Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Bingjie Lv"'
Autor:
Muyang Gu, Guo-Ping Shi, Yuzhi Lu, Jingyong Li, Jiao Jiao, Ni Xia, Xiang-Ping Yang, Bingjie Lv, Shaofang Nie, Yu Hu, Dan Li, Shuang Wen, Tingting Tang, Xiang Cheng, Mengyang Liao, Yuhua Liao
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 39:446-458
Objective— Inflammation occurs during the progression of abdominal aortic aneurysm (AAA). IL (interleukin)-33 is a pleiotropic cytokine with multiple immunomodulatory effects, yet its role in AAA remains unknown. Approach and Results— Immunoblot,
Autor:
Qianqian Li, Xin Tu, Xiang Cheng, Shaofang Nie, Tingting Tang, Ni Xia, Zhipeng Zeng, Yuan Yuan Li, Ke Wang, Min Zhang, Bingjie Lv
Publikováno v:
Journal of Biological Chemistry. 292:6004-6013
CD4+ T cells are abnormally activated in patients with dilated cardiomyopathy (DCM) and might be associated with the immunopathogenesis of the disease. However, the underlying mechanisms of CD4+ T cell activation remain largely undefined. Our aim was
Publikováno v:
Medicine
Rationale: Hantaviruses cause two forms of diseases in humans, namely hemorrhagic fever with renal syndrome (HFRS) and hantavirus pulmonary syndrome. Hantavirus infections can occur in pregnant women, and could influence the maternal and fetal outcom
Autor:
Yuzhi Lu, Tongjie Xu, Yuhua Liao, Shaofang Nie, Shuang Wen, Ni Xia, Bing Sun, Bingjie Lv, Chenping Jia, Wenyong Dong, Daan Nie, Yuan Yuan Li, Jiao Jiao, Longxian Cheng, Tingting Tang, Zhengfeng Zhu, Xiang Cheng, Wen-cai Zhang, Su-Feng Zhou
Publikováno v:
Cardiovascular research. 106(3)
Aims Recently, interleukin (IL)-9 was found to be involved in the pathogenesis of many inflammatory diseases. Here, we tested whether IL-9 was related to atherosclerosis and investigated the underlying mechanisms. Methods and results IL-9R was expres
Publikováno v:
Science China. Life sciences. 57(8)
Aortic aneurysms are common and life threatening problems with high rates of death. The initiation and progression of aneurysms are characterized by extensive extracellular matrix degradation and immune cells invasion within arterial wall. During the