Human herpesvirus-6B protein U19 contains a p53 BOX I homology motif for HDM2 binding and p53 stabilization

Autor: Kofod-Olsen, Emil, Pettersson, Susanne, Wallace, Maura, Abduljabar, Ahmed Basim, Øster, Bodil, Hupp, Ted, Höllsberg, Per

Publikováno v: In Virology 5 January 2014 448:33-42

A Sensitive Quantification of HHV-6B by Real-time PCR

Autor: Øster Bodil, Höllsberg Per

Publikováno v: Biological Procedures Online, Vol 4, Iss 1, Pp 88-93 (2002)

Human herpesvirus (HHV)-6B is a pathogen causing latent infection in virtually all humans. Nevertheless, the interaction of HHV-6B with its host cells is poorly understood. Although HHV-6B is approximately 90% homologous to HHV-6A, it expresses certa

Externí odkaz: https://doaj.org/article/f32b35e8efa3487b8f4abf8aafe7a968

A Central Role for CK1 in Catalyzing Phosphorylation of the p53 Transactivation Domain at Serine 20 after HHV-6B Viral Infection

Autor: MacLaine, Nicola J., Øster, Bodil, Bundgaard, Bettina, Fraser, Jennifer A., Buckner, Carolyn, Lazo, Pedro A., Meek, David W., Höllsberg, Per, Hupp, Ted R.

Publikováno v: In Journal of Biological Chemistry 17 October 2008 283(42):28563-28573

Tumor-specific usage of alternative transcription start sites in colorectal cancer identified by genome-wide exon array analysis

Autor: Laurila Kirsti, Mansilla Francisco, Eller Asger, Pedersen Jakob, Lamy Philippe, Hansen Kristian Q, Wang Kai, Vang Søren, Rasmussen Mads H, Øster Bodil, Schepeler Troels, Thorsen Kasper, Wiuf Carsten, Laurberg Søren, Dyrskjøt Lars, Ørntoft Torben F, Andersen Claus L

Publikováno v: BMC Genomics, Vol 12, Iss 1, p 505 (2011)

Abstract Background Approximately half of all human genes use alternative transcription start sites (TSSs) to control mRNA levels and broaden the transcriptional output in healthy tissues. Aberrant expression patterns promoting carcinogenesis, howeve

Externí odkaz: https://doaj.org/article/b06ffd3760064e718d4cabbb00ff11b6

Keratin23 (KRT23) knockdown decreases proliferation and affects the DNA damage response of colon cancer cells:KRT23 in human colon cancer

Autor: Birkenkamp-Demtröder, Karin, Hahn, Stephan, Mansilla, Francisco, Thorsen, Kasper, Maghnouj, Abdelouahid, Christensen, Rikke, Øster, Bodil, Ørntoft, Torben Falck

Publikováno v: Birkenkamp-Demtröder, K, Hahn, S, Mansilla, F, Thorsen, K, Maghnouj, A, Christensen, R, Øster, B & Ørntoft, T F 2013, ' Keratin23 (KRT23) knockdown decreases proliferation and affects the DNA damage response of colon cancer cells : KRT23 in human colon cancer ', P L o S One, vol. 8, no. 9 . https://doi.org/10.1371/journal.pone.0073593

Keratin 23 (KRT23) is strongly expressed in colon adenocarcinomas but absent in normal colon mucosa. Array based methylation profiling of 40 colon samples showed that the promoter of KRT23 was methylated in normal colon mucosa, while hypomethylated i

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pure_au_____::807439ee06347c85ac05fd107c1d220d
https://pure.au.dk/portal/da/publications/keratin23-krt23-knockdown-decreases-proliferation-and-affects-the-dna-damage-response-of-colon-cancer-cells(c9be60ed-6b8c-4c19-9181-f4506bb49b01).html