Zobrazeno 1 - 10
of 362
pro vyhledávání: '"A. Benito-Vicente"'
Autor:
Antonio Veloso-Fernández, José Manuel Laza, Leire Ruiz-Rubio, Ane Martín, Asier Benito-Vicente, Cesar Martín, José Luis Vilas-Vilela
Publikováno v:
Materials, Vol 17, Iss 19, p 4770 (2024)
Cytotoxicity is a critical parameter for materials intended for biological applications, such as food packaging. Shape-memory polyurethanes (SMPUs) have garnered significant interest due to their versatile properties and adaptability in synthesis. Ho
Externí odkaz:
https://doaj.org/article/9604bdca17f746efa6f5ae351de1ce18
Autor:
Asier Larrea‐Sebal, Iñaki Sasiain, Shifa Jebari‐Benslaiman, Unai Galicia‐Garcia, Kepa B. Uribe, Asier Benito‐Vicente, Irene Gracia‐Rubio, Harbil Bediaga‐Bañeres, Sonia Arrasate, Ana Cenarro, Fernando Civeira, Humberto González‐Díaz, Cesar Martín
Publikováno v:
Advanced Science, Vol 11, Iss 13, Pp n/a-n/a (2024)
Abstract Familial hypercholesterolemia (FH) is an inherited metabolic disease affecting cholesterol metabolism, with 90% of cases caused by mutations in the LDL receptor gene (LDLR), primarily missense mutations. This study aims to integrate six comm
Externí odkaz:
https://doaj.org/article/91edba494e384699a0634101a006d890
Autor:
Veloso-Fernández, Antonio, Laza, José Manuel, Ruiz-Rubio, Leire, Martín, Ane, Benito-Vicente, Asier, Martín, Cesar, Vilas-Vilela, José Luis
Publikováno v:
Materials (1996-1944); Oct2024, Vol. 17 Issue 19, p4770, 16p
Autor:
Larrea-Sebal, Asier, Benito-Vicente, Asier, Fernandez-Higuero, José A., Jebari-Benslaiman, Shifa, Galicia-Garcia, Unai, Uribe, Kepa B., Cenarro, Ana, Ostolaza, Helena, Civeira, Fernando, Arrasate, Sonia, González-Díaz, Humberto, Martín, César
Publikováno v:
In JACC: Basic to Translational Science November 2021 6(11):815-827
Autor:
Alves, Ana Catarina, Azevedo, Sílvia, Benito-Vicente, Asier, Graça, Rafael, Galicia-Garcia, Unai, Barros, Patrícia, Jordan, Peter, Martin, Cesar, Bourbon, Mafalda
Publikováno v:
In Atherosclerosis July 2021 329:14-21
Autor:
Nicolás M. Suárez, Shifa Jebari-Benslaiman, Roberto Jiménez-Monzón, Asier Benito-Vicente, Yeray Brito-Casillas, Laida Garcés, Ana M. González-Lleo, Antonio Tugores, Mauro Boronat, César Martin, Ana M. Wägner, Rosa M. Sánchez-Hernández
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 14, p 11319 (2023)
The p.(Tyr400_Phe402del) mutation in the LDL receptor (LDLR) gene is the most frequent cause of familial hypercholesterolaemia (FH) in Gran Canaria. The aim of this study was to determine the age and origin of this prevalent founder mutation and to e
Externí odkaz:
https://doaj.org/article/c1d7477560414ecb82a1f4bf2b208f71
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 8, p 7659 (2023)
This Special Issue, “Cardiovascular Disease, Atherosclerosis and Familial Hypercholesterolemia: From Molecular Mechanisms Causing Pathogenicity to New Therapeutic Approaches”, contributes to advancing our knowledge of the molecular mechanisms tha
Externí odkaz:
https://doaj.org/article/28e3571fdcec466fbbef48d89b917eb3
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Autor:
Asier Larrea-Sebal, Chiara Trenti, Shifa Jebari-Benslaiman, Stefano Bertolini, Sebastiano Calandra, Emanuele A. Negri, Efrem Bonelli, Asier Benito-Vicente, Leire Uraga-Gracianteparaluceta, César Martín, Tommaso Fasano
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 4, p 3330 (2023)
Familial hypercholesterolaemia (FH) is an autosomal dominant dyslipidaemia, characterised by elevated LDL cholesterol (LDL-C) levels in the blood. Three main genes are involved in FH diagnosis: LDL receptor (LDLr), Apolipoprotein B (APOB) and Protein
Externí odkaz:
https://doaj.org/article/83ca415e3111468c8d3293155e452740
Autor:
Carmen Navarro-Gonzalez, Héctor Carceller, Marina Benito Vicente, Irene Serra, Marta Navarrete, Yaiza Domínguez-Canterla, Ángela Rodríguez-Prieto, Ana González-Manteiga, Pietro Fazzari
Publikováno v:
Neurobiology of Disease, Vol 157, Iss , Pp 105442- (2021)
Neuregulin 1 (NRG1) and its receptor ERBB4 are schizophrenia (SZ) risk genes that control the development of both excitatory and inhibitory cortical circuits. Most studies focused on the characterization ErbB4 deficient mice. However, ErbB4 deletion
Externí odkaz:
https://doaj.org/article/26c10ea82df1412d8fd84ffb7e3c1d0f