Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Homira Behbahani"'
Autor:
Swetha Vijayaraghavan, Azadeh Karami, Shahin Aeinehband, Homira Behbahani, Alf Grandien, Bo Nilsson, Kristina N Ekdahl, Rickard P F Lindblom, Fredrik Piehl, Taher Darreh-Shori
Publikováno v:
PLoS ONE, Vol 8, Iss 6, p e65936 (2013)
Acetylcholine (ACh), the classical neurotransmitter, also affects a variety of nonexcitable cells, such as endothelia, microglia, astrocytes and lymphocytes in both the nervous system and secondary lymphoid organs. Most of these cells are very distan
Externí odkaz:
https://doaj.org/article/3c04f7e8fbc744c1ba9aaf71448958b7
Autor:
Susanne Frykman, Ji-Yeun Hur, Jenny Frånberg, Mikio Aoki, Bengt Winblad, Jarmila Nahalkova, Homira Behbahani, Lars O Tjernberg
Publikováno v:
PLoS ONE, Vol 5, Iss 1, p e8948 (2010)
A key player in the development of Alzheimer's disease (AD) is the gamma-secretase complex consisting of at least four components: presenilin, nicastrin, Aph-1 and Pen-2. gamma-Secretase is crucial for the generation of the neurotoxic amyloid beta-pe
Externí odkaz:
https://doaj.org/article/9e22371927fa4938a8616fb043913923
Autor:
Anna Sandebring, Kelly Jean Thomas, Alexandra Beilina, Marcel van der Brug, Megan M Cleland, Rili Ahmad, David W Miller, Ibardo Zambrano, Richard F Cowburn, Homira Behbahani, Angel Cedazo-Mínguez, Mark R Cookson
Publikováno v:
PLoS ONE, Vol 4, Iss 5, p e5701 (2009)
PTEN-induced novel kinase 1 (PINK1) mutations are associated with autosomal recessive parkinsonism. Previous studies have shown that PINK1 influences both mitochondrial function and morphology although it is not clearly established which of these are
Externí odkaz:
https://doaj.org/article/186a9b17a4b94e1aa0e80a3257dd5884
Autor:
Maria Eriksdotter, Sumonto Mitra, Lars Wahlberg, Lars O. Tjernberg, Bengt Linderoth, Homira Behbahani, Manuel Navarro-Oviedo
Publikováno v:
Experimental Cell Research. 371:175-184
Alzheimer's disease (AD) is characterized by early degeneration of cholinergic neurons and decreased levels of nerve growth factor (NGF). Thus, increasing the NGF levels by for instance encapsulated cell bio-delivery (ECB) is a potential treatment st
Autor:
Maria Eriksdotter, Ruchi Gera, Bengt Linderoth, Sumonto Mitra, Lars Wahlberg, Homira Behbahani, Per Almqvist, Göran Lind
Publikováno v:
Advances in Experimental Medicine and Biology ISBN: 9783030740450
Age-dependent progressive neurodegeneration and associated cognitive dysfunction represent a serious concern worldwide. Currently, dementia accounts for the fifth highest cause of death, among which Alzheimer’s disease (AD) represents more than 60%
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f96aa3f3d657f80a71da8fbd561b9b75
https://doi.org/10.1007/978-3-030-74046-7_11
https://doi.org/10.1007/978-3-030-74046-7_11
Autor:
Bengt Linderoth, Homira Behbahani, Maria Eriksdotter, Sumonto Mitra, Silvia Turchetto, Lars Wahlberg
Publikováno v:
Alzheimer's & Dementia. 16
Autor:
Homira Behbahani, Charlotte Forsell, Eva-Britt Samuelsson, Saara Tikka, Kirsten Coupland, Mahmod Panahi, Erik Sundström, Naeimeh Yousefi Mesri, Helena Karlström, Matti Viitanen, Bengt Winblad, Caroline Graff
Publikováno v:
Journal of Cellular and Molecular Medicine
Cerebral autosomal-dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) is a familial fatal progressive degenerative disorder. One of the pathological hallmarks of CADASIL is a dramatic reduction of vascular smooth muscle
Autor:
Vesna Jelic, Petter Järemo, Homira Behbahani, Bengt Winblad, Alenka Jejcic, Magnus Oweling, Tasmin Shahnaz
Publikováno v:
Current Alzheimer research. 16(9)
Background: Alzheimer’s Disease (AD) features the accumulation of β-amyloid in erythrocytes. The subsequent red cell damage may well affect their oxygen-carrying capabilities. 2,3- diphosphoglycerate (2,3-DPG) binds to the hemoglobin thereby promo
Autor:
Petter Järemo, Bengt Winblad, Magnus Oweling, Tasmin Shahnaz, Homira Behbahani, Vesna Jelic, Alenka Jejcic
Publikováno v:
Current Alzheimer research. 16(11)
Introduction: We recently showed that Amyloid Beta (Aβ)40 accumulates in erythrocytes and possibly causes cell damage as evidenced by an increased number of assumed injured low-density (kg/L) erythrocytes. Furthermore, we have suggested a separation
Publikováno v:
Frontiers in Neuroscience
Frontiers in Neuroscience, Vol 13 (2019)
Frontiers in Neuroscience, Vol 13 (2019)
Alzheimer’s disease (AD) is a progressive neurodegenerative disorder associated with abnormal protein modification, inflammation and memory impairment. Aggregated amyloid beta (Aβ) and phosphorylated tau proteins are medical diagnostic features. L