Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Bradley T. Andresen"'
Publikováno v:
The FASEB Journal. 32
Autor:
Justin L. Grobe, Christopher C. Barney, Bradley T. Andresen, Elizabeth M. Schanhals, Michael Traver
Publikováno v:
Physiological Reports
The effects of heat acclimation on water intake and urine output responses to thermal dehydration and other thirst stimuli were studied in male Sprague–Dawley rats. Rats were heat acclimated by continuous exposure to a 34°C environment for at leas
Publikováno v:
Journal of Pharmacology and Experimental Therapeutics. 309:425-431
Previous studies indicate that angiotensin (AT)(1) receptor-induced activation of phospholipase D (PLD) may importantly contribute to vascular hypertrophy, injury, and contraction. However, the role of AT(2) receptors in regulating AT(1) receptor-ind
Autor:
James R. Sowers, Catherine Sinak, Alex Meuth, Thomas J. Thekkumkara, Robert C. Speth, Bradley T. Andresen, Ravi Nistala, Lakshmi Pulakat, Chirag Mandavia, Adam Whaley-Connell
Blockade of the angiotensin (ANG) II receptor type 1 (AT1R) with angiotensin receptor blockers (ARBs) is widely used in the treatment of hypertension. However, ARBs are variably effective in reducing blood pressure, likely due, in part, to polymorphi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::446d58686d54c02d8822ec3b9cba5302
https://europepmc.org/articles/PMC3625841/
https://europepmc.org/articles/PMC3625841/
Autor:
Bradley T. Andresen, Catherine E. Erickson, Tammy Liu, Jenny P. Nguyen, Murat Bastepe, Christopher P. Blessing, Lakshmi Pulakat, Edwin K. Jackson, Rukhsana Gul
Publikováno v:
PLoS ONE, Vol 8, Iss 8, p e71980 (2013)
PLoS ONE
PLoS ONE
Nebivolol, a third generation β-adrenoceptor (β-AR) antagonist (β-blocker), causes vasodilation by inducing nitric oxide (NO) production. The mechanism via which nebivolol induces NO production remains unknown, resulting in the genesis of much of
Publikováno v:
Experimental biology and medicine (Maywood, N.J.). 235(10)
Genetic factors are important in the pathogenesis of diabetic retinopathy (DR); there is a clear association of increased expression of vascular endothelial growth factor (VEGF) with DR as well as numerous VEGF polymorphisms that are linked to increa
Publikováno v:
World journal of cardiology. 2(12)
Reactive oxygen species (ROS) have long been proposed to be mediators of experimental cardiovascular pathology. There is also a wealth of data indicating that ROS are involved in clinical cardiovascular pathology. However, multiple clinical studies h
Autor:
Javad Habibi, Randall L. Heller, S. A. Cooper, Vincent G. DeMarco, James R. Sowers, James P. Bosanquet, Kimberly Delcour, Rebecca I. Schneider, Melvin R. Hayden, Bradley T. Andresen, Kevin C. Dellsperger, Adam Whaley-Connell
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 294(6)
The transgenic (mRen2)27 (Ren2) rat overexpresses mouse renin in extrarenal tissues, causing increased local synthesis of ANG II, oxidative stress, and hypertension. However, little is known about the role of oxidative stress induced by the tissue re
Autor:
Bradley T. Andresen, Nathan Rehmer, Vincent G. DeMarco, Carlos M. Ferrario, Ravi Nistala, Melvin R. Hayden, Yongzhong Wei, Adam Whaley-Connell, Poorna R. Karuparthi, Javad Habibi, James R. Sowers, Shawna A. Cooper
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 51(2)
Activation of reduced nicotinamide-adenine dinucleotide phosphate (NADPH) oxidase by angiotensin II is integral to the formation of oxidative stress in the vasculature and the kidney. 3-Hydroxy-3-methylglutaryl-coenzyme A reductase inhibition is asso
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 324(1)
Angiotensin II can cause hypertension through enhanced vasoconstriction of renal vasculature. One proposed mechanism for reduction of angiotensin II-induced hypertension is through inhibition of the mitogen-activated protein kinase kinase (MEK)/extra