Zobrazeno 1 - 10
of 18
pro vyhledávání: '"İlknur Sur‐Erdem"'
Publikováno v:
Brain and Spine, Vol 3, Iss , Pp 101702- (2023)
Introduction: Primary glioblastoma (GBM) cell cultures have become a very important tool for future research due to the patient-specific genetic makeup of GBM cells and different treatment response variations. In sodium fluorescein-guided surgeries,
Externí odkaz:
https://doaj.org/article/fb504947ae06459faa70185308e6b297
Publikováno v:
Brain Structure and Function. 226:2113-2123
The synchronization of astrocytes via gap junctions (GJ) is a crucial mechanism in epileptic conditions, contributing to the synchronization of the neuronal networks. Little is known about the endogenous response of GJ in genetic absence epileptic an
Publikováno v:
Brain Structure and Function. 228:1039-1039
Autor:
Nareg Pinarbasi-Degirmenci, Ilknur Sur-Erdem, Vuslat Akcay, Yasemin Bolukbasi, Ugur Selek, Ihsan Solaroglu, Tugba Bagci-Onder
Publikováno v:
International Journal of Molecular Sciences
International Journal of Molecular Sciences; Volume 23; Issue 13; Pages: 7051
International Journal of Molecular Sciences; Volume 23; Issue 13; Pages: 7051
Glioblastoma is the most malignant primary brain tumor, and a cornerstone in its treatment is radiotherapy. However, tumor cells surviving after irradiation indicates treatment failure; therefore, better understanding of the mechanisms regulating rad
Autor:
Nareg Pinarbasi Degirmenci, Ilknur Sur-Erdem, Vuslat Akcay, Yasemin Bolukbasi, Ugur Selek, Ihsan Solaroglu, Tugba Bagci-Onder
Glioblastoma is the most common type of primary brain tumor with an aggressive clinical course, and one of the cornerstones in its treatment regimen is radiotherapy. However, tumor cells surviving after radiation is an indicator of treatment failure;
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bb1fe9f9924b62fe964a9cf6493360ee
https://doi.org/10.1101/2022.04.16.488495
https://doi.org/10.1101/2022.04.16.488495
Autor:
Muhammad Jawad Hassan, Holger Thiele, Mohammad R. Toliat, Haseeb Anwar, Birgit Budde, Ilknur Sur-Erdem, M. Asif, Ludwig Eichinger, Arwa Ishaq A. Khayyat, Maria Iqbal, Peter Nürnberg, Muhammad Tariq, Angelika A. Noegel, Jamshaid Mahmood Baig, Ehtisham Ul Haq Makhdoom, Ilyas Ahmed, Muhammad Sher, Sarah Fischer, Uzma Abdullah, Christian Becker, Sigrid Tinschert, Stefan Höning, Muhammad Mohsin Ali Khan, Naveed Altaf Malik, Muhammad Jameel, Muhammad Sajid Hussain, Emrah Kaygusuz, Shahid Mahmood Baig
Publikováno v:
Clinical Genetics. 100:486-488
Jawad syndrome is a multiple congenital anomaly and intellectual disability syndrome with mutation in RBBP8 reported only in two families. Here, we report on two new families from Pakistan and identified a previously reported variant in RBBP8, NM_002
Publikováno v:
Neurological research. 44(8)
The role of white matter astrocytes in absence epilepsy is unknown. The present study aims to quantify astrocytic markers glial fibrillary acidic protein (GFAP), gap junction's proteins connexin 30 (Cx30) and connexin 43 (Cx43) in the corpus callosum
Publikováno v:
Synapse (New York, N.Y.)REFERENCES. 76(3-4)
Intercellular communication via gap junctions (GJs) has a wide variety of complex and essential functions in the CNS. In the present developmental study, we aimed to quantify the number of astrocytic GJs protein connexin 30 (Cx30) of genetic model of
Autor:
Birgit Budde, Ilknur Sur-Erdem, Muhammad Mohsin Ali Khan, Mohammad R. Toliat, Stefan Höning, Angelika A. Noegel, Muhammad Jawad Hassan, Christian Becker, Sarah Fischer, Muhammad Sher, Naveed Altaf Malik, Jamshaid Mahmood Baig, Peter Nürnberg, Ehtisham Ul Haq Makhdoom, Ilyas Ahmed, Sigrid Tinschert, Haseeb Anwar, Muhammad Sajid Hussain, Uzma Abdullah, Shahid Mahmood Baig, Muhammad Tariq, Holger Thiele, Emrah Kaygusuz, Muhammad Jameel, Arwa Ishaq A. Khayyat, Maria Iqbal, M. Asif, Ludwig Eichinger
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4eac63d60fab7642873248ec926e6542
https://doi.org/10.1111/cge.14028/v2/response1
https://doi.org/10.1111/cge.14028/v2/response1
Autor:
Filiz Senbabaoglu, Ezgi Kaya-Aksoy, Fidan Seker, Tolga Lokumcu, Sercin Karahuseyinoglu, Gizem Nur Sahin, Tugba Bagci-Onder, Ilknur Sur-Erdem, Ahmet Cingoz, Alisan Kayabolen
Publikováno v:
Cell Death Discovery, Vol 5, Iss 1, Pp 1-12 (2019)
Cell Death Discovery
Cell Death Discovery
Harakiri (HRK) is a BH3-only protein of the Bcl-2 family and regulates apoptosis by interfering with anti-apoptotic Bcl-2 and Bcl-xL proteins. While its function is mainly characterized in the nervous system, its role in tumors is ill-defined with fe