Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Abdullah Onul"'
Autor:
G. K. Haines, James A. Radosevich, Benjamin J. Vesper, Abdullah Onul, Michael B. Altman, Kim M. Elseth, John C. Roeske, William A. Paradise, Gabor Tarjan, H. De Vitto, Bulent Aydogan, Jiaping Xue
Publikováno v:
Tumor Biology. 34:203-214
There is a lack of understanding of the casual mechanisms behind the observation that some breast adenocarcinomas have identical morphology and comparatively different cellular growth behavior. This is exemplified by a differential response to radiat
Autor:
Benjamin J. Vesper, Eftychia Gouvas, Michael D. Colvard, James A. Radosevich, Kim M. Elseth, Zane Deliu, Abdullah Onul, William A. Paradise, Kelly D. Garcia, William J. Pestle
Publikováno v:
Journal of Histochemistry & Cytochemistry. 60:683-693
Electrocautery and directed energy devices (DEDs) such as lasers, which are used in surgery, result in tissue damage that cannot be readily detected by traditional histological methods, such as hematoxylin and eosin staining. Alternative staining met
Autor:
Jiaping Xue, B. S. Mendonça, William A. Paradise, Claudia Vitoria de Moura Gallo, Kim M. Elseth, Abdullah Onul, Benjamin J. Vesper, H. De Vitto, Franklin David Rumjanek, James A. Radosevich
Publikováno v:
Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine. 34(1)
Nutrient deprivation and reactive oxygen species (ROS) play an important role in breast cancer mitochondrial adaptation. Adaptations to these conditions allow cells to survive in the stressful microenvironment of the tumor bed. This study is directed
Autor:
Abdullah Onul, James A. Radosevich, Gabor Tarjan, Humberto De Vitto, Kim M. Elseth, G. Kenneth Haines, Franklin David Rumjanek, William A. Paradise, Benjamin J. Vesper
Publikováno v:
Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine. 33(3)
Previously, we demonstrated that A549, a human lung cancer cell line, could be adapted to the free radical nitric oxide (NO •). NO • is known to be over expressed in human tumors. The original cell line, A549 (parent), and the newly adapted A549-