Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Samuel K. Handelman"'
Autor:
Eun-Young Choi, Yanhua Chen, Asmita Pant, Annapurna Kuppa, Jonathan Z. Sexton, Lillias H. Maguire, Thomas L. Saunders, Samuel K. Handelman, Devika P. Bagchi, Elizabeth K. Speliotes, Lawrence F. Bielak, Yue Chen, Sean M McCarty, Rishel B. Vohnoutka, Xiaomeng Du, Brian D. Halligan, Vincent L. Chen, Yash Hegde
Human genome-wide association studies found SNPs near LYPLAL1 that have sex-specific effects on fat distribution and metabolic traits. To determine whether altering LYPLAL1 affects obesity and metabolic disease we created and characterized a mouse kn
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d6221f8d37a11b2b18d58c7dd95f3c84
https://doi.org/10.1101/2021.08.05.455257
https://doi.org/10.1101/2021.08.05.455257
Autor:
Elizabeth K. Speliotes, Samuel K. Handelman, Yanhua Chen, Andrew P. Wright, Michelle T. Long, Xiaomeng Du, Brian D. Halligan, Vincent L. Chen, Douglas P. Kiel
Publikováno v:
Hepatology Communications
Hepatology Communications, Vol 3, Iss 8, Pp 1073-1084 (2019)
Hepatology Communications, Vol 3, Iss 8, Pp 1073-1084 (2019)
Up to 25% of patients with nonalcoholic fatty liver disease (NAFLD) are not obese but may have a fat or muscle composition that predisposes them to NAFLD. Our aim was to determine whether body composition parameters associate with NAFLD and to identi
Autor:
Elizabeth K. Speliotes, Yue Chen, James G. Terry, Adolfo Correa, Bratati Kahali, Yii-Der Ida Chen, Vilmundur Gudnason, Yanhua Chen, Jeffrey R. O'Connell, Brian D. Halligan, Tamara B. Harris, Matthew A. Allison, Thomas H. Mosley, Xiuqing Guo, Matthew J. Budoff, Donald W. Bowden, J. Jeffrey Carr, Lenore J. Launer, Sharon L.R. Kardia, Kathleen A. Ryan, Jian Yang, Xiaomeng Du, Samuel K. Handelman, Breland F. Crudup, Laura M. Yerges-Armstrong, Lawrence F. Bielak, Yash Hegde, Shannon Carskadon, Ariella Cohain, Nicholette D. Palmer, Gudny Eiriksdottir, Solomon K. Musani, Eric E. Schadt, Nallasivam Palanisamy, Lynne E. Wagenknecht, Michael A. Province, Yi-Ping Fu, Christopher J. O'Donnell, Patricia A. Peyser, Albert V. Smith, Mary F. Feitosa, Jerome I. Rotter, Jiankang Liu, Andrew Bakshi, Lindsay Stetson, Jill M. Norris
Publikováno v:
J Clin Endocrinol Metab
Context Glycogen storage diseases are rare. Increased glycogen in the liver results in increased attenuation. Objective Investigate the association and function of a noncoding region associated with liver attenuation but not histologic nonalcoholic f
Autor:
Xiuqing Guo, Matthew J. Budoff, Lawrence F. Bielak, Abigail S. Baldridge, Elizabeth K. Speliotes, J. Jeffrey Carr, Thomas H. Mosley, Mary F. Feitosa, Lenore J. Launer, Sharon L.R. Kardia, Myriam Fornage, Albert V. Smith, Laura J. Rasmussen-Torvik, Yii-Der Ida Chen, Vilmundur Gudnason, Laura M. Yerges-Armstrong, Kathleen A. Ryan, Jerome I. Rotter, Tamara B. Harris, Matthew A. Allison, Ingrid B. Borecki, Brian D. Halligan, Samuel K. Handelman, Jie Yao, Lisa B. VanWagner, Patricia A. Peyser, Llilda Barata, Michael A. Province, Nicholette D. Palmer, Jeffrey R. O'Connell
Publikováno v:
Hepatology Communications
Hepatology communications, vol 3, iss 7
Hepatology Communications, Vol 3, Iss 7, Pp 894-907 (2019)
Hepatology communications, vol 3, iss 7
Hepatology Communications, Vol 3, Iss 7, Pp 894-907 (2019)
The accumulation of excess fat in the liver (hepatic steatosis) in the absence of heavy alcohol consumption causes nonalcoholic fatty liver disease (NAFLD), which has become a global epidemic. Identifying metabolic risk factors that interact with the
Publikováno v:
Gastroenterology. 158:S-1272
Autor:
Philip M. Brown, Olga V. Belyaeva, Cara Nestlerode, Natalia Y. Kedishvili, Katherine Valles, Yaron Rotman, Arun J. Sanyal, Yanhua Chen, Emmanuel Thomas, Yanling Ma, Vincent L. Chen, Ynto S. de Boer, Elizabeth K. Speliotes, Suman Karki, Joseph M. Zmuda, Christopher Koh, Xiaomeng Du, Koji Fujita, Samuel K. Handelman, David E. Kleiner, T. Jake Liang
Publikováno v:
Hepatology (Baltimore, Md.). 69(4)
Non-alcoholic fatty liver disease (NAFLD) is a common cause of chronic liver disease. A single nucleotide polymorphism (SNPs), rs6834314, was associated with serum liver enzymes in the general population, presumably reflecting liver fat or injury. We