Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Yoshimi, Tsuda"'
Autor:
Fumiya Kozawa, Kumiko Yoshimatsu, Kisho Noda, Devinda S. Muthusinghe, Rashid Manzoor, Sithumini M. W. Lokupathirage, Kodai Ikegame, Kenta Shimizu, Yoshimi Tsuda
Publikováno v:
Scientific Reports
Scientific Reports, Vol 11, Iss 1, Pp 1-11 (2021)
Scientific Reports, Vol 11, Iss 1, Pp 1-11 (2021)
Severe fever with thrombocytopenia syndrome virus (SFTSV) is an emerging bunyavirus that causes novel zoonotic diseases in Asian countries including China, Japan, South Korea, and Vietnam. In phleboviruses, viral proteins play a critical role in vira
Autor:
Yoshimi Tsuda, Jiro Arikawa, Kumiko Yoshimatsu, Manabu Igarashi, Fumiya Kozawa, Kisho Noda, Kenta Shimizu
Publikováno v:
Viruses
Volume 13
Issue 1
Viruses, Vol 13, Iss 33, p 33 (2021)
Volume 13
Issue 1
Viruses, Vol 13, Iss 33, p 33 (2021)
Severe fever with thrombocytopenia syndrome virus subclone B7 shows strong plaque formation and cytopathic effect induction compared with other subclones and the parental strain YG1. Compared to YG1 and the other subclones, only B7 possesses a single
Autor:
Kentaro Yoshii, Jiro Arikawa, Hiroaki Kariwa, Kumiko Yoshimatsu, Ryo Ito, Yoshimi Tsuda, Kenta Shimizu, Tapiwa Lundu, Shintaro Kobayashi
Publikováno v:
Biomedical Research. 39:27-38
Severe fever with thrombocytopenia syndrome phlebovirus (SFTSV) is a newly emerged phlebovirus identified in China, Japan, and South Korea. Phlebovirus glycoproteins (GP) play a key role in targeting viral structural components to the budding compart
Autor:
Kumiko Yoshimatsu, Sanae Nishio, Manabu Igarashi, Kenta Shimizu, Ryo Ito, Yoshimi Tsuda, Jiro Arikawa
Publikováno v:
Biomedical Research. 38:89-97
Severe fever with thrombocytopenia syndrome virus (SFTSV) is a novel phlebovirus responsible for causing an emerging zoonotic disease. We previously established subclones from SFTSV strain YG1 based on differences in low-pH-dependent cell fusion acti
Autor:
Naganori Nao, Junya Yamagishi, Hiroko Miyamoto, Manabu Igarashi, Rashid Manzoor, Aiko Ohnuma, Yoshimi Tsuda, Wakako Furuyama, Asako Shigeno, Masahiro Kajihara, Noriko Kishida, Reiko Yoshida, Ayato Takada, Mary K. Estes
Publikováno v:
mBio, Vol 8, Iss 1 (2017)
mBio, Vol 8, Iss 1, p e02298-16 (2017)
mBio
mBio, Vol 8, Iss 1, p e02298-16 (2017)
mBio
Highly pathogenic avian influenza viruses with H5 and H7 hemagglutinin (HA) subtypes evolve from low-pathogenic precursors through the acquisition of multiple basic amino acid residues at the HA cleavage site. Although this mechanism has been observe
Publikováno v:
Virus Research. 140:194-198
Many influenza A viruses form plaques on Madin-Darby canine kidney (MDCK) cells in the presence of trypsin. A/duck/Siberia/272/1998 (H13N6) (Sib272), however, does not form plaque on MDCK cells. After three blind passages of the strain on MDCK cells,