKI and AKI contributes to progression of CKD. Recently a transcriptomics appro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::90106eb0590ce5e9667a5b951d1b9675
https://pubmed.ncbi.nlm.nih.gov/23093094
https://pubmed.ncbi.nlm.nih.gov/23093094
Autor:
Carlos, Fernández, Adrián M, Ramos, Patricia, Sancho, Donna, Amrán, Elena, de Blas, Patricio, Aller
Publikováno v:
The Journal of biological chemistry. 279(5)
Arsenic trioxide (As(2)O(3)) caused apoptosis in U-937 human promonocytic cells. This effect was potentiated by the simultaneous addition of the glutathione (GSH) synthesis inhibitor DL-buthionine-(R,S)-sulfoximine or the protein kinase C activators
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a700624bfec0cd6eed0d9bc0645de30e
https://pubmed.ncbi.nlm.nih.gov/14610070
https://pubmed.ncbi.nlm.nih.gov/14610070