Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Bart J.M. van Vlijmen"'
Autor:
Yvonne K. Jongejan, Jimmy F.P. Berbée, Jeroen Eikenboom, Marion J.J. Gijbels, Bart J.M. van Vlijmen
Publikováno v:
Journal of Thrombosis and Thrombolysis
Journal of Thrombosis and Thrombolysis, 52(3), 715-719. Springer, Cham
Journal of thrombosis and thrombolysis, 52(3), 715-719. Springer Netherlands
Journal of Thrombosis and Thrombolysis, 52, 715-719. SPRINGER
Journal of Thrombosis and Thrombolysis, 52(3), 715-719. Springer, Cham
Journal of thrombosis and thrombolysis, 52(3), 715-719. Springer Netherlands
Journal of Thrombosis and Thrombolysis, 52, 715-719. SPRINGER
Murine atherosclerosis models are key for investigation of atherosclerosis pathophysiology and drug development. However, they do not feature spontaneous atherothrombosis as a final stage of atherosclerosis. Transgenic mice expressing both the human
Autor:
Sarah A. Michaud, Christoph H. Borchers, Julia Tilburg, Henri H. Versteeg, Yassene Mohammed, Bart J.M. van Vlijmen, Chrissta X. Maracle
Publikováno v:
Thrombosis and Haemostasis, 120(3), 423-436. GEORG THIEME VERLAG KG
The plasma compartment of the blood holds important information on the risk to develop cardiovascular diseases such as venous thrombosis (VT). Mass spectrometry-based targeted proteomics with internal standards quantifies proteins in multiplex allowi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9b4bb63a1a4fe8590b2eb9c6bc6eb806
https://hdl.handle.net/1887/3182306
https://hdl.handle.net/1887/3182306
Autor:
Henri M. H. Spronk, Julia Tilburg, Madeline Jackson, Michael Holinstat, David C. Tuk, Megan Hawley, Bart J.M. van Vlijmen, Thankam S. Nair, Chrissta X. Maracle, Reheman Adili, Henri H. Versteeg, Thomas E. Carey
Publikováno v:
Thrombosis Research, 171, 155-159. Elsevier Science
Thrombosis research, 171, 155-159. Elsevier Limited
Thrombosis research, 171, 155-159. Elsevier Limited
Introduction: Recent genome wide association studies (GWAS) identified a novel susceptibility locus for thrombosis, harbouring the SLC44A2 gene which encodes the Solute Carrier Family 44 Member 2 protein (SLC44A2). Thus far, SLC44A2 has not been stud
Autor:
Salam Salloum-Asfar, Thomas Renné, Daniel Kirchhofer, El Houari Laghmani, Sacha Zeerleder, Brenda M. Luken, Daniela C.F. Salvatori, Henri M. H. Spronk, Marco Heestermans, Henri H. Versteeg, Gerry T. M. Wagenaar, Pieter H. Reitsma, Bart J.M. van Vlijmen, Suzanne J.A. Korporaal, Tom Streef
Publikováno v:
Blood, 133(19), 2090-2099. American Society of Hematology
Blood, 133(19), 2090-2099. AMER SOC HEMATOLOGY
Blood, 133(19), 2090-2099. The American Society of Hematology
Blood, 133(19), 2090-2099. AMER SOC HEMATOLOGY
Blood, 133(19), 2090-2099. The American Society of Hematology
Tissue factor, coagulation factor XII, platelets, and neutrophils are implicated as important players in the pathophysiology of (experimental) venous thrombosis (VT). Their role became evident in mouse models in which surgical handlings were required
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7974966ef7a0a4a3c0430369b3285502
https://hdl.handle.net/1887/122393
https://hdl.handle.net/1887/122393
Autor:
Bart J.M. van Vlijmen, Jasmin Hassan, Meander Kloosterman, Marion J.J. Gijbels, Amber B. Ouweneel, Miranda Van Eck, Marco Heestermans, Pieter H. Reitsma
Publikováno v:
Scientific reports, 8(1):15106. Nature Publishing Group
Scientific Reports
Scientific Reports, 8
Scientific Reports, 8(1), 15106
Scientific Reports, 8:15106. Nature Publishing Group
Scientific Reports, Vol 8, Iss 1, Pp 1-13 (2018)
Scientific Reports
Scientific Reports, 8
Scientific Reports, 8(1), 15106
Scientific Reports, 8:15106. Nature Publishing Group
Scientific Reports, Vol 8, Iss 1, Pp 1-13 (2018)
Silencing of anticoagulant protein C using RNA interference (siProc) evokes low incident but spontaneous atherothrombosis in the aortic root of apolipoprotein E–deficient (Apoe−/−) mice. The aims of the current study were (1) to analyze if plaq
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36dfc6bb6c59efaf2337044c3d85ba5b
