Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Rafael Baltiérrez-Hoyos"'
Autor:
Juan Manuel Velázquez-Enríquez, Alma Aurora Ramírez-Hernández, Luis Manuel Sánchez Navarro, Itayetzi Reyes-Avendaño, Karina González-García, Cristian Jiménez-Martínez, Luis Castro-Sánchez, Xariss Miryam Sánchez-Chino, Verónica Rocío Vásquez-Garzón, Rafael Baltiérrez-Hoyos
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 9, p 5032 (2022)
Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive, irreversible lung disorder of unknown cause. This disease is characterized by profibrotic activation of resident pulmonary fibroblasts resulting in aberrant deposition of extracellular ma
Externí odkaz:
https://doaj.org/article/68861dc5404249308d3a26ea34474fde
Autor:
Juan Manuel Velázquez-Enríquez, Jovito Cesar Santos-Álvarez, Alma Aurora Ramírez-Hernández, Edilburga Reyes-Jiménez, Armando López-Martínez, Socorro Pina-Canseco, Sergio Roberto Aguilar-Ruiz, María de los Ángeles Romero-Tlalolini, Luis Castro-Sánchez, Jaime Arellanes-Robledo, Verónica Rocío Vásquez-Garzón, Rafael Baltiérrez-Hoyos
Publikováno v:
Biomedicines, Vol 9, Iss 8, p 1058 (2021)
Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive, irreversible, and highly fatal disease. It is characterized by the increased activation of both fibroblast and myofibroblast that results in excessive extracellular matrix (ECM) depositio
Externí odkaz:
https://doaj.org/article/c1023706b4024e238c768343304cdafd
Autor:
Jaime Arellanes-Robledo, Luis Castro-Sánchez, Armando López-Martínez, Rafael Baltiérrez-Hoyos, Edilburga Reyes-Jiménez, Verónica R. Vásquez-Garzón, Juan Manuel Velázquez-Enríquez, Jovito Cesar Santos-Álvarez, Alma Aurora Ramírez-Hernández, Sergio R. Aguilar-Ruiz, Socorro Pina-Canseco, María de Los Ángeles Romero-Tlalolini
Publikováno v:
Biomedicines, Vol 9, Iss 1058, p 1058 (2021)
Biomedicines
Volume 9
Issue 8
Biomedicines
Volume 9
Issue 8
Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive, irreversible, and highly fatal disease. It is characterized by the increased activation of both fibroblast and myofibroblast that results in excessive extracellular matrix (ECM) depositio