Zobrazeno 1 - 4
of 4
pro vyhledávání: '"A. L. Schwartsman"'
Autor:
D. G. Kulabukhova, L. A. Garaeva, A. K. Emelyanov, K. A. Senkevich, E. V. Gracheva, I. V. Miliukhina, E. Y. Varfolomeeva, A. A. Timofeeva, A. L. Schwartsman, T. A. Shtam, S. N. Pchelina
Publikováno v:
Molecular Biology. 55:297-303
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f6c594788c256fee40c8124f447be9a8
https://doi.org/10.1134/s002689332101009x
https://doi.org/10.1134/s002689332101009x
Autor:
E.V. Gracheva, A A Timofeeva, Irina Miliukhina, Elena Yu. Varfolomeeva, D. G. Kulabukhova, Konstantin Senkevich, L. A. Garaeva, Tatiana Shtam, S N Pchelina, A. Emelyanov, A L Schwartsman
Publikováno v:
Молекулярная биология. 55:338-345
Parkinson's disease (PD) is the second most common neurodegenerative disorder. Alpha-synuclein misfolding and aggregation resulting in neurototoxicity is a hallmark of PD. The prion properties of alpha-synuclein are still under discussion. Exosomes (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bfaa76a1b56d139e0e5e2a4edfc56604
https://doi.org/10.31857/s0026898421010092
https://doi.org/10.31857/s0026898421010092
Autor:
D G, Kulabukhova, L A, Garaeva, A K, Emelyanov, K A, Senkevich, E V, Gracheva, I V, Miliukhina, E Y, Varfolomeeva, A A, Timofeeva, A L, Schwartsman, T A, Shtam, S N, Pchelina
Publikováno v:
Molekuliarnaia biologiia. 55(2)
Parkinson's disease (PD) is the second most common neurodegenerative disorder. Alpha-synuclein misfolding and aggregation resulting in neurototoxicity is a hallmark of PD. The prion properties of alpha-synuclein are still under discussion. Exosomes (
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::5845626c292749e17b1167072833668c
https://pubmed.ncbi.nlm.nih.gov/33871446
https://pubmed.ncbi.nlm.nih.gov/33871446
Autor:
S. V. Sarantseva, Michael P. Vitek, O. L. Runova, Yu. A. Tatishcheva, E. I. Talalaeva, A. L. Schwartsman
Publikováno v:
Biophysics. 51:740-743
Most cases of early-onset familial Alzheimer’s disease are caused by mutations in the presenilin 1 gene. Nonetheless, the function of presenilin 1 are not yet completely understood. It was shown that endogenous presenilin 1, as well as the adhesion
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3608b8399fa461873c1e9cfc5e41447c
https://doi.org/10.1134/s0006350906050125
https://doi.org/10.1134/s0006350906050125