Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Abby E. Macbeth"'
Autor:
Simon de Lusignan, Andrew R Thompson, Matthew Harries, Christos Tziotzios, Abby E Macbeth, Susan Holmes, Wing Sin Chiu, William Romero Gallardo, Andrew G Messenger
Publikováno v:
BMJ Open, Vol 11, Iss 11 (2021)
Introduction Alopecia areata (AA) is a common cause of immune-mediated non-scarring hair loss. Links between AA and common mental health, autoimmune and atopic conditions, and common infections have previously been described but remain incompletely e
Externí odkaz:
https://doaj.org/article/0e2508d70679492aa9ba6e9052e8371d
Autor:
Andrew G Messenger, Matthew Harries, Abby E Macbeth, Wing Sin Chiu, Susan Holmes, Christos Tziotzios, Simon de Lusignan
Publikováno v:
Clinical and Experimental Dermatology. 48:332-338
Background It is not known whether alopecia areata (AA) is associated with a greater or reduced risk for infection. Aim We undertook a population-based study exploring associations between AA and common infections. Methods We extracted primary care r
Autor:
Susan Holmes, Matthew Harries, Abby E Macbeth, Wing S Chiu, Simon de Lusignan, Andrew G Messenger, Christos Tziotzios
Publikováno v:
Clinical and Experimental Dermatology. 48:325-331
Background Alopecia areata (AA) has features of both autoimmune and atopic pathogenesis, but information on the risk of people with AA developing autoimmune and atopic conditions is limited. Objective To assess the prevalence and incidence of atopic
Autor:
Abby E. Macbeth, Susan Holmes, Matthew Harries, Wing Sin Chiu, Christos Tziotzios, Simon de Lusignan, Andrew G. Messenger, Andrew R. Thompson
Publikováno v:
British Journal of Dermatology. 187:73-81
Alopecia areata (AA) is a common cause of nonscarring hair loss that can have a profound psychological impact.To assess the co-occurrence of depression and anxiety in adults with AA compared with the general population, and to evaluate the mental hea
Publikováno v:
Journal of dermatological case reports. 7(3)
We report a case of dermatitis herpetiformis co-localised with segmental vitiligo in a 37-year-old woman with a background history of autoimmune polyglandular syndrome type 2. We propose genetic mosaicism as a possible mechanism. There has only been