Zobrazeno 1 - 10
of 253
pro vyhledávání: '"α receptors"'
Autor:
Claudio R. Bertevello, Bruno R. A. Russo, Ana C. Tahira, Ednilson Hilário Lopes-Junior, Ricardo DeMarco, Katia C. Oliveira
Publikováno v:
Parasites & Vectors, Vol 13, Iss 1, Pp 1-12 (2020)
Abstract Background The TNF signaling pathway is involved in the regulation of many cellular processes (such as apoptosis and cell proliferation). Previous reports indicated the effect of human TNF-α on metabolism, physiology, gene expression and pr
Externí odkaz:
https://doaj.org/article/9b2907a5c87a442da55813b1fc81b774
Autor:
Е. V. Zvezdina, J. V. Dayronas, I. I. Bochkareva, I. N. Zilfikarov, E. Yu. Babaeva, E. V. Ferubko, Z. A. Guseynova, F. K. Serebryanaya, S. R. Kaibova, T. A. Ibragimov
Publikováno v:
Фармация и фармакология (Пятигорск), Vol 8, Iss 1, Pp 4-28 (2020)
The aim of this work is to review and analyze the data published in the modern scientific literature obtained in pharmacological, pharmacognostic and pharmacotechnological studies of various types of raw materials obtained from members of the family
Externí odkaz:
https://doaj.org/article/5dc059e6389e4358b499e4eeb980e27c
Autor:
Dewan S. A. Majid, Alexander Castillo
Publikováno v:
Physiological Reports, Vol 9, Iss 15, Pp n/a-n/a (2021)
Abstract Intravenous infusion of relatively higher doses of angiotensin II (AngII) elicits natriuresis as opposed to its usual anti‐natruretic response. As AngII can induce tumor necrosis factor‐α (TNFα) production which elicits natriuresis via
Externí odkaz:
https://doaj.org/article/9116e7334a014b849884c4bc6ac3faad
Publikováno v:
Physiological Reports, Vol 9, Iss 16, Pp n/a-n/a (2021)
Abstract In hypertension induced by angiotensin II (AngII) administration with high salt (HS) intake, intrarenal angiotensinogen (AGT) and tumor necrosis factor‐alpha (TNF‐α) levels increase. However, TNF‐α has been shown to suppress AGT form
Externí odkaz:
https://doaj.org/article/06ed98b1a3104f7eb664bb34ebd5a9be
Autor:
Bárbara Pesce, Carolina H. Ribeiro, Milton Larrondo, Verónica Ramos, Lilian Soto, Diego Catalán, Juan Carlos Aguillón
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 16, p 9306 (2022)
Tumor necrosis factor (TNF)-α is a pleiotropic cytokine implicated in the etiology of several autoimmune diseases, including rheumatoid arthritis (RA). TNF-α regulates diverse effector functions through the activation of TNF-α receptor (TNFR)1 and
Externí odkaz:
https://doaj.org/article/9c3ef8d3e4c3450a9600c9849f9675a6
Autor:
Ajello, F., La Licata, R., Lodato, M., Vitale, F., Bonura, F., Valenti, R., Bruno, M., Romano, N.
Publikováno v:
European Journal of Epidemiology, 2000 Mar 01. 16(3), 209-216.
Externí odkaz:
https://www.jstor.org/stable/3582273
Autor:
Dragica Selakovic, Jovana Joksimovic, Nemanja Jovicic, Slobodanka Mitrovic, Vladimir Mihailovic, Jelena Katanic, Dragan Milovanovic, Suzana Pantovic, Natasa Mijailovic, Gvozden Rosic
Publikováno v:
Frontiers in Behavioral Neuroscience, Vol 13 (2019)
The aim of this study was to evaluate alterations in depressive-like behaviors in rats following chronic administration of a supraphysiological dose of anabolic androgenic steroids (AASs) as well as exposure to a prolonged exercise protocol. The role
Externí odkaz:
https://doaj.org/article/0787c9d0e5c54164ac9720a63b24e5a0
Akademický článek
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Autor:
Franziska Rohde, Benjamin Schusser, Tomáš Hron, Helena Farkašová, Jiří Plachý, Sonja Härtle, Jiří Hejnar, Daniel Elleder, Bernd Kaspers
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
Tumor necrosis factor-α (TNF-α) is a pleiotropic cytokine playing critical roles in host defense and acute and chronic inflammation. It has been described in fish, amphibians, and mammals but was considered to be absent in the avian genomes. Here,
Externí odkaz:
https://doaj.org/article/42befca8088e4f4d8babe0f6b4853b14
Publikováno v:
Journal of Inorganic and Organometallic Polymers and Materials. 31:2990-2998
Two novel metal–organic compounds have been prepared via altering metal ions under the hydrothermal conditions, i.e. [Zn(HL)(4,4′-bipy)(H2O)2]n (1) and [Cd(HL)(4,4′-bipy)]n (2) (4,4′-bipy = 4,4′-bipyridine and H3L = 5-(4-carboxyphenoxy)-iso