Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Aboud, Lina"'
Autor:
Reading, N. Scott, Sirdah, Mahmoud M., Shubair, Mohammad E., Nelson, Benjamin E., Al-Kahlout, Mustafa S., Al-Tayeb, Jamal M., Aboud, Lina N., Shaban, Maysaa Abu, Luzzatto, Lucio, Prchal, Josef T.
Publikováno v:
In Blood Cells, Molecules and Diseases September 2016 60:58-64
Autor:
Sirdah, Mahmoud, Reading, N. Scott, Vankayalapati, Hariprasad, Perkins, Sherrie L., Shubair, Mohammad E., Aboud, Lina, Roper, David, Prchal, Josef T.
Publikováno v:
In Blood Cells, Molecules and Diseases 15 October-15 December 2012 49(3-4):152-158
Autor:
Sirdah, Mahmoud, Reading, N. Scott, Perkins, Sherrie L., Shubair, Mohammad, Aboud, Lina, Prchal, Josef T.
Publikováno v:
In Blood Cells, Molecules and Diseases 15 April 2012 48(4):203-208
Autor:
Reading, N Scott, Reading, Noel S, Sirdah, Mahmoud, Perkins, Sherrie L, Wilson, Andrew R, Shubair, Mohammad E., Aboud, Lina, Prchal, Josef T
Glucose-6-phosphate dehydrogenase (G6PD) deficiency, affecting more than 500 million people worldwide, is one of the most common inherited red blood cell disorders. There are 186 G6PD mutations published, with some that cluster within defined ethnic/
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______4294::4f2449bcb5dfc436e3ee636ed284bb7a
https://www.researchgate.net/profile/Mahmoud_Sirdah/publication/272492428_G6PD_Deficient_Variants_Differ_in_Risk_of_Hospitalization_Among_Gaza_Strip_Palestinian_Children/links/54e645450cf277664ff4de24.pdf
https://www.researchgate.net/profile/Mahmoud_Sirdah/publication/272492428_G6PD_Deficient_Variants_Differ_in_Risk_of_Hospitalization_Among_Gaza_Strip_Palestinian_Children/links/54e645450cf277664ff4de24.pdf
Autor:
Reading, N Scott, Sirdah, Mahmoud, Shubair, Mohammad E., Nelson, Benjamin E, Al-Kahlout, Mustafa S, Al-Tayeb, Jamal M, Aboud, Lina, Abu Shaban, Maysaa, Luzzatto, Lucio, Prchal, Josef T
Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a common genetic abnormality known to predispose to acute hemolytic anemia (AHA), which can be triggered by certain drugs or infection. However, the commonest trigger is fava beans (Vicia faba) i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______4294::134ef7214dfa54721653a0bb25d2b137
https://www.sciencedirect.com/science/article/pii/S1079979616300821
https://www.sciencedirect.com/science/article/pii/S1079979616300821
Autor:
Reading, N Scott, Sirdah, Mahmoud, Shubair, Mohammad E., Nelson, Benjamin E, Al-Kahlout, Mustafa S, Al-Tayeb, Jamal M, Aboud, Lina, Abu Shaban, Maysaa, Luzzatto, Lucio, Prchal, Josef T
Conclusion: We conclude that children with G6PD A-deficiency are also susceptible to AHA, but demonstrate in direct comparison within this same population that G6PD Mediterranean and G6PD Cairo are more severe forms of deficiency than G6PD A‐. Furt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f2a981834834e0fa698017f232575aea
Background The G6PD c.563 C > T deficient mutation is endemic among Mediterranean populations but its clinical significance is not well delineated. We set up to estimate the proportion of G6PD deficient children presenting with hemolytic anemia at Al
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______4294::63d4dd89f9a3d593ea34e9a50ce3878b
https://www.sciencedirect.com/science/article/pii/S1079979612000307
https://www.sciencedirect.com/science/article/pii/S1079979612000307
Autor:
Aboud, Lina Nimer
Background: The Glucose-6-phosphate dehydrogenase (G6PD) deficiency has been considered as the commonest enzymopathic inherited disorder of red blood cells, which affecting more than 500 million people worldwide. The G6PD gene in human is X-linked so
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______4294::000b137a177545fb90ff99521b2816f2
Autor:
Reading, Noel S. *, Sirdah, Mahmoud M. *, Perkins, Sherrie L., Wilson, Andrew R. *, Shubair, Mohammad E. *, Aboud, Lina *, Prchal, Josef T.
Publikováno v:
In Blood 16 November 2012 120(21):4268-4268