Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Adrián M Ramos"'
Autor:
Gina Córdoba-David, Jorge García-Giménez, Regiane Cardoso Castelo-Branco, Susana Carrasco, Pablo Cannata, Alberto Ortiz, Adrián M. Ramos
Publikováno v:
Frontiers in Pharmacology, Vol 13 (2022)
The type I interferon (TI-IFN) pathway regulates innate immunity, inflammation, and apoptosis during infection. However, the contribution of the TI-IFN pathway or upstream signaling pathways to tubular injury in kidney disease is poorly understood. U
Externí odkaz:
https://doaj.org/article/d9d07cab29264c209ddd190484ee81c1
Autor:
Priscila Villalvazo, Sol Carriazo, Jorge Rojas-Rivera, Adrián M Ramos, Alberto Ortiz, Maria Vanessa Perez-Gomez
Publikováno v:
Clinical Kidney Journal. 15:1973-1980
Systemic lupus erythematosus (SLE) is a chronic and inflammatory autoimmune disease of unknown origin that may cause kidney disease, i.e. lupus nephritis (LN). Within a wider trend towards an expanding field of genetic causes of kidney disease, two r
Autor:
Sol Carriazo, Adrián M Ramos, Ana B Sanz, Maria Dolores Sanchez-Niño, Mehmet Kanbay, Alberto Ortiz
Publikováno v:
Toxins, Vol 12, Iss 3, p 151 (2020)
Multiple physiological variables change over time in a predictable and repetitive manner, guided by molecular clocks that respond to external and internal clues and are coordinated by a central clock. The kidney is the site of one of the most active
Externí odkaz:
https://doaj.org/article/1932f043838640edbe2132c0d9eaf8d0
Autor:
Priscila, Villalvazo, Sol, Carriazo, Jorge, Rojas-Rivera, Adrián M, Ramos, Alberto, Ortiz, Maria Vanessa, Perez-Gomez
Publikováno v:
Clinical kidney journal. 15(11)
Systemic lupus erythematosus (SLE) is a chronic and inflammatory autoimmune disease of unknown origin that may cause kidney disease, i.e. lupus nephritis (LN). Within a wider trend towards an expanding field of genetic causes of kidney disease, two r
Autor:
Alvaro C Ucero, Sergio Berzal, Carlos Ocaña-Salceda, Mónica Sancho, Mar Orzáez, Angel Messeguer, Marta Ruiz-Ortega, Jesús Egido, María J Vicent, Alberto Ortiz, Adrián M Ramos
Publikováno v:
PLoS ONE, Vol 8, Iss 1, p e51992 (2013)
The polyglutamic acid/peptoid 1 (QM56) nanoconjugate inhibits apoptosis by interfering with Apaf-1 binding to procaspase-9. We now describe anti-inflammatory properties of QM56 in mouse kidney and renal cell models.In cultured murine tubular cells, Q
Externí odkaz:
https://doaj.org/article/6f5a9a96097b45528fc4ebcc8d5f0ccc
Autor:
Cristian, González-Guerrero, José Luis, Morgado-Pascual, Pablo, Cannata-Ortiz, María Angeles, Ramos-Barron, Carlos, Gómez-Alamillo, Manuel, Arias, Sergio, Mezzano, Jesús, Egido, Marta, Ruiz-Ortega, Alberto, Ortiz, Adrián M, Ramos
Publikováno v:
The Journal of pathology. 246(2)
The chemokine CCL20 activates the CCR6 receptor and has been implicated in the pathogenesis of glomerular injury. However, it is unknown whether it contributes to acute kidney injury (AKI). We identified CCL20 as upregulated in a systems biology stra
Autor:
Sergio, Berzal, Cristian, González-Guerrero, Sandra, Rayego-Mateos, Álvaro, Ucero, Carlos, Ocaña-Salceda, Jesús, Egido, Alberto, Ortiz, Marta, Ruiz-Ortega, Adrián M, Ramos
Publikováno v:
Journal of cellular physiology. 230(7)
The tubular epithelium may be intrinsically involved in promoting kidney injury by junctional instability, epithelial-mesenchymal transition (EMT) and extracellular matrix remodelling. In this work, we investigated whether the pleiotropic and proinfl
Autor:
M Concepción, Izquierdo, Ana B, Sanz, M Dolores, Sánchez-Niño, M Vanessa, Pérez-Gómez, Marta, Ruiz-Ortega, Jonay, Poveda, Olga, Ruiz-Andrés, Adrián M, Ramos, Juan A, Moreno, Jesús, Egido, Alberto, Ortiz
Publikováno v:
Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia. 32(6)
There are no pathophysiolgical therapeutic approaches to acute kidney injury (AKI) and the mortality remains high. In addition chronic kidney disease (CKD) predisposes to AKI and AKI contributes to progression of CKD. Recently a transcriptomics appro
Autor:
Carlos, Fernández, Adrián M, Ramos, Patricia, Sancho, Donna, Amrán, Elena, de Blas, Patricio, Aller
Publikováno v:
The Journal of biological chemistry. 279(5)
Arsenic trioxide (As(2)O(3)) caused apoptosis in U-937 human promonocytic cells. This effect was potentiated by the simultaneous addition of the glutathione (GSH) synthesis inhibitor DL-buthionine-(R,S)-sulfoximine or the protein kinase C activators
Autor:
Villalvazo, Priscila1 (AUTHOR), Carriazo, Sol1,2,3 (AUTHOR), Rojas-Rivera, Jorge1,2 (AUTHOR), Ramos, Adrián M1,4 (AUTHOR), Ortiz, Alberto1,3,4 (AUTHOR), Perez-Gomez, Maria Vanessa1,3,4 (AUTHOR) mvanessa@fjd.es
Publikováno v:
Clinical Kidney Journal. Nov2022, Vol. 15 Issue 11, p1973-1980. 8p.