Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Shadi Salloum"'
Autor:
Wenting Li, Xiaoqiong Duan, Chuanlong Zhu, Xiao Liu, Andre J. Jeyarajan, Min Xu, Zeng Tu, Qiuju Sheng, Dong Chen, Chuanwu Zhu, Tuo Shao, Zhimeng Cheng, Shadi Salloum, Esperance A. Schaefer, Annie J. Kruger, Jacinta A. Holmes, Raymond T. Chung, Wenyu Lin
Publikováno v:
J Immunol
Hepatitis B virus (HBV)/hepatitis C virus (HCV) coinfection accelerates liver fibrosis progression compared with HBV or HCV monoinfection. Octamer binding transcription factor 4 (OCT4) and Nanog are direct targets of the profibrogenic TGF-β1 signali
Autor:
Nadia Alatrakchi, Tuo Shao, Myung-Ho Kim, Wenyu Lin, Shadi Salloum, Raymond T. Chung, Annie J. Kruger, Andre J Jeyarajan, Kathleen E. Corey, Stuti Shroff, Andrew Kassa, Zhu Zhuo, Sanjoy K. Khan, Mozhdeh Sojoodi, Jacinta A Holmes, Esperance A. Schaefer
Publikováno v:
Cellular and Molecular Gastroenterology and Hepatology, Vol 12, Iss 4, Pp 1297-1310 (2021)
Cellular and Molecular Gastroenterology and Hepatology
Cellular and Molecular Gastroenterology and Hepatology
Background & Aims Patients with simple steatosis (SS) and nonalcoholic steatohepatitis can develop progressive liver fibrosis, which is associated with liver-related mortality. The mechanisms contributing to liver fibrosis development in SS, however,
Autor:
Akif Altinbas, Kara W. Chew, Isabel S Seguin, Shadi Salloum, Nadia Alatrakchi, Ma Somsouk, Anna Lidofsky, Esperance A. Schaefer, Steven G. Deeks, Raymond T. Chung, Annie J. Kruger, Peter W. Hunt, Jenna L. Gustafson, Jacinta A. Holmes, Eoin R. Feeney, Hui Zheng, Kathleen E. Corey, Ricard Masia
Publikováno v:
The Journal of infectious diseases, vol 218, iss 9
BACKGROUND: Coinfection with human immunodeficiency virus (HIV) accelerates hepatitis C virus (HCV)–related liver fibrosis. Macrophages are triggered during both viral infections and are critical in liver inflammation/fibrogenesis. Liver fibrosis s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::714f9bbf778e71afa1b0494a7ec1f09d
https://europepmc.org/articles/PMC6151081/
https://europepmc.org/articles/PMC6151081/
Autor:
Michael Roggendorf, Robert Thimme, Jörg Timm, David N. Frick, Paul Klenerman, Christoph Neumann-Haefelin, Jing Jing Wang, Markus Reiser, Andrea Biezynski, Oliver G. Pybus, Sergei Viazov, Thomas Eiermann, Gagandeep Narula, Shadi Salloum, Anna Eckart
Failure of the adaptive immune response to control infection with the hepatitis C virus (HCV) can result from mutational escape in targeted T-cell epitopes. Recent studies suggest that T-cell immune pressure is an important factor in the evolution of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6adc28c329b56b964fb68fdf6dcde8e9
https://ora.ox.ac.uk/objects/uuid:6f66b11b-717c-488c-867f-eae4fc410dce
https://ora.ox.ac.uk/objects/uuid:6f66b11b-717c-488c-867f-eae4fc410dce
The detection and quantification of hepatitis C virus (HCV) core antigen in serum or plasma by the use of different assay formats have previously been shown to represent useful markers of viral replication. In the present study, the intrinsic perform
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::76d6dde5c93c2fdcdee7bcc865370d02
https://www.ncbi.nlm.nih.gov/pubmed/20107102
https://www.ncbi.nlm.nih.gov/pubmed/20107102