Výsledky vyhledávání

TRH and TRH-Like Peptide Levels Co-Vary with Reproductive and Metabolic Rhythms

Autor: A. Sattin, E. Pekary

Publikováno v: Hormone and Metabolic Research. 49:86-94

Photoperiod-synchronized rhythms in non-CSN tissues persist in total darkness. Clock genes involved in maintaining regular biorhythms within the suprachiasmatic nucleus (SCN) of the hypothalamus are expressed in extra-CNS tissues and continue periodi

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::efadba0b401c3ff42b14428e3f236b95
https://doi.org/10.1055/s-0042-111012

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Tumor Necrosis Factor-α (TNF-α) and Transforming Growth Factor-βl (TGF-βl) Inhibit the Expression and Activity of Na+/K+-ATPase in FRTL-5 Rat Thyroid Cells

Autor: L Berg, S. R. Levin, Jerome M. Hershman, A. E. Pekary, D G Johnson

Publikováno v: Journal of Interferon & Cytokine Research. 17:185-195

We recently reported that tumor necrosis factor-alpha (TNF-alpha) induction of the synthesis and secretion of transforming growth factor (TGF)-beta 1 by FRTL-5 cells is a thyroid-stimulating hormone (TSH)-dependent and age-dependent process. TNF-alph

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::db63873090dcbc877eff70c58c7ca7ae
https://doi.org/10.1089/jir.1997.17.185

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TNF-alpha, TSH, and aging regulate TGF-beta synthesis and secretion in FRTL-5 rat thyroid cells

Autor: L. Berg, P. Lee, S. M. Dubinett, Jianyi Wang, A. E. Pekary, Jerome M. Hershman

Publikováno v: American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 268:R808-R815

Tumor necrosis factor-alpha (TNF-alpha), a cytokine produced by macrophages in response to a variety of pathological conditions, can inhibit thyroid cell function in vitro and in vivo. TNF-alpha induction of transforming growth factor-beta 1 (TGF-bet

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5217a2ed800b07d0c921705fad58c128
https://doi.org/10.1152/ajpregu.1995.268.3.r808

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TRH and Related Peptides

Autor: Patricia M. Hinkle, Albert Sattin, Mark Paulson, J. A. Meyerhoff, A. E. Pekary, Kym F. Faull, Robert L. Lloyd

Publikováno v: Annals of the New York Academy of Sciences. 1003:458-460

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::6d8206e1e5cd0a8a07a4eb9a90ce7208
https://doi.org/10.1196/annals.1300.053

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Thyrotropic activity of basic isoelectric forms of human chorionic gonadotropin extracted from hydatidiform mole tissues

Autor: M. Yoshimura, Xuan-Ping Pang, A. E. Pekary, Jerome M. Hershman, T. M. Goodwin, L. Berg

Publikováno v: The Journal of Clinical Endocrinology & Metabolism. 78:862-866

hCG is known to have thyroid-stimulating activity and may cause hyperthyroidism in patients with trophoblastic diseases. hCG occurs in normal and molar pregnancy with breaks or nicks in the alpha- or beta-subunit peptide linkage and with substantial

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7b72aacb1c2de52f8d6907d4358ace41
https://doi.org/10.1210/jcem.78.4.8157712

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Effect of peptide nicking in the human chorionic gonadotropin β-subunit on stimulation of recombinant human thyroid-stimulating hormone receptors

Autor: A. E. Pekary, L. Berg, L. A. Cole, Jerome M. Hershman, A. Kardana, M. Yoshimura, Xuan-Ping Pang

Publikováno v: European Journal of Endocrinology. 130:92-96

Yoshimura M, Pekary AE, Pang X-P, Berg L, Cole LA, Kardana A, Hershman JM. Effect of peptide nicking in the human chorionic gonadotropin β-subunit on stimulation of recombinant human thyroid-stimulating hormone receptors. Eur J Endocrinol 1994;130:9

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::70be4849cbe29fe62b5fb5ea317fcd2e
https://doi.org/10.1530/eje.0.1300092

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Increased in vitro thyrotropic activity of partially sialated human chorionic gonadotropin extracted from hydatidiform moles of patients with hyperthyroidism

Autor: A. E. Pekary

Publikováno v: Journal of Clinical Endocrinology & Metabolism. 76:70-74

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::ce57b612a60c66b58c4d75f94e81365d
https://doi.org/10.1210/jc.76.1.70

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The mechanism of action of tumour necrosis factor-α and interleukin 1 on FRTL-5 rat thyroid cells

Autor: V. Smith, Jerome M. Hershman, A. E. Pekary, Xuan-Ping Pang, Masahiro Sugawara

Publikováno v: Acta Endocrinologica. 123:203-210

Previous work showed that treatment of rats with tumour necrosis factor-α produced a model of nonthyroid illness in which there was reduction of circulating thyroid hormones and TSH, reduced thyroid response to TSH, and reduced thyroid iodide uptake

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::548c12845c0fe0c0feb9b7846b58f71e
https://doi.org/10.1530/acta.0.1230203

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Autor: A, Sattin, A E, Pekary, R L, Lloyd, M, Paulson, J A, Meyerhoff, P M, Hinkle, K, Faull

Publikováno v: Annals of the New York Academy of Sciences. 1003

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::1d3982f8ede73cf1eb3a3252f4c3141e
https://pubmed.ncbi.nlm.nih.gov/14684488

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Tumor necrosis factor, ceramide, transforming growth factor-beta1, and aging reduce Na+/I- symporter messenger ribonucleic acid levels in FRTL-5 cells

Autor: A E, Pekary, J M, Hershman

Publikováno v: Endocrinology. 139(2)

Iodide uptake, which is necessary for thyroid hormone synthesis, can be inhibited by aging, withdrawal of TSH, or increased tumor necrosis factor (TNF) and transforming growth factor (TGF)-beta1 levels resulting from the nonthyroid illness syndrome.

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::533cdec666b5093c399282c10b8b1ab0
https://pubmed.ncbi.nlm.nih.gov/9449644

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