Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Adam Belcher"'
Autor:
Shubash Adhikari, MD, MS, FACP, Adam Belcher, PhD, Frank Annie, PhD, Muhammad Amer, MD, FACP, FACC, FHRS
Publikováno v:
CJC Open, Vol 6, Iss 2, Pp 104-107 (2024)
Externí odkaz:
https://doaj.org/article/a0fdd2a5bb6e4392b392ab50ed2c4dd6
Publikováno v:
Cardiovascular Revascularization Medicine.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::eccab04ea21966b97b4b3aa1b0cd2acf
https://doi.org/10.1016/j.carrev.2023.05.009
https://doi.org/10.1016/j.carrev.2023.05.009
Publikováno v:
Arterioscler Thromb Vasc Biol
Objective: Current antiplatelet medications increase the risk of bleeding, which leads to a clear clinical need in developing novel mechanism-based antiplatelet drugs. TYMP (Thymidine phosphorylase), a cytoplasm protein that is highly expressed in pl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f6a391519934e8649f74b0b2617b62b1
https://doi.org/10.1161/atvbaha.120.315109
https://doi.org/10.1161/atvbaha.120.315109
Publikováno v:
Diabetes. 70
Obesity is a major independent risk factor for the development of type II diabetes (T2DM), nonalcoholic fatty liver disease (NAFLD), and cardiovascular disease (CVD). Consequently, controlling obesity constitutes a key strategy for reducing the preva
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7bf5a4796245e23c44b12f739825e497
https://doi.org/10.2337/db21-1211-p
https://doi.org/10.2337/db21-1211-p
RationaleMost of the current anti-platelet drugs inhibit platelet function permanently and have systemic side effects, including thrombocytopenia and hemorrhage. We previously found that thymidine phosphorylase (TYMP), a platelet cytoplasmic protein,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c480e4b3b3ead32be1d64f95cbc40d4a
Autor:
Yueheng Wu, Roy L. Silverstein, Philip A. Klenotic, Amira F. Gohara, Oliver Li, Hong Yue, Adam Belcher, Shaoxian Chen, Bin Kuang, Maria Febbraio, Wei Li, David J. Kennedy, Thomas M. McIntyre
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology. 39(2)
Objective— Dysregulated proliferation of vascular smooth muscle cells (VSMC) plays an essential role in neointimal hyperplasia. CD36 functions critically in atherogenesis and thrombosis. We hypothesize that CD36 regulates VSMC proliferation and con
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0e5d8343a2bee1a068581d18ce888c50
https://pubmed.ncbi.nlm.nih.gov/30567481
https://pubmed.ncbi.nlm.nih.gov/30567481