Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Yongzhi Cui"'
Autor:
Sandra Burkett, Haven R. Garber, Masahiro Onozawa, Leigh Samsel, Xiaolin Wu, J. Philip McCoy, Ziyao Wang, Peter D. Aplan, Crystal L. Mackall, Yongzhi Cui
Publikováno v:
Blood. 125:2958-2967
T-cell receptors (TCRs) and chimeric antigen receptors recognizing tumor-associated antigens (TAAs) can now be engineered to be expressed on a wide array of immune effectors. Engineered receptors targeting TAAs have most commonly been expressed on ma
Autor:
Warren J. Leonard, Kairong Cui, Wei Liao, Dustin E. Schones, Tae-Young Roh, Keji Zhao, Yongzhi Cui, Jangsuk Oh
Publikováno v:
Nature immunology
T helper type 2 (T(H)2) cells are essential for humoral immunity and host defense. Interleukin 4 (IL-4) drives T(H)2 differentiation and IL-2 augments the accessibility of Il4 chromatin. Here we demonstrate that IL-2, by inducing binding of STAT5 to
Autor:
Greg Riedlinger, Chu-Xia Deng, Gertraud W. Robinson, Lothar Hennighausen, Wei Tang, Yongzhi Cui, Keiko Miyoshi, Cuiling Li
Publikováno v:
Molecular and Cellular Biology. 24:8037-8047
This study explored the functions of the signal transducers and activators of transcription 5a and 5b (referred to as Stat5 here) during different stages of mouse mammary gland development by using conditional gene inactivation. Mammary gland morphog
Publikováno v:
Cancer Research
Cancer Research, American Association for Cancer Research, 2005, 65 (15), pp.6864-6873. ⟨10.1158/0008-5472.CAN-05-0181⟩
Cancer Research, American Association for Cancer Research, 2005, 65 (15), pp.6864-6873. ⟨10.1158/0008-5472.CAN-05-0181⟩
The tumor suppressor phosphatase and tensin homologue (PTEN) is involved in cell proliferation, adhesion, and apoptosis. PTEN overexpression in mammary epithelium leads to reduced cell number and impaired differentiation and secretion. In contrast, o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8fe2c610a47a54640aae07f9a6246dcc
https://hal.inrae.fr/hal-02682540
https://hal.inrae.fr/hal-02682540
Autor:
Jurg Rohrer, Raquel Hontecillas, Yongzhi Cui, Kathryn A Reynolds, Josep Bassaganya-Riera, Alejandro Uribe Benninghoff, Susan Martino-Catt, Frank J. Gonzalez, Lothar Hennighausen
Publikováno v:
Gastroenterology. 127(3)
The molecular targets for the protective actions of conjugated linoleic acid (CLA) on experimental inflammatory bowel disease (IBD) are unknown. We used a loss-of-function approach to investigate whether CLA ameliorated colitis through a peroxisome p