Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Mark Segal"'
Autor:
Abdel Alli, Dhruv Desai, Ahmed Elshika, Marcus Conrad, Bettina Proneth, William Clapp, Carl Atkinson, Mark Segal, Louis Searcy, Nancy Denslow, Subhashini Bolisetty, Borna Mehrad, Laurence Morel, Yogesh Scindia
Publikováno v:
Clin. Immunol. 248:109213 (2022)
While the deposition of immune complexes in the glomeruli is thought to initiate lupus nephritis, the extent of tubular damage better predicts progression to end stage kidney disease than the glomerular injury. However, the mechanisms underlying tubu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::459bb467445304587160135b4e0fd47c
https://doi.org/10.21203/rs.3.rs-2038767/v1
https://doi.org/10.21203/rs.3.rs-2038767/v1
Chronic Critical Illness and the Persistent Inflammation, Immunosuppression, and Catabolism Syndrome
Autor:
Russell B. Hawkins, Steven L. Raymond, Julie A. Stortz, Hiroyuki Horiguchi, Scott C. Brakenridge, Anna Gardner, Philip A. Efron, Azra Bihorac, Mark Segal, Frederick A. Moore, Lyle L. Moldawer
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
Frontiers in Immunology
Frontiers in Immunology
Dysregulated host immune responses to infection often occur, leading to sepsis, multiple organ failure, and death. Some patients rapidly recover from sepsis, but many develop chronic critical illness (CCI), a debilitating condition that impacts funct
Autor:
Michael McNicoll, Peter Martin, Mark Segal, Gabriel Calzada, Young S. Oh, Terry Y. Shibuya, Marc Camilon, Majid Al-Thobaiti
Publikováno v:
Endocrinology&Metabolism International Journal. 2
Sebaceous glands are holocrine adnexal components of the skin that are usually found in close association with hair follicles. Historically, malignancies of the ocular adnexa were felt to be more aggressive than extra-ocular lesions, having greater p
Publikováno v:
Inflammation. 6(2)
Oxygen radicals have been shown to alter granulocyte function by injury to the cell membrane or cytoskeleton. We have investigated the effect of such injury on the aggregation of granulocytes upon C5a or PMA stimulation. Granulocyte aggregation was n