Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Ulf Hedin"'
Autor:
Mariette Lengquist, Mathilde Frederikke Bjørn Bonde, G. Kees Hovingh, Heidi Engslev Lund, Bidda Rolin, Stefan Zahn, Michael Nyberg, Karin Hultman, Lars Maegdefessel, Valentina Manfè, Pernille Gry Wulff-Larsen, Karin Conde-Knape, Ulf Hedin, Djordje Djordjevic, Rikke Kaae Kirk, Ljubica Perisic Matic, Niels Blume, Günaj Rakipovski, Maj Petersen, Natasha Barascuk
Publikováno v:
Atherosclerosis, 314, 1-9. Elsevier Ireland Ltd
Background and aims Clinical interventions targeting nonlipid risk factors are needed given the high residual risk of atherothrombotic events despite effective control of dyslipidemia. Dickkopf-1 (DKK1) plays a lipid-independent role in vascular path
Autor:
Ulf Hedin, Azadeh Nilchian, Estelle Plant, Malgorzata M Parniewska, Ana Santiago, Aránzazu Rossignoli, Josefin Skogsberg, Ljubica Perisic Matic, Jonas Fuxe
Publikováno v:
The Journal of infectious diseases
The Journal of Infectious Diseases
The Journal of Infectious Diseases
Multiple viruses are implicated in atherosclerosis, but the mechanisms by which they infect cells and contribute to plaque formation in arterial walls are not well understood. Based on reports showing the presence of enterovirus in atherosclerotic pl
Autor:
Xiaohui Wu, Angela Cheng, Thomas Q. de Aguiar Vallim, Elizabeth J. Tarling, Joan Cheng, Lars Maegdefessel, Ulf Hedin, Bethan L. Clifford, Nathalie Pamir, Tamer Sallam
Publikováno v:
Arterioscler Thromb Vasc Biol
Objective: Atherosclerosis is a leading cause of death in developed countries. MicroRNAs act as fine-tuners of gene expression and have been shown to have important roles in the pathophysiology and progression of atherosclerosis. We, and others, prev
Autor:
Ying Wang, Nicholas J. Leeper, Philip S. Tsao, Ljubica Perisic Matic, Kai-Uwe Jarr, Lars Maegdefessel, Greg Winski, Shen Lao, Vivek Nanda, Valentina Paloschi, Jianqin Ye, Pavlos Tsantilas, Alyssa M. Flores, Monika Vaerst, Zhiyuan Wu, Yoko Kojima, Hans-Henning Eckstein, Jaroslav Pelisek, Anne V. Eberhard, Ulf Hedin
Publikováno v:
Cardiovasc Res
Aims Atherosclerotic cerebrovascular disease underlies the majority of ischaemic strokes and is a major cause of death and disability. While plaque burden is a predictor of adverse outcomes, plaque vulnerability is increasingly recognized as a driver
Autor:
Mona, Ahmed, Tetyana, Tegnebratt, Thuy A, Tran, Li, Lu, Peter, Damberg, Anton, Gisterå, Laura, Tarnawski, Dianna, Bone, Ulf, Hedin, Per, Eriksson, Staffan, Holmin, Björn, Gustafsson, Kenneth, Caidahl
Publikováno v:
International Journal of Nanomedicine
Background Beyond clinical atherosclerosis imaging of vessel stenosis and plaque morphology, early detection of inflamed atherosclerotic lesions by molecular imaging could improve risk assessment and clinical management in high-risk patients. To iden
Autor:
Mariette Lengquist, Malin Kronqvist, Ulf Hedin, Cih-Yi Yen, Chi-Nan Tseng, Einar E. Eriksson, Ya-Ting Chang
Publikováno v:
Thrombosis and haemostasis. 119(12)
Inflammatory processes contribute to intimal hyperplasia (IH) and long-term failure of vein grafts used in bypass surgery. Leukocyte recruitment on endothelial cells of vessels during inflammation is regulated by P-selectin and P-selectin glycoprotei
Autor:
Denuja Karunakaran, Jason E. Fish, Kristofferson Tandoc, Michele Geoffrion, Katey J. Rayner, Henry S. Cheng, Ljubica Perisic Matic, My Anh Nguyen, Ulf Hedin, Lars Maegdefessel
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 38:49-63
Objective— During inflammation, macrophages secrete vesicles carrying RNA, protein, and lipids as a form of extracellular communication. In the vessel wall, extracellular vesicles (EVs) have been shown to be transferred between vascular cells durin