https://pure.amc.nl/en/publications/predilection-of-low-protein-cinduced-spontaneous-atherothrombosis-for-the-right-coronary-sinus-in-apolipoprotein-e-deficient-mice(713483a8-93c7-477b-8065-a4712e5c5438).html
https://pure.amc.nl/en/publications/predilection-of-low-protein-cinduced-spontaneous-atherothrombosis-for-the-right-coronary-sinus-in-apolipoprotein-e-deficient-mice(713483a8-93c7-477b-8065-a4712e5c5438).html
Autor:
Amber B. Ouweneel, Marion J.J. Gijbels, Bart J.M. van Vlijmen, Pieter H. Reitsma, Marco Heestermans, Robin A.F. Verwilligen, Miranda Van Eck
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology, 37(5), 782
Arteriosclerosis, thrombosis, and vascular biology, 37(5), 782-785. Lippincott Williams and Wilkins
Arteriosclerosis Thrombosis and Vascular Biology, 37(5), 782-785. LIPPINCOTT WILLIAMS & WILKINS
Arteriosclerosis, thrombosis, and vascular biology, 37(5), 782-785. Lippincott Williams and Wilkins
Arteriosclerosis Thrombosis and Vascular Biology, 37(5), 782-785. LIPPINCOTT WILLIAMS & WILKINS
Objective— Murine atherosclerosis models do not spontaneously develop atherothrombotic complications. We investigated whether disruption of natural anticoagulation allows preexisting atherosclerotic plaques to progress toward an atherothrombotic ph
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1c8df47343fcbcbee2f12c9d4ea43efd
http://hdl.handle.net/1887/115900
http://hdl.handle.net/1887/115900
Autor:
Salam Salloum-Asfar, Gerry T. M. Wagenaar, Pieter H. Reitsma, Suzanne J.A. Korporaal, Marco Heestermans, Sacha Zeerleder, El Houari Laghmani, Daniela C.F. Salvatori, Henri M. H. Spronk, Bart J.M. van Vlijmen, Brenda M. Luken
Publikováno v:
Blood, 127(21), 2630-2637. American Society of Hematology
Blood, 127(21), 2630. American Society of Hematology
Blood, 127(21), 2630-2637
Blood, 127(21), 2630. American Society of Hematology
Blood, 127(21), 2630-2637
Recently, platelets, neutrophils, and factor XII (FXII) have been implicated as important players in the pathophysiology of venous thrombosis. Their role became evident in mouse models in which surgical handling was used to provoke thrombosis. Inhibi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a7c5ac4812f050da13d8b2ec66935d74
https://dspace.library.uu.nl/handle/1874/342728
https://dspace.library.uu.nl/handle/1874/342728
Autor:
Bart J.M. van Vlijmen, Audrey C. A. Cleuren, Berthe Van Hoef, H. Roger Lijnen, Marc Hoylaerts
Publikováno v:
Thrombosis and Haemostasis. 102:993-1000
SummaryObesity and oral estrogens are independent risk factors for venous thrombosis, and their combined effect is stronger than the sum of the isolated factors. It was the objective of this study to investigate the interaction between obesity and es
Autor:
Louis M. Havekes, Jef J. Emeis, Bart J.M. van Vlijmen, Teake Kooistra, A. Susanne M. Zadelaar, Lianne S.M. Boesten, Germán Camejo, Erik Lundholm, J. Wouter Jukema
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 26:2560-2566
OBJECTIVE - We investigated whether the dual PPARα/γ agonist tesaglitazar has anti-atherogenic effects in ApoE*3Leiden mice with reduced insulin sensitivity. METHODS AND RESULTS - ApoE*3Leiden transgenic mice were fed a high-fat (HF) insulin-resist
Autor:
Abbey Schepers, Johannes Waltenberger, René A. Tio, Pieter A. Doevendans, J. Wouter Jukema, PS Monraats, Willem R.P. Agema, Moniek P.M. de Maat, Robbert J. de Winter, Ernst E. van der Wall, Aeilko H. Zwinderman, Douwe E. Atsma, Rune R. Frants, Paul H.A. Quax, Nuno Pires, Bart J.M. van Vlijmen, Arnoud van der Laarse
Publikováno v:
Circulation, 16, 112, 2417-2425
Circulation, 112(16), 2417-2425. Lippincott Williams & Wilkins
Circulation, 112(16), 2417-2425. Lippincott Williams and Wilkins
Circulation, 112(16), 2417-2425. LIPPINCOTT WILLIAMS & WILKINS
Circulation, 112(16), 2417-2425. Lippincott Williams & Wilkins
Circulation, 112(16), 2417-2425. Lippincott Williams and Wilkins
Circulation, 112(16), 2417-2425. LIPPINCOTT WILLIAMS & WILKINS
Background— Restenosis is a negative effect of percutaneous coronary intervention (PCI). No clinical factors are available that allow good risk stratification. However, evidence exists that genetic factors are important in the restenotic process as