Zobrazeno 1 - 10
of 92
pro vyhledávání: '"Yunzeng Zou"'
Autor:
Baoli, Zhang, Peng, Yu, Enyong, Su, Jianguo, Jia, Chunyu, Zhang, Shiyao, Xie, Zhenhui, Huang, Ying, Dong, Jinguo, Ding, Yunzeng, Zou, Hong, Jiang, Junbo, Ge
Publikováno v:
Current Pharmaceutical Design. 28:751-759
Background and Objective: Myocardial infarction (MI) leads to pathological cardiac remodeling and heart failure. Sodium tanshinone IIA sulfonate (STS) shows to possess therapeutic potential. The present study aimed to explore the potential role of ST
Autor:
Xue Yang, Baoli Zhang, Peng Yu, Ming Liu, Chunyu Zhang, Enyong Su, Shiyao Xie, Yunzeng Zou, Hong Jiang, Junbo Ge
Publikováno v:
Biochemical and biophysical research communications. 611
Pressure overload induced cardiac remodeling is associated with a complex spectrum of pathophysiological mechanisms. As inflammatory cells, macrophages maintain a critical position in mechanical stress-induced myocardial remodeling. HMGB1 is a highly
Autor:
Yunzeng Zou, Lei Zhang, Jingfeng Wang, Baoli Zhang, Jian Wu, Ying Yu, Junbo Ge, Hong Jiang, Yangang Su
Publikováno v:
International Heart Journal. 62:162-170
High-mobility group box 1 (HMGB1) is increased in the myocardium under pressure overload (PO) and is involved in PO-induced cardiac remodeling. The mechanisms of the upregulation of cardiac HMGB1 expression have not been fully elucidated. In the pres
Autor:
Xuan Li, Fangjie Dai, Hao Wang, Ge Wei, Qiu Jiang, Peipei Yin, Shijun Wang, Junbo Ge, Cheng Yang, Jian Wu, Yunzeng Zou
Publikováno v:
Clinical and Translational Medicine. 12
Publikováno v:
Journal of Cellular and Molecular Medicine
To investigate the prevention of cardiac remodelling and inflammatory immune response after myocardial infarction (MI) via ACEI regulating dendritic cells (DCs), we explored whether the protective effect of ACEI was repressed under hyperlipidemic env
Publikováno v:
Journal of Cellular and Molecular Medicine
Exercise training is believed to have a positive effect on cardiac hypertrophy after hypertension. However, its mechanism is still not fully understood. Herein, our findings suggest that heat shock transcription factor 1 (HSF1) improves exercise‐in
Autor:
Junbo Ge, Wenlong Yang, Shuning Zhang, Yunzeng Zou, Hao Jiang, Juying Qian, Jianbing Zhu, Daile Jia, Zhiyong Qi, Tiantong Ou, Aijun Sun
Publikováno v:
Journal of Molecular and Cellular Cardiology. 134:119-130
Trimethylamine N-oxide (TMAO), a gut microbe-derived metabolite of dietary choline and other trimethylamine-containing nutrients, has been associated with poor prognosis in coronary heart disease. However, the role and underlying mechanisms of TMAO i
Autor:
Yi Shen, Xiang Wang, Chao Yin, Yan Zou, Juying Qian, Yunzeng Zou, Ying Wang, Hao Wang, Jianguo Jia, Chenxing Huang, Xuejuan Jin, Junbo Ge, Le Pan, Hui Gong
Publikováno v:
Cell Death and Disease, Vol 12, Iss 10, Pp 1-14 (2021)
Cell Death & Disease
Cell Death & Disease
Progressive cardiac fibrosis accelerates the development of heart failure. Here, we aimed to explore serum Wnt5a and Wnt11 levels in hypertension patients, the roles of Wnt5a and Wnt11 in cardiac fibrosis and potential mechanisms under pressure overl
Autor:
Aijun Sun, Zheng Dong, Leilei Ma, Junbo Ge, Xingxu Wang, Yunzeng Zou, Kai-Yue Xin, Fei-juan Kong
Publikováno v:
Cell Proliferation
Background Ischaemic preconditioning elicited by brief periods of coronary occlusion and reperfusion protects the heart from a subsequent prolonged ischaemic insult. Here, we test the hypothesis that short‐term non‐ischaemic stimulation of hypert
Autor:
Junbo Ge, Xiangdong Yang, Hui Li, Mieradilijiang Abudupataer, Timothy C. Wang, Shuning Zhang, Zheliang Zhou, Lili Xu, Suling Ding, Yunzeng Zou, Weiwei Zhang, Tao Hong, Jinmiao Chen
Publikováno v:
Cell Death and Disease, Vol 9, Iss 6, Pp 1-20 (2018)
Cell Death & Disease
Cell Death & Disease
Histamine is a widely distributed biogenic amine involved in the regulation of an array of biological processes. Serum histamine level is markedly elevated in the early stages of acute myocardial infarction, whereas the role it plays remains unclear.