Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Boris Melloni"'
Publikováno v:
Thorax. 51:781-786
BACKGROUND: The proliferative response of type II cells is an important event following silica-induced lung injury. Alveolar macrophages, when activated by fibrogenic agents, secrete various biological mediators which affect cell growth. METHODS: Hum
Publikováno v:
Journal of Leukocyte Biology. 55:574-580
The proliferation of lung epithelial cells is a prominent feature of lung tissue response following silica-induced lung injury and alveolar macrophages are recognized as a major contributing cell to the lung inflammatory processes. In previous studie
Autor:
A Cantin, Pierre Larivée, Raymond Bégin, L Guojian, M. Martel, Olivier Lesur, Boris Melloni, T Bouhadiba
Publikováno v:
Thorax. 48:840-844
BACKGROUND--Some of the prominent features of silicosis are hyperplasia and hypertrophy of epithelial type II cells, which in experimental animals are often accompanied by accumulation of phospholipids in the lung. METHODS--The total phospholipid con
Publikováno v:
Experimental Lung Research. 18:173-190
The contribution of the type II pneumocyte to the pathogenesis of silicosis is largely unknown. Prominent features of silicosis are hyperplasia and hypertrophy of type II epithelial cells, often accompanied by phospholipid accumulation in the lung. T
Publikováno v:
Cellular and Molecular Effects of Mineral and Synthetic Dusts and Fibres ISBN: 9783642790430
Recent advances in the isolation procedures and culture conditions maintaining the type II pneumocyte in a differentiated state in vitro (Mason et al, 1977) have documented that the type II epithelial cell, in addition to producing surfactant, acts a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::c6b05f4809fb4ed5e8ae361fe159174b
https://doi.org/10.1007/978-3-642-79041-6_10
https://doi.org/10.1007/978-3-642-79041-6_10
Publikováno v:
Experimental lung research. 18(5)
The type II pneumocyte changes in silicosis are characterized by hyperplasic and hypertrophic epithelial cells, and increased surfactant phospholipids in the bronchoalveolar lavage fluids (BALF). To assess the proliferative activity of alveolar linin