Zobrazeno 1 - 10
of 29
pro vyhledávání: '"A. Faure-Dussert"'
Publikováno v:
Journal of Endocrinology. 160:87-95
Because 1,25 dihydroxyvitamin D3 (1,25(OH)2D3) is known to activate the biosynthesis of numerous proteins in various tissues, experiments were undertaken to compare the influence of 1,25(OH)2D3 in vitro on both the secretion and biosynthesis of insul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d03a99b0b93d46c2f8dd655ca949b64c
https://doi.org/10.1677/joe.0.1600087
https://doi.org/10.1677/joe.0.1600087
Publikováno v:
British Journal of Pharmacology. 121:751-758
Previous studies have shown that vitamin D3 deficiency impairs the insulin response to glucose via an alteration of signal transduction pathways, such as Ca2+ handling and the phosphoinositide pathway. In the present study the adenylyl cyclase pathwa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ef4181f516600d3081362739fd168a9e
https://doi.org/10.1038/sj.bjp.0701204
https://doi.org/10.1038/sj.bjp.0701204
Publikováno v:
Journal of Endocrinological Investigation. 18:673-682
The effect of 10−8 M 1,25-dihydroxyvitamin D3 [1,25 (OH)2D3] on the phosphoinositide pathway, was studied on [3H] inositol and 45Ca2+ efflux and on insulin release of islets from vitamin D-deficient rats, during an acetylcholine (Ach) stimulus in p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0064cd0eb26fe329bd19816dbd23d9de
https://doi.org/10.1007/bf03349788
https://doi.org/10.1007/bf03349788
Publikováno v:
Diabetesmetabolism. 24(4)
After 5 weeks of vitamin D3 deficiency, rats exhibited signs of rachitism, hypocalcaemia and hypoinsulinaemia. As the glucose-induced insulin release process requires calcium and energy production from glucose metabolism within beta cells of Langerha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::e2358101cc7599cbc27f1232722654b5
https://pubmed.ncbi.nlm.nih.gov/9805645
https://pubmed.ncbi.nlm.nih.gov/9805645
1. Previous studies have shown that vitamin D3 deficiency impairs the insulin response to glucose via an alteration of signal transduction pathways, such as Ca2+ handling and the phosphoinositide pathway. In the present study the adenylyl cyclase pat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::cf7bfe73b6fbbde8d2fe6ca12a2de679
https://europepmc.org/articles/PMC1564753/
https://europepmc.org/articles/PMC1564753/
Publikováno v:
The Journal of endocrinology. 148(2)
The pancreatic B cell is equipped with specific receptors for 1,25-dihydroxyvitamin D3 (1,25-(OH)2D3) and contains vitamin D-dependent calcium binding proteins (calbindin-D). Insulin secretion is impaired by vitamin D deficiency and is restored by 1,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::225689bd6a6cd1d0b1be47d18d9f0032
https://pubmed.ncbi.nlm.nih.gov/8699136
https://pubmed.ncbi.nlm.nih.gov/8699136
Publikováno v:
Vitamin D
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cb40109b63384ec8c8899ed4e796715a
https://doi.org/10.1515/9783110882513-136
https://doi.org/10.1515/9783110882513-136
Akademický článek
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Akademický článek
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Publikováno v:
Journal of Endocrinology; Jul1999, Vol. 162 Issue 1, p101-109, 9p