Zobrazeno 1 - 10
of 60
pro vyhledávání: '"A L, Shvartsman"'
Autor:
S. N. Pchelina, T. S. Usenko, N. A. Bogankova, A. F. Yakimovskii, A. K. Emelyanov, T. V. Vavilova, A. L. Shvartsman
Publikováno v:
Медицинская иммунология, Vol 14, Iss 1-2, Pp 149-152 (2014)
Abstract. Progressive loss of dopaminergic neurons from substantia nigra is the major pathomorphologicasign in Parkinson’s disease (PD). Neuronal death is suggested to occur by programmed cell death (apoptosis) in PD patients, thus being involved i
Externí odkaz:
https://doaj.org/article/5554853da64c40bbacf5a76c91bd7f11
Autor:
S. V. Sarantseva, A L Shvartsman, S. I. Timoshenko, Michael P. Vitek, G. A. Kislik, E. M. Latypova
Publikováno v:
Biomeditsinskaya Khimiya. 60:515-521
The neuroprotective activity of apolipoprotein E (apoE) peptide mimetic Cog1410, containing amino acid sequence of the receptor-binding domain apoE, has been investigated in transgenic lines of Drosophila melanogaster expressing human APP and beta-se
Autor:
N A Bogan'kova, A L Shvartsman, A F Iakimovskiĭ, T. V. Vavilova, S N Pchelina, A K Emel'ianov, T S Usenko
Publikováno v:
Cell and Tissue Biology. 6:171-175
Mutations in the Leucine Reach Repeat Kinase 2 (LRRK2) gene are the most frequent cause of familial Parkinson's disease (PD). Although the precise physiological and pathological role of LRRK2 is unclear, a direct link between mutant LRRK2 and apoptos
Publikováno v:
Cell and Tissue Biology. 6:60-68
One of the earliest neuropathological symptoms of Alzheimer's disease is the loss of synapses, which preceed the formation of amyloidosis and neurodegeneration. Although most cases of early-onset familial Alzheimer's disease are caused by mutations i
Autor:
A. L. Shvartsman, K. V. Solovyov, Vladimir V. Egorov, T. D. Aleinikova, A. A. Gasteva, A. K. Sirotkin, Mikhail M. Shavlovsky
Publikováno v:
Biochemistry (Moscow). 71:543-549
Polypeptide chain fragments of recombinant transthyretin (TTR) with leucine-55 substituted by proline (L55P), which are involved in abnormal fibrillogenesis of this protein, were studied. No fibrils were produced in purified preparations of TTR(L55P)
Autor:
V V, Miroshnikova, E P, Demina, N V, Maĭorov, V V, Davydenko, P S, Kur'ianov, V N, Vavilov, A G, Vinogradov, A D, Denisenko, A L, Shvartsman
Publikováno v:
Tsitologiia. 56(3)
Accumulation of cholesterol in arterial wall macrophages is a main hallmark of atherosclerosis. The ABCG1 transporter mediates cholesterol efflux to high density lipoproteins (HDL) and plays an important role in macrophage foam cell formation. The go
Publikováno v:
Tsitologiia. 55(8)
ABCA1 transporter is one of the key factors defining the level of antiatherogenic HDL in plasma. It is actively involved in the removal of cholesterol from peripheral tissues by reverse cholesterol transport. However, the influence of the level of AB
Publikováno v:
Tsitologiia. 54(5)
Alzheimer's disease (AD) is a neurodegenerative disorder characterized by the loss of neurocortical and hippocampal synapses that precedes amyloidosis and neurodegeneration and closely correlates with memory impairment. Mutations in the amyloid precu
Publikováno v:
Tsitologiia. 53(12)
One of the earliest neuropathological symptoms of Alzheimer's disease is the loss of synapses, which preceed the formation of amyloidosis and neurodegeneration. Although most cases of early-onset familial Alzheimer's disease are caused by mutations i
In this chapter we present our work aimed at interweaving e-learning and face-to-face learning in Calculus courses for undergraduate engineering students. This type of blended learning (BL) contains the best properties of e-learning and face-to-face
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8e35e85880fcdb1b15f2a0c074a7699c
https://doi.org/10.4018/978-1-60960-875-0.ch002
https://doi.org/10.4018/978-1-60960-875-0.ch002