Zobrazeno 1 - 10
of 495
pro vyhledávání: '"A.G. Herman"'
Publikováno v:
British Journal of Pharmacology. 158:920-930
Background and purpose: Reduction of intracellular calcium ([Ca2+]i) in smooth muscle cells (SMCs) is an important mechanism by which nitric oxide (NO) dilates blood vessels. We investigated whether modes of Ca2+ mobilization during SMC contraction i
Autor:
A.G. Herman, B.Y. De Winter, L. Van Nassauw, H. U. De Schepper, Paul A. Pelckmans, Jean-Pierre Timmermans, J. G. De Man
Publikováno v:
Gut : the journal of the British Society for Gastroenterology
Background and aims: Patients with inflammatory bowel disease often present with abnormal gut motility away from the inflammatory site. We studied remote motility disturbances and their pathophysiology in a rat model of colitis. Methods: Colitis was
Publikováno v:
British Journal of Pharmacology. 150:88-96
Background and purpose: cGMP mediates nitrergic relaxations of intestinal smooth muscle, but several studies have indicated that cGMP-independent mechanisms may also be involved. We addressed this contention by studying the effect of ODQ and NS2028,
Autor:
Hidde Bult, T. J.L. Van De Parre, Dominique M. Jans, Mark M. Kockx, Wim Martinet, G.R.Y. De Meyer, A.G. Herman
Publikováno v:
Cardiovascular & Hematological Disorders-Drug Targets. 6:21-34
Macrophage activation in atherosclerotic plaques plays a role in plaque destabilization, rupture and subsequent atherothrombosis. Platelet phagocytosis that occurs within human atherosclerotic plaques can activate macrophages and it has been suggeste
Regional differences in gastrointestinal motility disturbances during acute necrotising pancreatitis
Publikováno v:
Neurogastroenterology and motility
Patients with acute pancreatitis often suffer from intestinal motility disturbances but the mechanism of this dysfunction is largely unknown. We studied the effect of acute necrotising pancreatitis (ANP) on in vivo gastrointestinal motility and in vi
Publikováno v:
Experimental and toxicological pathology
Summary Rabbits receiving a dietary cholesterol supplement of 0.5 % develop atheromatous plaques in the systemic arteries, the pulmonary artery, the pulmonary veins and the venous cavities of the plexus pampiniformis in the funiculus spermaticus. Six
Autor:
J.M. Boeynaems, A.G. Herman
A Workshop Symposium on Prostaglandins, prostacyclin and thromboxanes measurement; methodological problems and clinical prospects, Nivelles, Belgium, November 15-16, 1979
2 The free internal Ca+ concentration in human red cells is set according to the leak 2 and-pump principle: There is a finite passive Ca+ influx at the physiological 2 2 Ca+ -gradient across the membrane which is compensated by Ca+ pumping in the o