Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Abdulruhman S. Al-Mulhim"'
Publikováno v:
Zeitschrift für Naturforschung C. 70:175-181
Oxidative/nitrosative stress, inflammation, and apoptosis play a crucial role in the pathogenesis of arsenic-induced testicular injury. Telmisartan, the angiotensin II-receptor antagonist, possesses antioxidant and anti-inflammatory activities. The p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5ad019f2a7325b92c458f889cdce5cb
https://doi.org/10.1515/znc-2015-5031
https://doi.org/10.1515/znc-2015-5031
Publikováno v:
Journal of Trace Elements in Medicine and Biology. 27:355-363
The protective effect of cannabidiol, the non-psychoactive component of Cannabis sativa, against liver toxicity induced by a single dose of cadmium chloride (6.5 mgkg(-1) i.p.) was investigated in rats. Cannabidiol treatment (5 mgkg(-1)/day, i.p.) wa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8be2d963d722d390fa5b8e58d246f63d
https://doi.org/10.1016/j.jtemb.2013.07.001
https://doi.org/10.1016/j.jtemb.2013.07.001
Publikováno v:
Environmental Toxicology and Pharmacology. 36:347-357
The potential protective effect of cannabidiol, the major non-psychotropic Cannabis constituent, was investigated against doxorubicin cardiotoxicity in rats. Cardiotoxicity was induced by six equal doses of doxorubicin (2.5mgkg(-1) i.p., each) given
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1d51f81653751488767335da7f1c121c
https://doi.org/10.1016/j.etap.2013.04.018
https://doi.org/10.1016/j.etap.2013.04.018
Publikováno v:
Environmental Toxicology and Pharmacology. 35:100-108
The therapeutic potential of coenzyme Q10 was investigated in rats with hepatocellular carcinoma induced by trichloroacetic acid (0.5g/kg/day, p.o., for five days). Coenzyme Q10 treatment (0.4mg/kg/day, i.p.) was applied for four weeks following tric
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dd4b2cf439842dafdf2f2a699436b89a
https://doi.org/10.1016/j.etap.2012.11.016
https://doi.org/10.1016/j.etap.2012.11.016
Publikováno v:
European Journal of Pharmacology. 693:64-71
The therapeutic potential of telmisartan was investigated in mice exposed to acute hepatotoxicity induced by a single dose of acetaminophen (500 mg/kg, p.o.). Telmisartan treatment (two i.p. injections, 10mg/kg, each) was given at 1 and 12h following
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c28d7dbe14c4807190d866981855891c
https://doi.org/10.1016/j.ejphar.2012.07.049
https://doi.org/10.1016/j.ejphar.2012.07.049
Publikováno v:
Journal of Pharmacy and Pharmacology. 65:243-252
Objectives To investigate the potential protective effects of captopril, the angiotensin-converting enzyme inhibitor, in diabetic rats exposed to ischaemia/reperfusion (I/R) renal injury. Methods Following successful induction of diabetes, captopril
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c9c808bd496fe454e5325ceb56249681
https://doi.org/10.1111/j.2042-7158.2012.01585.x
https://doi.org/10.1111/j.2042-7158.2012.01585.x
Publikováno v:
Toxicology. 300(3)
The protective effect of telmisartan, the angiotensin II-receptor antagonist, against liver toxicity induced by sodium arsenite (5 mg/kg/day, p.o., for 30 days) was investigated in mice. Telmisartan treatment (10 mg/kg/day, p.o.) was applied for 30 d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5f8d45324be2d380f000c94a41cc0b9f
https://pubmed.ncbi.nlm.nih.gov/22750630
https://pubmed.ncbi.nlm.nih.gov/22750630
Publikováno v:
Life sciences. 91(7-8)
Aims To investigate the protective effect of cannabidiol, the major non-psychotropic Cannabis constituent, against renal ischemia/reperfusion injury in rats. Main methods Bilateral renal ischemia was induced for 30 min followed by reperfusion for 24
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::12c5672d8632e7195ba013fb216441fe
https://pubmed.ncbi.nlm.nih.gov/22877651
https://pubmed.ncbi.nlm.nih.gov/22877651