Zobrazeno 1 - 8
of 8
pro vyhledávání: '"A. A. Kashintsova"'
Autor:
O. L. Kolomiets, E. E. Bragina, A. A. Kashintsova, V. E. Spangenberg, L. A. Nikulina, Yu. N. Korolev, L. V. Mikhailik
Publikováno v:
Андрология и генитальная хирургия, Vol 21, Iss 4, Pp 76-88 (2021)
Introduction. Metabolic syndrome (MS) can cause impaired spermatogenesis and a decrease in sperm counts. However, the details of the effect of MS on developing spermatogenic cells remain unclear. Difficulties in solving this problem, the inconsistenc
Externí odkaz:
https://doaj.org/article/3231de42a41347d786699846e335c95a
Autor:
E. E. Bragina, I. I. Vityazeva, M. A. Lelekova, A. A. Kashintsova, S. V. Bogolyubov, M. Yu. Gabliya, I. V. Vinogradov, V. E. Spanberg, O. L. Kolomiets
Publikováno v:
Андрология и генитальная хирургия, Vol 20, Iss 1, Pp 43-54 (2019)
The study objective is to determine the connection between meiosis abnormalities and changes in the basement membrane (BM) ultrastructure of the seminiferous tubules in patients with spermatogenesis disorders: azoospermia or severe oligospermia.Mater
Externí odkaz:
https://doaj.org/article/8ad14c62024b4cc2aadd0c10fc1da639
Autor:
V. G. Tambovtseva, S. N. Matveevsky, A. A. Kashintsova, A. V. Tretiakov, O. L. Kolomiets, I. Yu. Bakloushinskaya
Publikováno v:
Вавиловский журнал генетики и селекции, Vol 23, Iss 2, Pp 239-243 (2019)
Chromosomal rearrangements can lead to the formation of new stable karyotypes, nevertheless changing the architectonics of the nucleus. The differences in locations might promote Robertsonian (Rb) translocations and encourage meiotic drive in favour
Externí odkaz:
https://doaj.org/article/d0cd2386da374d72aea2ca0b9491217c
Autor:
O. L. Kolomiets, M. A. Lelekova, A. A. Kashintsova, L. F. Kurilo, E. E. Bragina, V. B. Chernykh, M. Yu. Gabliya, I. V. Vinogradov, I. I. Vityazeva, S. V. Bogolyubov, V. E. Spangenberg
Publikováno v:
Андрология и генитальная хирургия, Vol 19, Iss 1, Pp 24-35 (2018)
Introduction. Infertility is diagnosed in 10–15 % of couples wishing to have children. In about half cases, the cause is a disorders of male fertility. Defects of spermatogenesis are often caused by the damages of key events of prophase I meiosis
Externí odkaz:
https://doaj.org/article/11e3b1be8b9a44a4869310354ce4d26c
Autor:
Sergey Matveevsky, Artemii Tretiakov, Anna Kashintsova, Irina Bakloushinskaya, Oxana Kolomiets
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 20, p 7630 (2020)
Genome functioning in hybrids faces inconsistency. This mismatch is manifested clearly in meiosis during chromosome synapsis and recombination. Species with chromosomal variability can be a model for exploring genomic battles with high visibility due
Externí odkaz:
https://doaj.org/article/398bc04e9dee4e078808fb30191f43fd
Autor:
V. E. Spangenberg, Nikulina La, L V Mikhailik, E. E. Bragina, Oxana Kolomiets, Yu N Korolev, A. A. Kashintsova
Publikováno v:
Andrologiâ i Genitalʹnaâ Hirurgiâ, Vol 21, Iss 4, Pp 76-88 (2021)
Introduction. Metabolic syndrome (MS) can cause impaired spermatogenesis and a decrease in sperm counts. However, the details of the effect of MS on developing spermatogenic cells remain unclear. Difficulties in solving this problem, the inconsistenc
Autor:
Anna Kashintsova, Artemii Tretiakov, Oxana Kolomiets, Sergey Matveevsky, Irina Bakloushinskaya
Publikováno v:
International Journal of Molecular Sciences
Volume 21
Issue 20
International Journal of Molecular Sciences, Vol 21, Iss 7630, p 7630 (2020)
Volume 21
Issue 20
International Journal of Molecular Sciences, Vol 21, Iss 7630, p 7630 (2020)
Genome functioning in hybrids faces inconsistency. This mismatch is manifested clearly in meiosis during chromosome synapsis and recombination. Species with chromosomal variability can be a model for exploring genomic battles with high visibility due
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