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Mitochondrial dysfunction reactivates α-fetoprotein expression that drives copper-dependent immunosuppression in mitochondrial disease models.

Autor: Jett KA; Department of Biochemistry, Microbiology and Immunology, University of Saskatchewan, Saskatoon, Canada., Baker ZN; Department of Biochemistry, Microbiology and Immunology, University of Saskatchewan, Saskatoon, Canada., Hossain A; Department of Biochemistry, Microbiology and Immunology, University of Saskatchewan, Saskatoon, Canada., Boulet A; Department of Biochemistry, Microbiology and Immunology, University of Saskatchewan, Saskatoon, Canada., Cobine PA; Department of Biological Sciences, Auburn University, Auburn, Alabama, USA., Ghosh S; Department of Biochemistry and Biophysics, Texas A&M University, College Station, Texas, USA., Ng P; Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA., Yilmaz O; Department of Biochemistry, Microbiology and Immunology, University of Saskatchewan, Saskatoon, Canada., Barreto K; Department of Laboratory and Pathology Medicine, University of Saskatchewan, Saskatoon, Canada., DeCoteau J; Department of Laboratory and Pathology Medicine, University of Saskatchewan, Saskatoon, Canada., Mochoruk K; Department of Laboratory and Pathology Medicine, University of Saskatchewan, Saskatoon, Canada., Ioannou GN; Division of Gastroenterology.; Research and Development, Veterans Affairs Puget Sound Health Care System and the.; Division of Gastroenterology, Department of Medicine, University of Washington, Seattle, Washington, USA., Savard C; Division of Gastroenterology.; Research and Development, Veterans Affairs Puget Sound Health Care System and the.; Division of Gastroenterology, Department of Medicine, University of Washington, Seattle, Washington, USA., Yuan S; Department of Animal and Avian Sciences, University of Maryland, College Park, Maryland, USA., Abdalla OH; Department of Biology, University of Toronto Mississauga, Mississauga, Ontario, Canada.; Department of Cell and Systems Biology, University of Toronto, Toronto, Canada., Lowden C; Department of Biology, University of Toronto Mississauga, Mississauga, Ontario, Canada.; Department of Cell and Systems Biology, University of Toronto, Toronto, Canada., Kim BE; Department of Animal and Avian Sciences, University of Maryland, College Park, Maryland, USA., Cheng HM; Department of Biology, University of Toronto Mississauga, Mississauga, Ontario, Canada.; Department of Cell and Systems Biology, University of Toronto, Toronto, Canada., Battersby BJ; Institute of Biotechnology, University of Helsinki, Helsinki, Finland., Gohil VM; Department of Biochemistry and Biophysics, Texas A&M University, College Station, Texas, USA., Leary SC; Department of Biochemistry, Microbiology and Immunology, University of Saskatchewan, Saskatoon, Canada.

Publikováno v: The Journal of clinical investigation [J Clin Invest] 2023 Jan 03; Vol. 133 (1). Date of Electronic Publication: 2023 Jan 03.

The validity of AG-NOR profile in schistosomal lesions of the urinary bladder.

Autor: Mangoud AM; Department of Pathology, Faculty of Medicine, Zagazig University., Naser AN, eL Adl M, Kamhawy MA, Abdalla OH, Salama MM

Publikováno v: Journal of the Egyptian Society of Parasitology [J Egypt Soc Parasitol] 1992 Apr; Vol. 22 (1), pp. 265-9.