Zobrazeno 1 - 10
of 202
pro vyhledávání: '"A. Raszeja-Specht"'
Autor:
Golański, Jacek, Raszeja-Specht, Anna
Publikováno v:
Journal of Laboratory Diagnostics / Diagnostyka Laboratoryjna; 2023, Vol. 59 Issue 4, p1-18, 18p
Autor:
Anna Raszeja-Specht, Anna Michno
Publikováno v:
Diagnostyka Laboratoryjna. 51:221-228
A new generation of oral anticoagulants, direct thrombin or activated factor X inhibitors gradually replaces the existing antithrombotic agents, administered parenterally (heparin) or orally (vitamin K antagonists). Currently, the most widely used cl
Autor:
Anna Raszeja-Specht, Agnieszka Jankowska-Kulawy, Andrzej Szutowicz, Anna Michno, Tadeusz Pawelczyk
Publikováno v:
Clinical Chemistry. 51:1673-1682
Background: Excessive blood platelet activity contributes to vascular complications in diabetic persons. Increased acetyl-CoA in platelets from diabetic persons has been suggested to be a cause of this hyperactivity. We therefore investigated whether
Publikováno v:
Nephron. 92:36-42
Platelet secretion products may play an important role in the pathogenesis and progression of the kidney disease. Amongst the parameters describing platelet hyperactivity the measurement of spontaneous platelet aggregation (SPA) seems particularly us
Publikováno v:
American Journal of Hematology. 68:32-36
Three subgroups have been distinguished in essential thrombocythaemia (ET) patients, on the basis of clinical and laboratory findings. ET patients with bleeding incidents had smaller platelet volume, lower concentrations of b-thromboglobulin and plat
Publikováno v:
American Journal of Hematology; 2001, Vol. 68 Issue 1, p32-36, 5p
Publikováno v:
Metabolism: clinical and experimental. 53(1)
Diabetes is known to increase blood platelet activity. Activities of pyruvate dehydrogenase (PDH), adenosine triphosphate (ATP)-citrate lyase (ATPCL), acetyl-coenzyme A (acetyl-CoA) content, malonyl dialdehyde (MDA), synthesis, and platelet aggregati
Publikováno v:
Clinical chemistry and laboratory medicine. 41(9)
Blood platelets take up glucose through insulin-independent GLUT-3 transporter. It is, however, unclear how diabetes affects further steps of glucose and glucose-derived acetyl-coenzyme A (CoA) metabolism in platelets. There is no evidence to explain
Publikováno v:
Clinical Chemistry & Laboratory Medicine; Sep2003, Vol. 41 Issue 9, p1136-1143, 8p