Zobrazeno 1 - 10
of 73
pro vyhledávání: '"Abdallah Gharib"'
Autor:
Yves Gouriou, Muhammad Rizwan Alam, Zeina Harhous, Claire Crola Da Silva, Delphine Baetz, Sally Badawi, Etienne Lefai, Jennifer Rieusset, Annie Durand, Rania Harisseh, Abdallah Gharib, Michel Ovize, Gabriel Bidaux
Publikováno v:
Cells, Vol 10, Iss 9, p 2171 (2021)
The authors and the Cells Editorial Office would like to add the section “Materials and Methods”, which was missing in the original version [...]
Externí odkaz:
https://doaj.org/article/b28244c6b6374f06b0da095018c713f5
Autor:
Camille Loubiere, Stephan Clavel, Jerome Gilleron, Rania Harisseh, Jeremy Fauconnier, Issam Ben-Sahra, Lisa Kaminski, Kathiane Laurent, Stephanie Herkenne, Sandra Lacas-Gervais, Damien Ambrosetti, Damien Alcor, Stephane Rocchi, Mireille Cormont, Jean-François Michiels, Bernard Mari, Nathalie M. Mazure, Luca Scorrano, Alain Lacampagne, Abdallah Gharib, Jean-François Tanti, Frederic Bost
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-9 (2017)
Abstract Mitochondrial integrity is critical for the regulation of cellular energy and apoptosis. Metformin is an energy disruptor targeting complex I of the respiratory chain. We demonstrate that metformin induces endoplasmic reticulum (ER) stress,
Externí odkaz:
https://doaj.org/article/3c81dba573894f44b340dafd88a5e78b
Autor:
Sophie Le Page, Marjorie Niro, Jérémy Fauconnier, Laura Cellier, Sophie Tamareille, Abdallah Gharib, Arnaud Chevrollier, Laurent Loufrani, Céline Grenier, Rima Kamel, Emmanuelle Sarzi, Alain Lacampagne, Michel Ovize, Daniel Henrion, Pascal Reynier, Guy Lenaers, Delphine Mirebeau-Prunier, Fabrice Prunier
Publikováno v:
PLoS ONE, Vol 11, Iss 10, p e0164066 (2016)
Recent data suggests the involvement of mitochondrial dynamics in cardiac ischemia/reperfusion (I/R) injuries. Whilst excessive mitochondrial fission has been described as detrimental, the role of fusion proteins in this context remains uncertain.To
Externí odkaz:
https://doaj.org/article/9a94a432c8f143a69675a6b6f979acba
Publikováno v:
Neurobiology of Disease, Vol 16, Iss 2, Pp 291-299 (2004)
Sleep–wake homeostasis is crucial for behavioral performances and memory both in the general population and in patients with learning disability, among whom were Down syndrome (DS) patients. We investigated, in mouse models of DS, cortical EEG and
Externí odkaz:
https://doaj.org/article/febf06fe4e1141f68b767b090b7f5d33
Publikováno v:
Anais dos Seminários de Iniciação Científica.
Assim como a cegueira, o amor é também um tema amplamente discutido e re-visitado. Enquanto sobre a cegueira “as representações comuns de pessoas com defici-ência reforçam as conotações majoritariamente negativas associadas às deficiência
Autor:
Rania Harisseh, Michel Ovize, Sally Badawi, Zeina Harhous, Muhammad Rizwan Alam, Abdallah Gharib, Annie Durand, Delphine Baetz, Gabriel Bidaux, Etienne Lefai, Yves Gouriou, Jennifer Rieusset, Claire Crola Da Silva
Publikováno v:
Cells
Cells, Vol 10, Iss 2171, p 2171 (2021)
Cells, Vol 10, Iss 2171, p 2171 (2021)
The authors and the Cells Editorial Office would like to add the section “Materials and Methods”, which was missing in the original version [...]
Autor:
Pauline Sueur, Rania Harisseh, Michel Ovize, Pascal Chiari, Abdallah Gharib, Maryline Abrial, Camille Villedieu, Jean-Luc Fellahi
Publikováno v:
Journal of Cardiovascular Pharmacology
12. International Symposium for Mechanisms of Vasodilatation
12. International Symposium for Mechanisms of Vasodilatation, Nov 2016, Rochester, United States. ⟨10.1097/FJC.0000000000000479⟩
12. International Symposium for Mechanisms of Vasodilatation, Nov 2016, Rochester United States. ⟨10.1097/FJC.0000000000000479⟩
12. International Symposium for Mechanisms of Vasodilatation
12. International Symposium for Mechanisms of Vasodilatation, Nov 2016, Rochester, United States. ⟨10.1097/FJC.0000000000000479⟩
12. International Symposium for Mechanisms of Vasodilatation, Nov 2016, Rochester United States. ⟨10.1097/FJC.0000000000000479⟩
Background: Volatile anesthetics are known to limit myocardial ischemia-reperfusion injuries. Mitochondria were shown to be major contributors to cardioprotection. Cyclophilin D (CypD) is one of the main regulators of mitochondria-induced cell death.
Autor:
Rania, Harisseh, Maryline, Abrial, Pascal, Chiari, Ribal, Al-Mawla, Camille, Villedieu, Nolwenn, Tessier, Gabriel, Bidaux, Michel, Ovize, Abdallah, Gharib
Publikováno v:
J Biol Chem
Calcium homeostasis is essential for cell survival and is precisely controlled by several cellular actors such as the sarco/endoplasmic reticulum and mitochondria. Upon stress induction, Ca(2+) released from sarco/endoplasmic reticulum stores and fro
Autor:
Abdallah Gharib, Annie Varennes, Thomas Bochaton, Vincent Jahandiez, Laurent Argaud, Martin Cour, Michel Ovize, Joseph Loufouat, Maryline Abrial
Publikováno v:
Basic Research in Cardiology
Basic Research in Cardiology, Springer Verlag, 2017, 112 (4), ⟨10.1007/s00395-017-0624-3⟩
Basic Research in Cardiology, Springer Verlag, 2017, 112 (4), ⟨10.1007/s00395-017-0624-3⟩
The opening of the mitochondrial permeability transition pore (PTP), which is regulated by the matrix protein cyclophilin D (CypD), plays a key role in the pathophysiology of post-cardiac arrest (CA) syndrome. We hypothesized that therapeutic hypothe
Autor:
Claire Crola Da Silva, Thomas Delale, Lionel Augeul, Abdallah Gharib, Bruno Pillot, Michel Ovize, Rania Harisseh, Noelle Gallo-Bona, Joseph Loufouat, Thierry Abribat, Soraya Allas, René Ferrera
Publikováno v:
Basic Research in Cardiology
Basic Research in Cardiology, Springer Verlag, 2017, 112 (1), ⟨10.1007/s00395-016-0595-9⟩
Basic Research in Cardiology, Springer Verlag, 2017, 112 (1), ⟨10.1007/s00395-016-0595-9⟩
Reperfusion injury is responsible for an important part of myocardial infarct establishment due notably to triggering cardiomyocytes death at the first minutes of reperfusion. AZP-531 is an optimized analog of unacylated ghrelin currently in clinical
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a79ec9b3314224f504decf23e5d9b397
https://hal.archives-ouvertes.fr/hal-01607428
https://hal.archives-ouvertes.fr/hal-01607428