Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Tara B. Hendry-Hofer"'
Autor:
Tara B. Hendry-Hofer, Subrata Bhadra, Adriano Chan, Sari B. Mahon, Matthew Brenner, Jangwoen Lee, Gerry R. Boss, Renate B. Pilz, Nesta Bortey-Sam, Brian A. Logue, Vikhyat S. Bebarta
Publikováno v:
Clin Toxicol (Phila)
Clinical toxicology (Philadelphia, Pa.), vol 60, iss 3
Clinical toxicology (Philadelphia, Pa.), vol 60, iss 3
ContextHydrogen cyanide and methanethiol are two toxic gases that inhibit mitochondrial cytochrome c oxidase. Cyanide is generated in structural fires and methanethiol is released by decaying organic matter. Current treatments for cyanide exposure do
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dee8add4cc1470c4d7305ee4d92fefa0
https://europepmc.org/articles/PMC8800944/
https://europepmc.org/articles/PMC8800944/
Autor:
Raymond C. Rancourt, Matthew D. McGraw, Wesley W. Holmes, Jacqueline S. Rioux, Carol Burns, Rhonda B. Garlick, Russell W. Smith, Dana R. Anderson, Jorge Di Paola, Tara B. Hendry-Hofer, Livia A. Veress, Christopher M. Osborne, Carl W. White, Emily J. Mastej, Danielle C. Paradiso, Gates B. Roe
Publikováno v:
Toxicological Sciences. 159:461-469
Sulfur mustard (SM) is a chemical warfare agent. When inhaled, SM causes significant injury to the respiratory tract. Although the mechanism involved in acute airway injury after SM inhalation has been well described previously, the mechanism of SM
Autor:
Livia A. Veress, Raymond C. Rancourt, Carl W. White, Jacqueline S. Rioux, Rhonda B. Garlick, Aftab Ahmad, Tara B. Hendry-Hofer
Publikováno v:
Toxicology and Applied Pharmacology. 272:86-95
Sulfur mustard (SM) inhalation causes airway injury, with enhanced vascular permeability, coagulation, and airway obstruction. The objective of this study was to determine whether recombinant tissue factor pathway inhibitor (TFPI) could inhibit this
Autor:
Carl W. White, Tara B. Hendry-Hofer, Jacqueline S. Rioux, Rhonda B. Garlick, Livia A. Veress, Joan E. Loader
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology. 48:439-447
Sulfur mustard (SM) inhalation causes the rare but life-threatening disorder of plastic bronchitis, characterized by bronchial cast formation, resulting in severe airway obstruction that can lead to respiratory failure and death. Mortality in those r
Autor:
Joan E. Loader, Tara B. Hendry-Hofer, Brian J. Day, Heidi C. O'Neill, David J. Orlicky, Carl W. White
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology. 45:323-331
The inhalation of sulfur mustard (SM) causes substantial deposition in the nasal region. However, specific injury has not been characterized. 2-chloroethyl ethyl sulfide (CEES) is an SM analogue used to model injury and screen potential therapeutics.
Autor:
Tara B. Hendry-Hofer, Raymond C. Rancourt, Joan E. Loader, Livia A. Veress, Carl W. White, Heidi C. O'Neill
Publikováno v:
American Journal of Respiratory and Critical Care Medicine. 182:1352-1361
Sulfur mustard (SM) is a frequently used chemical warfare agent, even in modern history. SM inhalation causes significant respiratory tract injury, with early complications due to airway obstructive bronchial casts, akin to those seen after smoke inh
Autor:
Brian J. Day, Elysia Min, Heidi C. O'Neill, Raymond C. Rancourt, Livia A. Veress, Tara B. Hendry-Hofer, Jie Huang, Joan E. Loader, Carl W. White
Publikováno v:
Free Radical Biology and Medicine. 48:1188-1196
Sulfur mustard (bis-2-(chloroethyl) sulfide, SM) is a highly reactive vesicating and alkylating chemical warfare agent. A SM analog, 2-chloroethyl ethyl sulfide (CEES) has been utilized to elucidate mechanisms of toxicity and as a screen for therapeu
Autor:
Carl W. White, Livia A. Veress, Xiaoling Guo, Tara N. Jones, Raymond C. Rancourt, Tara B. Hendry-Hofer
Publikováno v:
American journal of physiology. Lung cellular and molecular physiology. 302(1)
Acute lung injury is a principal cause of morbidity and mortality in response to mustard gas (SM) inhalation. Obstructive, fibrin-containing airway casts have recently been reported in a rat inhalation model employing the SM analog 2-chloroethyl ethy