Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Anna Siskova"'
Autor:
Anna Siskova, Jan Kral, Jana Drabova, Klara Cervena, Kristyna Tomasova, Jiri Jungwirth, Tomas Hucl, Pavel Kohout, Sandra Summerova, Ludmila Vodickova, Pavel Vodicka, Veronika Vymetalkova
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 14; Pages: 7656
A colorectal adenoma, an aberrantly growing tissue, arises from the intestinal epithelium and is considered as precursor of colorectal cancer (CRC). In this study, we investigated structural and numerical chromosomal aberrations in adenomas, hypothes
Autor:
Miriam J. Kavec, Marketa Urbanova, Pavol Makovicky, Alena Opattová, Kristyna Tomasova, Michal Kroupa, Klara Kostovcikova, Anna Siskova, Nazila Navvabi, Michaela Schneiderova, Veronika Vymetalkova, Ludmila Vodickova, Pavel Vodicka
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 10; Pages: 5704
Oxidative stress, oxidative DNA damage and resulting mutations play a role in colorectal carcinogenesis. Impaired equilibrium between DNA damage formation, antioxidant status, and DNA repair capacity is responsible for the accumulation of genetic mut
Publikováno v:
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 21, Iss 3260, p 3260 (2020)
International Journal of Molecular Sciences, Vol 21, Iss 3260, p 3260 (2020)
Colorectal cancer (CRC) is a malignant disease with an incidence of over 1.8 million new cases per year worldwide. CRC outcome is closely related to the respective stage of CRC and is more favorable at less advanced stages. Detection of early colorec
Autor:
Alena Opattova, Veronika Vymetalkova, Michaela Schneiderova, Markéta Urbanová, Kristyna Tomasova, Ludmila Vodickova, Pavol Makovicky, Rudolf Stetina, Klara Kostovcikova, Anna Siskova, Pavel Vodicka, Michal Kroupa
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 2473, p 2473 (2020)
International Journal of Molecular Sciences
International Journal of Molecular Sciences
Oxidative stress with subsequent premutagenic oxidative DNA damage has been implicated in colorectal carcinogenesis. The repair of oxidative DNA damage is initiated by lesion-specific DNA glycosylases (hOGG1, NTH1, MUTYH). The direct evidence of the