Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Natali Gulbahce"'
Autor:
Kelan G. Tantisira, Conway C. Huang, Maksim Kitsak, Benjamin A. Raby, Weiliang Qiu, Edwin K. Silverman, Scott T. Weiss, Amitabh Sharma, Nidhi Sahni, Xiaobo Zhou, Radu Dobrin, Hao Liu, Albert-László Barabási, Linh Voung, Joel Tocker, Marc Vidal, Elliot S. Barnathan, Susan Dina Ghiassian, Frédéric Baribaud, Natali Gulbahce, Tatiana Ort, Feng Guo, Derek Thibault, Jörg Menche, Reynold A. Panettieri
Publikováno v:
Human molecular genetics. 24(11)
Recent advances in genetics have spurred rapid progress towards the systematic identification of genes involved in complex diseases. Still, the detailed understanding of the molecular and physiological mechanisms through which these genes affect dise
Autor:
Alyce A. Chen, David E. Hill, Amy M. Holthaus, Theodore R. Pak, Mariet C.W. Feltkamp, Danielle Byrdsong, James A. DeCaprio, Rachel Franchi, Melissa Duarte, Saurav Singh, Manor Askenazi, Orit Rozenblatt-Rosen, Jennifer M. Spangle, Rameen Beroukhim, Larisa Litovchick, Natali Gulbahce, Sam Pevzner, Miranda Grace, Sabrina Rabello, Karl Münger, Elliott Kieff, Anne-Ruxandra Carvunis, Yun Shen, Scott B. Ficarro, Thomas Rolland, Tong Hao, Brijesh K. Garg, Michael A. Calderwood, Anna Korkhin, Renee Rubio, Jessica C. Mar, Maria Tavares, Shelly Wanamaker, Murat Tasan, Frederick P. Roth, Fieda Abderazzaq, Robert James, James T. Webber, Albert-László Barabási, Jarrod A. Marto, Mick Correll, Changyu Fan, John Quackenbush, Megha Padi, Guillaume Adelmant, Rahul C. Deo, Amélie Dricot, Marc Vidal, Jingwei Cheng, Michael E. Cusick, Jennifer Roecklein-Canfield, Eric Johannsen
Publikováno v:
Nature, 487(7408), 491-495
Genotypic differences greatly influence susceptibility and resistance to disease. Understanding genotype-phenotype relationships requires that phenotypes be viewed as manifestations of network properties, rather than simply as the result of individua
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f9c84d9cde906583091007b3dbfeaf34
https://hdl.handle.net/1887/106305
https://hdl.handle.net/1887/106305
Autor:
Han Yan, Karin Hellner, Marc Vidal, Jarrod A. Marto, Megha Padi, Jessica C. Mar, Amy Baldwin, Amélie Dricot, Danielle Byrdsong, Nicolas Simonis, Nicholas A. Christakis, Bo Zhao, David E. Hill, Alyce A. Chen, Karl Münger, Renee Rubio, Elliott Kieff, Frederick P. Roth, John Quackenbush, Michael A. Calderwood, Albert-László Barabási, Michael E. Cusick, Rachel Franchi, Deok-Sun Lee, Pascal Braun, Natali Gulbahce, Miranda Grace, Balaji Santhanam, Orit Rozenblatt-Rosen, Jennifer Roecklein-Canfield, James A. DeCaprio, Kyung-Won Huh
Publikováno v:
PLoS Computational Biology
PLoS Computational Biology, Vol 8, Iss 6, p e1002531 (2012)
PLoS computational biology, 8 (6
PLoS Computational Biology, Vol 8, Iss 6, p e1002531 (2012)
PLoS computational biology, 8 (6
Many human diseases, arising from mutations of disease susceptibility genes (genetic diseases), are also associated with viral infections (virally implicated diseases), either in a directly causal manner or by indirect associations. Here we examine w
Publikováno v:
Briefings in functional genomics. 10(5)
Despite the considerable progress in disease gene discovery, we are far from uncovering the underlying cellular mechanisms of diseases since complex traits, even many Mendelian diseases, cannot be explained by simple genotype ^ phenotype relationship