Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Ada Kawecka"'
Autor:
Alicja Braczko, Gabriela Harasim, Ada Kawecka, Iga Walczak, Małgorzata Kapusta, Magdalena Narajczyk, Klaudia Stawarska, Ryszard T. Smoleński, Barbara Kutryb-Zając
Publikováno v:
Frontiers in Physiology, Vol 14 (2023)
Background: Statins and proprotein convertase subtilisin/kexin type 9 inhibitors (PCSK9i) are cornerstones of therapy to prevent cardiovascular disease, acting by lowering lipid concentrations and only partially identified pleiotropic effects. This s
Externí odkaz:
https://doaj.org/article/f4e70ca59ea24d348eb8ab297afb9518
Autor:
Barbara Kutryb-Zając, Ada Kawecka, Khrystyna Nasadiuk, Alicja Braczko, Klaudia Stawarska, Elisabetta Caiazzo, Patrycja Koszałka, Carla Cicala
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 165, Iss , Pp 115184- (2023)
Adenosine is an endogenous nucleoside that regulates many physiological and pathological processes. It is derived from either the intracellular or extracellular dephosphorylation of adenosine triphosphate and interacts with cell-surface G-protein-cou
Externí odkaz:
https://doaj.org/article/738b3dd0f90a46bbad51ed4bdf06bbc4
Autor:
Barbara Kutryb-Zajac, Ada Kawecka, Fionä Caratis, Krzysztof Urbanowicz, Alicja Braczko, Tomomi Furihata, Bartosz Karaszewski, Ryszard T. Smolenski, Aleksandra Rutkowska
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 15 (2022)
BackgroundAdenosine deaminase (ADA) via two isoenzymes, ADA1 and ADA2, regulates intra- and extracellular adenosine concentrations by converting it to inosine. In the central nervous system (CNS), adenosine modulates the processes of neuroinflammatio
Externí odkaz:
https://doaj.org/article/a7c4d3fc01674f688619ce6a937392de
Autor:
Barbara Kutryb-Zajac, Ada Kawecka, Alicja Braczko, Marika Franczak, Ewa M. Slominska, Roberto Giovannoni, Ryszard T. Smolenski
Publikováno v:
Biomedicines, Vol 10, Iss 7, p 1540 (2022)
Chronic hypoxia drives vascular dysfunction by various mechanisms, including changes in mitochondrial respiration. Although endothelial cells (ECs) rely predominantly on glycolysis, hypoxia is known to alter oxidative phosphorylation, promote oxidati
Externí odkaz:
https://doaj.org/article/f1ce3c754dac4508a64e0ab3cf00dab9
Autor:
Patrycja Jablonska, Paulina Mierzejewska, Marta Tomczyk, Patrycja Koszalka, Marika Franczak, Ada Kawecka, Barbara Kutryb-Zajac, Alicja Braczko, Ryszard T. Smolenski, Ewa M. Slominska
Publikováno v:
Biology, Vol 11, Iss 5, p 675 (2022)
The disruption of the metabolism of extracellular NAD+ and NMN may affect related signaling cascades and pathologies, such as cardiovascular or respiratory system diseases. We aimed to study NAD+ and NMN hydrolysis on surface endothelial cells of div
Externí odkaz:
https://doaj.org/article/57b5eb8fb0a64c7ba25decd71c3be461
Autor:
Klaudia Stawarska, Alicja Braczko, Ada Kawecka, Agata Jedrzejewska, Marzena Romanowska-Kocejko, Milena Deptula, Malgorzata Zawrzykraj, Marika Franczak, Oliwia Krol, Gabriela Harasim, Michal Pikula, Marcin Hellmann, Barbara Kutryb-Zajac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5cb0ad84b8e12fe7bd5e911e5c5b3ec2
https://doi.org/10.2139/ssrn.4443003
https://doi.org/10.2139/ssrn.4443003
Autor:
Agata Jedrzejewska, Alicja Braczko, Ada Kawecka, Marcin Hellmann, Piotr Siondalski, Ewa Slominska, Barbara Kutryb-Zajac, Magdi H. Yacoub, Ryszard T. Smolenski
Publikováno v:
International journal of molecular sciences. 23(17)
LVAD therapy is an effective rescue in acute and especially chronic cardiac failure. In several scenarios, it provides a platform for regeneration and sustained myocardial recovery. While unloading seems to be a key element, pharmacotherapy may provi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::25b6f68fba89edbece5b977241a78ae6
https://pubmed.ncbi.nlm.nih.gov/36077285
https://pubmed.ncbi.nlm.nih.gov/36077285
Autor:
Barbara, Kutryb-Zajac, Ada, Kawecka, Alicja, Braczko, Marika, Franczak, Ewa M, Slominska, Roberto, Giovannoni, Ryszard T, Smolenski
Publikováno v:
Biomedicines. 10(7)
Chronic hypoxia drives vascular dysfunction by various mechanisms, including changes in mitochondrial respiration. Although endothelial cells (ECs) rely predominantly on glycolysis, hypoxia is known to alter oxidative phosphorylation, promote oxidati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::6890d7419106d5d5ae3ebba9a7cab532
https://pubmed.ncbi.nlm.nih.gov/35884844
https://pubmed.ncbi.nlm.nih.gov/35884844