Zobrazeno 1 - 2
of 2
pro vyhledávání: '"Alhussaini, A."'
Autor:
Jose Gomez-Arroyo, Donatas Kraskauskas, Norbert F. Voelkel, Takeshi Ishizaki, Aysar Alhussaini, Herman J Bogaard, Shiro Mizuno
Publikováno v:
Mizuno, S, Bogaard, H J, Kraskauskas, D, Alhussaini, A, Gomez-Arroyo, J, Voelkel, N F & Ishizaki, T 2011, ' p53 Gene deficiency promotes hypoxia-induced pulmonary hypertension and vascular remodeling in mice ', American Journal of Physiology. Lung Cellular and Molecular Physiology, vol. 300, no. 5, pp. L753-L761 . https://doi.org/10.1152/ajplung.00286.2010
American Journal of Physiology. Lung Cellular and Molecular Physiology, 300(5), L753-L761. American Physiological Society
American Journal of Physiology. Lung Cellular and Molecular Physiology, 300(5), L753-L761. American Physiological Society
Chronic hypoxia induces pulmonary arterial remodeling, resulting in pulmonary hypertension and right ventricular hypertrophy. Hypoxia has been implicated as a physiological stimulus for p53 induction and hypoxia-inducible factor-1α (HIF-1α). Howeve
Autor:
Donatas Kraskauskas, Daniela Farkas, Harm Jan Bogaard, Norbert F. Voelkel, Laszlo Farkas, Aysar Alhussaini, Jose Gomez-Arroyo
Publikováno v:
American Journal of Physiology. Lung Cellular and Molecular Physiology, 302(4), L363-L369. American Physiological Society
Gomez-Arroyo, J, Farkas, L, Alhussaini, A A, Farkas, D, Kraskauskas, D, Voelkel, N F & Bogaard, H J 2012, ' The monocrotaline model of pulmonary hypertension in perspective ', American Journal of Physiology. Lung Cellular and Molecular Physiology, vol. 302, no. 4, pp. L363-L369 . https://doi.org/10.1152/ajplung.00212.2011
Gomez-Arroyo, J, Farkas, L, Alhussaini, A A, Farkas, D, Kraskauskas, D, Voelkel, N F & Bogaard, H J 2012, ' The monocrotaline model of pulmonary hypertension in perspective ', American Journal of Physiology. Lung Cellular and Molecular Physiology, vol. 302, no. 4, pp. L363-L369 . https://doi.org/10.1152/ajplung.00212.2011
Severe forms of pulmonary arterial hypertension (PAH) are characterized by various degrees of remodeling of the pulmonary arterial vessels, which increases the pulmonary vascular resistance and right ventricular afterload, thus contributing to the de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::db62d9aa99758712aa40a10e8007ecce
https://research.vumc.nl/en/publications/9b7b44cf-b2ad-4d79-b56b-eff2006ae226
https://research.vumc.nl/en/publications/9b7b44cf-b2ad-4d79-b56b-eff2006ae226