Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Adina Stanculescu"'
Autor:
Adina Stanculescu, Chin-Yo Lin, Lance D. Miller, Yang Dai, Madhumita Pradhan, Aisha Weaver, Jonna Frasor
Supplementary Table 1 from Positive Cross-Talk between Estrogen Receptor and NF-κB in Breast Cancer
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13e886b514b7bcd0be8602838cf8e17a
https://doi.org/10.1158/0008-5472.22383266
https://doi.org/10.1158/0008-5472.22383266
Autor:
Adina Stanculescu, Chin-Yo Lin, Lance D. Miller, Yang Dai, Madhumita Pradhan, Aisha Weaver, Jonna Frasor
Estrogen receptors (ER) and nuclear factor-κB (NF-κB) are known to play important roles in breast cancer, but these factors are generally thought to repress each other's activity. However, we have recently found that ER and NF-κB can also act toge
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f633532738da3e0bfc905725dc59047b
https://doi.org/10.1158/0008-5472.c.6500480
https://doi.org/10.1158/0008-5472.c.6500480
Autor:
Mary Caskey, Ingrid Espinoza, Damian G. Romero, Christian R. Gomez, Adina Stanculescu, Paola Rizzo, Antonio Pannuti, Hamid Band, Barbara A. Osborne, Lucio Miele, Todd E. Golde, Maria Laura Avantaggiati, Keon Wook Kang, Luis A. Martinez, Vimla Band, Maurizio Bocchetta, Jieun Yun, Song Kyu Park, Hwan Mook Kim
Publikováno v:
Journal of Cellular Physiology. 230:3115-3127
p53 and Notch-1 play important roles in breast cancer biology. Notch-1 inhibits p53 activity in cervical and breast cancer cells. Conversely, p53 inhibits Notch activity in T-cells but stimulates it in human keratinocytes. Notch co-activator MAML1 bi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9356bee80ce13b1e18acd5cb4b6ca2b9
https://doi.org/10.1002/jcp.25052
https://doi.org/10.1002/jcp.25052
Autor:
Adina Stanculescu, Kristina Borgen, Jonna Frasor, Elizabeth L. Wiley, Leslie A. Bembinster, Anna Bergamaschi
Publikováno v:
Hormones and Cancer. 1:127-135
The estrogen receptor (ER) is a major prognostic and therapeutic marker that is expressed in nearly 75% of breast tumors. We have previously shown that the presence of inflammatory mediators can alter the genomic function of the estrogen receptor (ER
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b3188459a6dccdb21fc77deff4c54adb
https://doi.org/10.1007/s12672-010-0018-6
https://doi.org/10.1007/s12672-010-0018-6
Autor:
Aisha E. Weaver, Lance D. Miller, Yang Dai, Chin-Yo Lin, Madhumita Pradhan, Jonna Frasor, Adina Stanculescu
Publikováno v:
Cancer Research. 69:8918-8925
Estrogen receptors (ER) and nuclear factor-kappaB (NF-kappaB) are known to play important roles in breast cancer, but these factors are generally thought to repress each other's activity. However, we have recently found that ER and NF-kappaB can also
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a824fba017a8dd0317fc2ccc032858cb
https://doi.org/10.1158/0008-5472.can-09-2608
https://doi.org/10.1158/0008-5472.can-09-2608
Autor:
Aharon Solomon, Ishfaq Bhat, Yongtong Zhao, David E. Wazer, Hui Ma, James D. Griffin, Honey V. Reddi, Goberdhan P. Dimri, Laurie M. Delmolino, Channabasavaiah B. Gurumurthy, Ying Zhang, Qingshen Gao, Mu Sheng Zeng, Aisha Kung, Vimla Band, Adina Stanculescu, Lizi Wu, Aleksandra Germaniuk-Kurowska, Hamid Band, Lucio Miele, Rebecca B. Katzman
Publikováno v:
Journal of Biological Chemistry. 282:11969-11981
Members of the evolutionarily conserved Mastermind (MAM) protein family, including the three related mammalian Mastermind-like (MAML) proteins MAML1-3, function as crucial coactivators of Notch-mediated transcriptional activation. Given the recent ev
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::65fe4ec387473e528e8b1b674002daf2
https://doi.org/10.1074/jbc.m608974200
https://doi.org/10.1074/jbc.m608974200
Autor:
Jieun, Yun, Ingrid, Espinoza, Antonio, Pannuti, Damian, Romero, Luis, Martinez, Mary, Caskey, Adina, Stanculescu, Maurizio, Bocchetta, Paola, Rizzo, Vimla, Band, Hamid, Band, Hwan Mook, Kim, Song-Kyu, Park, Keon Wook, Kang, Maria Laura, Avantaggiati, Christian R, Gomez, Todd, Golde, Barbara, Osborne, Lucio, Miele
Publikováno v:
Journal of cellular physiology. 230(12)
p53 and Notch-1 play important roles in breast cancer biology. Notch-1 inhibits p53 activity in cervical and breast cancer cells. Conversely, p53 inhibits Notch activity in T-cells but stimulates it in human keratinocytes. Notch co-activator MAML1 bi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::dc0813351795fb8a354204a12f3265d6
https://pubmed.ncbi.nlm.nih.gov/26033683
https://pubmed.ncbi.nlm.nih.gov/26033683
Autor:
Anna Bergamaschi, Adina Stanculescu, Benita S. Katzenellenbogen, Jonna Frasor, Kristina Borgen, Kendrith M. Rowland, Patricia A. Johnson, Elizabeth L. Wiley
The 14-3-3ζ gene, on 8q22, is often amplified in breast cancer and encodes a survival factor that interacts with and stabilizes many key signaling proteins. We examined the relationship between the expression of 14-3-3ζ, estrogen receptor α (ERα)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::33eb9fb63eb96ea00f8861965b8c128e
https://europepmc.org/articles/PMC3632437/
https://europepmc.org/articles/PMC3632437/
Failed implantation is a major limiting factor in infertility and early pregnancy loss. In primates, human chorionic gonadotropin mediated inhibition of stromal cell apoptosis and their subsequent differentiation into decidual cells is critical for s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b27c20e97997cc782a45d889fe1ae550
https://europepmc.org/articles/PMC3455005/
https://europepmc.org/articles/PMC3455005/
Autor:
Anna Bergamaschi, Benita S. Katzenellenbogen, Kendrith M. Rowland, Jonna Frasor, Kristina Borgen, Patricia A. Johnson, Adina Stanculescu, Elizabeth L. Wiley
Publikováno v:
Cancer Research. 72:434-434
Introduction: The 14-3-3α gene, on 8q22, is often amplified in breast cancer. It encodes a survival factor that interacts with many key signaling proteins, thereby increasing cell proliferation and invasiveness, and contributing to therapy resistanc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::20fb1b2ed2035133d0f8037cbac0e5ce
https://doi.org/10.1158/1538-7445.am2012-434
https://doi.org/10.1158/1538-7445.am2012-434