Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Muhammad Asaduzzaman"'
Publikováno v:
British Journal of Pharmacology. 156:307-315
Neutrophil-mediated lung injury is an insidious feature in sepsis although the mechanisms regulating pulmonary recruitment of neutrophils remain elusive. Here, we investigated the role of P-selectin glycoprotein-ligand-1 (PSGL-1) in sepsis-induced ne
Autor:
Morley D. Hollenberg, John R. Gordon, Harissios Vliagoftis, Yingqi Wu, Muhammad Asaduzzaman, Narcy Arizmendi, Daniel B. Polley, Lisa Cameron, Courtney Davidson
Publikováno v:
Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology. 43(11)
SummaryBackground Many aeroallergens contain proteinase activity and are able to induce allergic sensitization when presented to mucosal surfaces. Some of these allergens activate proteinase-activated receptor-2 (PAR2). Objective To determine the rol
Autor:
Muhammad Asaduzzaman, Milladur Rahman, Henrik Thorlacius, Su Zhang, Shahram Lavasani, Oscar Ö. Braun, Bengt Jeppsson
Publikováno v:
Critical care medicine. 37(4)
Objective. Recent findings Indicate that platelets not only regulate thrombosis and hemostasis but may also be involved in proinflammatory activities. Herein, we hypothesized that platelets may play a role in sepsis by activating and priming circulat
Publikováno v:
Shock (Augusta, Ga.). 30(3)
Neutrophil-mediated lung damage is an insidious feature in septic patients, although the adhesive mechanisms behind pulmonary recruitment of neutrophils in polymicrobial sepsis remain elusive. The aim of the present study was to define the role of ly
Publikováno v:
Critical care medicine. 36(2)
OBJECTIVE: Leukocyte-mediated tissue damage is a key feature in septic lung injury, although the signaling mechanisms behind pulmonary recruitment of leukocytes remain elusive. The aim of the present study was to define the role of p38 mitogen-activa