Zobrazeno 1 - 10 of 62 pro vyhledávání: '"AMANDA V. STECKERT"'
1
Akademický článek
Effects of omega-3 supplementation on interleukin and neurotrophin levels in an animal model of schizophrenia
Autor: ALEXANDRA I. ZUGNO, LARA CANEVER, GUSTAVO MASTELLA, ALEXANDRA S. HEYLMANN, MARIANA B. OLIVEIRA, AMANDA V. STECKERT, ADALBERTO A. CASTRO, FELIPE DAL PIZZOL, JOÃO QUEVEDO, CLARISSA S. GAMA
Publikováno v: Anais da Academia Brasileira de Ciências, Vol 87, Iss 2 suppl, Pp 1475-1486 (2015)
ABSTRACTNew studies suggest that polyunsaturated fatty acids, such as omega-3, may reduce the symptoms of schizophrenia. The present study evaluated the preventive effect of omega-3 on interleukines (IL) and neurotrophin brain-derived neurotrophic fa
Externí odkaz: https://doaj.org/article/48b8a50d55384449a6abc86dcfdadf07
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2
Akademický článek
Modafinil Effects on Behavior and Oxidative Damage Parameters in Brain of Wistar Rats
Autor: Felipe Ornell, Samira S. Valvassori, Amanda V. Steckert, Pedro F. Deroza, Wilson R. Resende, Roger B. Varela, João Quevedo
Publikováno v: Behavioural Neurology, Vol 2014 (2014)
The effects of modafinil (MD) on behavioral and oxidative damage to protein and lipid in the brain of rats were evaluated. Wistar rats were given a single administration by gavage of water or MD (75, 150, or 300 mg/kg). Behavioral parameters were eva
Externí odkaz: https://doaj.org/article/61dd0ae8203a4f15abd79515efb55376
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3
Akademický článek
Ammonia exposition during gestation induces neonatal oxidative damage in the brain and long-term cognitive alteration in rats
Autor: DIOGO DOMINGUINI, DHÉBORA M. DALL’IGNA, LAURO NOGUEIRA, AMANDA V. STECKERT, RENATA C. GONÇALVES, MONIQUE MICHELS, JOÃO QUEVEDO, CRISTIANE RITTER, TATIANA BARICHELLO, FELIPE DAL-PIZZOL
Publikováno v: Anais da Academia Brasileira de Ciências
Abstract Ammonia is involved in the pathogenesis of neurological conditions associated with hyperammonemia, including hepatic encephalopathy. Few is known about the effects of gestational exposition to ammonia in the developing brain, and the possibl
Externí odkaz: https://doaj.org/article/aacc5ef931ef4212bd101f41650bdc9d
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5
Detailed Characterization of Brain Dysfunction in a Long-Term Rodent Model of Critical Illness
Autor: Josiane Budni, Diogo Dominguini, Milena Carvalho Silva, Francielle Mina, Mariane Abatti, Renata C. Gonçalves, Emilio L. Streck, Andressa Manfredini, Celso Carneiro Carvalho, Felipe Dal-Pizzol, Monique Michels, Amanda V. Steckert
Publikováno v: Neurochemical Research. 47:613-621
Critical illness encompasses a wide spectrum of life-threatening clinical conditions requiring intensive care. Our objective was to evaluate cognitive, inflammatory and cellular metabolism alterations in the central nervous system in an animal model
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9b72e0f42ccd71455ac61c48eb09acb7
https://doi.org/10.1007/s11064-021-03470-3
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6
The effects of anaesthetics and sedatives on brain inflammation
Autor: Jonathan P. Thompson, Cristiane Ritter, Diogo Dominguini, Mariana B Spies, Monique Michels, Amanda V. Steckert, Tatiana Barichello, Felipe Dal-Pizzol
Publikováno v: Neuroscience & Biobehavioral Reviews. 127:504-513
Microglia are involved in many dynamic processes in the central nervous system (CNS) including the development of inflammatory processes and neuromodulation. Several sedative, analgesic or anaesthetic drugs, such as opioids, ∝2-adrenergic agonists,
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2004419d4c3e1be6bb91fc380109da41
https://doi.org/10.1016/j.neubiorev.2021.05.009
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7
The Protective Effect of PK-11195 on Cognitive Impairment in Rats Survived of Polymicrobial Sepsis
Autor: Amanda V. Steckert, Jaqueline S. Generoso, Diogo Dominguini, Felipe Dal-Pizzol, Mariane Abatti, Tatiana Barichello
Publikováno v: Molecular Neurobiology. 58:2724-2733
Sepsis is an organ dysfunction caused by a host's unregulated response to infection, causing long-term brain dysfunction with microglial activation, the release of inflammatory components, and mitochondrial changes. Neuroinflammation can increase the
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::749361591f965f6fdb87ba607ad307c3
https://doi.org/10.1007/s12035-021-02294-0
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9
Mitochondrial dysfunction is associated with long-term cognitive impairment in an animal sepsis model
Autor: João Quevedo, Lara Mezzari Gomes, Diogo Dominguini, Cristiane Ritter, Larissa Constantino, Tatiana Barichello, Milton Castro Pinto, Amanda V. Steckert, Luiza Wilges Kist, Monique Michels, Maurício Reis Bogo, Mervyn Singer, Carmen Simioni, Henrique Burger, Milena Carvalho Silva, Felipe Dal-Pizzol, Andressa Manfredini, Emilio L. Streck
Publikováno v: Clinical Science. 133:1993-2004
Background: Several different mechanisms have been proposed to explain long-term cognitive impairment in sepsis survivors. The role of persisting mitochondrial dysfunction is not known. We thus sought to determine whether stimulation of mitochondrial
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6c01cd05fb1fcbfd63693e03678c733f
https://doi.org/10.1042/cs20190351
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10
Effects of low-intensity training on the brain and muscle in the congenital muscular dystrophy 1D model
Autor: Clarissa M, Comim, Jaime A, Soares, Adriano, Alberti, Viviane, Freiberger, Letícia, Ventura, Paula, Dias, Aryadnne L, Schactae, Leoberto R, Grigollo, Amanda V, Steckert, Daniel F, Martins, Rudy J Nodari, Junior, Mariz, Vainzof, João, Quevedo
Publikováno v: Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical NeurophysiologyReferences. 43(7)
Congenital Muscular Dystrophy type 1D (MDC1D) is characterized by a hypoglycosylation of α-dystroglycan protein (α-DG), and this may be strongly implicated in increased skeletal muscle tissue degeneration and abnormal brain development, leading to
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::d610e037b55d37396702f33211e48e3a
https://pubmed.ncbi.nlm.nih.gov/35353268
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