Autor:
Jan H.N. Lindeman, Ulf de Faire, Anders Hamsten, Ida Ericsson, Ewa Ehrenborg, Maria Gonzalez Diez, Silvia Aldi, Joëlle Magné, Thomas Quertermous, Valentina Paloschi, Per Eriksson, Clint L. Miller, Elena Tremoli, Maria Sabater-Lleal, Mariette Lengquist, Ljubica Perisic Matic, Ulf Hedin, Damiano Baldassarre, Hong Jin, Mattias Vesterlund, Yuhuang Li, Anton Razuvaev, Fabrizio Veglia, Urszula Rykaczewska, Janne Lehtiö, Gabrielle Paulsson-Berne, Vladana Vukojević, Göran K. Hansson, Jacob Odeberg, Steve E. Humphries, Joy Roy, Lars Maegdefessel, Malin Kronqvist, Samuel Röhl
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology, 36(9), 1947-1961
Arterioscler Thromb Vasc Biol
Arterioscler Thromb Vasc Biol
Objective— Key augmented processes in atherosclerosis have been identified, whereas less is known about downregulated pathways. Here, we applied a systems biology approach to examine suppressed molecular signatures, with the hypothesis that they ma
Autor:
Daniel Y. Li, Ljubica Perisic Matic, Fredrik Wermeling, Ulf Hedin, Dorothee Atzler, Sanjay V. Boddul, Lars Maegdesfessel, Sanna Hellberg, Hong Jin, Changyan Sun, Nienke J Verzaal, Albert Busch, Göran K. Hansson, Lisa Hofste, Christoph J. Binder, Esther Lutgens, Stephen Malin, Daniel F. J. Ketelhuth, Roland Baumgartner, Monica Centa, Sara Lind Enoksson
Publikováno v:
Circulation, 139(21), 2466-2482. Lippincott Williams and Wilkins
Background: Atherosclerosis progression is modulated by interactions with the adaptive immune system. Humoral immunity can help protect against atherosclerosis formation; however, the existence, origin, and function of putative atherogenic antibodies
Autor:
Wolfgang Poller, Marion Muhly-Reinholz, Daniel Y. Li, Shizuka Uchida, Andreas M. Zeiher, Sebastian Cremer, Christian Weber, Ulf Hedin, Thomas Braun, Larissa Pfisterer, Stefan Günther, Ljubica Perisic Matic, Reinier A. Boon, Lars Maegdefessel, David John, Oliver Soehnlein, Carla Winter, Ariane Fischer, Stefanie Dimmeler, Katharina M. Michalik, Nicolas Jaé
Publikováno v:
Circulation, 139(10), 1320-1334. LIPPINCOTT WILLIAMS & WILKINS
Cremer, S, Michalik, K M, Fischer, A, Pfisterer, L, Jaé, N, Winter, C, Boon, R A, Muhly-Reinholz, M, John, D, Uchida, S, Weber, C, Poller, W, Günther, S, Braun, T, Li, D Y, Maegdefessel, L, Perisic Matic, L, Hedin, U, Soehnlein, O, Zeiher, A & Dimmeler, S 2019, ' Hematopoietic Deficiency of the Long Noncoding RNA MALAT1 Promotes Atherosclerosis and Plaque Inflammation ', Circulation, vol. 139, no. 10, pp. 1320-1334 . https://doi.org/10.1161/CIRCULATIONAHA.117.029015
Circulation, 139(10), 1320-1334. Lippincott Williams and Wilkins
Circulation
Cremer, S, Michalik, K M, Fischer, A, Pfisterer, L, Jaé, N, Winter, C, Boon, R A, Muhly-Reinholz, M, John, D, Uchida, S, Weber, C, Poller, W, Günther, S, Braun, T, Li, D Y, Maegdefessel, L, Perisic Matic, L, Hedin, U, Soehnlein, O, Zeiher, A & Dimmeler, S 2019, ' Hematopoietic Deficiency of the Long Noncoding RNA MALAT1 Promotes Atherosclerosis and Plaque Inflammation ', Circulation, vol. 139, no. 10, pp. 1320-1334 . https://doi.org/10.1161/CIRCULATIONAHA.117.029015
Circulation, 139(10), 1320-1334. Lippincott Williams and Wilkins
Circulation
Background: The majority of the human genome comprises noncoding sequences, which are in part transcribed as long noncoding RNAs (lncRNAs). lncRNAs exhibit multiple functions, including the epigenetic control of gene expression. In this study, the ef
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8e509e66a5f3f6835b44ccf8218592ef
https://cris.maastrichtuniversity.nl/en/publications/0e91224f-542d-43a7-93b8-bbcfc3e04280
https://cris.maastrichtuniversity.nl/en/publications/0e91224f-542d-43a7-93b8-bbcfc3e04280