Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Barbara, Kronsteiner"'
Autor:
Raquel Hontecillas, Josep Bassaganya-Riera, Andrew Leber, Barbara Kronsteiner, Victoria Zoccoli-Rodriguez, Vida Abedi, Monica Viladomiu, Nuria Tubau-Juni, Casandra Philipson
Publikováno v:
Scientific Reports, Vol 10, Iss 1, Pp 1-15 (2020)
Scientific Reports
Scientific Reports
Helicobacter pylori is a gram-negative bacterium that persistently colonizes the human stomach by inducing immunoregulatory responses. We have used a novel platform that integrates a bone marrow-derived macrophage and live H. pylori co-culture with g
Autor:
Monica Viladomiu, Barbara Kronsteiner, Victoria Zoccoli-Rodriguez, Casandra Philipson, Raquel Hontecillas, Nuria Tubau-Juni, Andrew Leber, Josep Bassaganya-Riera
Helicobacter pylori, the dominant member of the human gastric microbiota, elicits immunoregulatory responses implicated in protective versus pathological outcomes. To evaluate the role of macrophages during infection, we employed a system with a shif
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3f974d3eab6c8eb4ff001c4ef3ad8f57
https://ora.ox.ac.uk/objects/uuid:2d48b4a1-599f-470d-976c-02a35ad91448
https://ora.ox.ac.uk/objects/uuid:2d48b4a1-599f-470d-976c-02a35ad91448
Autor:
Raquel Hontecillas, Casandra Philipson, Barbara Kronsteiner, Adria Carbo, Vida Abedi, Josep Bassaganya-Riera, Monica Viladomiu
Publikováno v:
Gut Microbes. 7:3-21
Helicobacter pylori is the dominant member of the gastric microbiota in over half of the human population of which 5–15% develop gastritis or gastric malignancies. Immune responses to H. pylori are characterized by mixed T helper cell, cytotoxic T
Autor:
Josep Bassaganya-Riera, Adria Carbo, Monica Viladomiu, Raquel Hontecillas, Barbara Kronsteiner, Mireia Pedragosa, Salvador Vento, Casandra Philipson
Publikováno v:
Infection and Immunity. 81:3803-3813
Helicobacter pylori infection is the leading cause for peptic ulcer disease and gastric adenocarcinoma. Mucosal T cell responses play an important role in mediating H. pylori -related gastric immunopathology. While induced regulatory T (iTreg) cells
Autor:
Josep Bassaganya-Riera, Stephen E. Girardin, Monica Viladomiu, Casandra Philipson, Pawel Michalak, Vida Abedi, Lin Kang, Stefan Hoops, Barbara Kronsteiner, Raquel Hontecillas
Publikováno v:
PLoS ONE
PLoS ONE, Vol 10, Iss 9, p e0137839 (2015)
PLoS ONE, Vol 10, Iss 9, p e0137839 (2015)
Helicobacter pylori colonizes half of the world’s population as the dominant member of the gastric microbiota resulting in a lifelong chronic infection. Host responses toward the bacterium can result in asymptomatic, pathogenic or even favorable he
Publikováno v:
Gut microbes. 5(3)
Helicobacter pylori chronically persists in 50% of the human population and causes serious gastric and duodenal pathologies in 15% of infected people. Research on the immune response to the infection has mainly focused on the induction of CD4+ T cell
Autor:
Michael Groeschl, Raquel Hontecillas, Gabriela Lopez-Velasco, Xiaoying Zhang, Josep Bassaganya-Riera, Shrinivasrao P. Mane, Pinyi Lu, Saroj Kant Mohapatra, Adria Carbo, Barbara Kronsteiner, Amir J. Guri, William T. Horne, Mireia Pedragosa, Bruno W. S. Sobral, Monica Viladomiu, Maria Gloria Dominguez-Bello
Publikováno v:
PLoS ONE, Vol 7, Iss 11, p e50069 (2012)
PLoS ONE
PLoS ONE
Background There is an inverse secular trend between the incidence of obesity and gastric colonization with Helicobacter pylori, a bacterium that can affect the secretion of gastric hormones that relate to energy homeostasis. H. pylori strains that c
Autor:
Maksudul Alam, Monica Viladomiu, Adria Carbo, Madhav V. Marathe, Stephen Eubank, Raquel Hontecillas, Josep Bassaganya-Riera, Stefan Hoops, Keith R. Bisset, Yongguo Mei, Barbara Kronsteiner, Mireia Pedragosa, Katherine Wendelsdorf, Xinwei Deng
Publikováno v:
PLoS ONE, Vol 8, Iss 9, p e73365 (2013)
PLoS ONE
PLoS ONE
T helper (Th) cells play a major role in the immune response and pathology at the gastric mucosa during Helicobacter pylori infection. There is a limited mechanistic understanding regarding the contributions of CD4+ T cell subsets to gastritis develo